Rui-Yun Bi

540 total citations
13 papers, 451 citations indexed

About

Rui-Yun Bi is a scholar working on Physiology, Cellular and Molecular Neuroscience and Complementary and Manual Therapy. According to data from OpenAlex, Rui-Yun Bi has authored 13 papers receiving a total of 451 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Physiology, 5 papers in Cellular and Molecular Neuroscience and 3 papers in Complementary and Manual Therapy. Recurrent topics in Rui-Yun Bi's work include Pain Mechanisms and Treatments (5 papers), Neuropeptides and Animal Physiology (4 papers) and Temporomandibular Joint Disorders (3 papers). Rui-Yun Bi is often cited by papers focused on Pain Mechanisms and Treatments (5 papers), Neuropeptides and Animal Physiology (4 papers) and Temporomandibular Joint Disorders (3 papers). Rui-Yun Bi collaborates with scholars based in China and United States. Rui-Yun Bi's co-authors include Ye-Hua Gan, Xiaoxing Kou, Zhen Meng, Yanheng Zhou, Xuchen Ma, Maomao Zeng, Jieni Zhang, Yan Liu, Danqing He and Xuedong Wang and has published in prestigious journals such as PLoS ONE, Scientific Reports and Journal of Dental Research.

In The Last Decade

Rui-Yun Bi

12 papers receiving 442 citations

Peers

Rui-Yun Bi
Rui-Yun Bi
Citations per year, relative to Rui-Yun Bi Rui-Yun Bi (= 1×) peers Anna Appelgren

Countries citing papers authored by Rui-Yun Bi

Since Specialization
Citations

This map shows the geographic impact of Rui-Yun Bi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rui-Yun Bi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rui-Yun Bi more than expected).

Fields of papers citing papers by Rui-Yun Bi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rui-Yun Bi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rui-Yun Bi. The network helps show where Rui-Yun Bi may publish in the future.

Co-authorship network of co-authors of Rui-Yun Bi

This figure shows the co-authorship network connecting the top 25 collaborators of Rui-Yun Bi. A scholar is included among the top collaborators of Rui-Yun Bi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rui-Yun Bi. Rui-Yun Bi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Zhang, Qiang, et al.. (2025). Fe7S8/FeS2 Heterostructures in Carbon Nanospheres for High-Performance Sodium Storage. ACS Applied Nano Materials. 8(14). 6886–6896.
2.
Bi, Rui-Yun, et al.. (2020). Progesterone Attenuates Allodynia of Inflamed Temporomandibular Joint through Modulating Voltage-Gated Sodium Channel 1.7 in Trigeminal Ganglion. Pain Research and Management. 2020. 1–11. 11 indexed citations
3.
Zhang, Peng, Rui-Yun Bi, & Ye-Hua Gan. (2018). Glial interleukin-1β upregulates neuronal sodium channel 1.7 in trigeminal ganglion contributing to temporomandibular joint inflammatory hypernociception in rats. Journal of Neuroinflammation. 15(1). 117–117. 34 indexed citations
4.
Zhang, Xiaoyu, Rui-Yun Bi, Peng Zhang, & Yehua Gan. (2018). Veratridine modifies the gating of human voltage-gated sodium channel Nav1.7. Acta Pharmacologica Sinica. 39(11). 1716–1724. 17 indexed citations
5.
Bi, Rui-Yun, et al.. (2018). Genistein Antagonizes 17β-Estradiol Effects on Glutamate-Evoked Masseter Muscle Hypernociception in Rats. Frontiers in Neurology. 9. 649–649. 8 indexed citations
6.
Kou, Xiaoxing, Chenshuang Li, Rui-Yun Bi, et al.. (2017). Progesterone attenuates temporomandibular joint inflammation through inhibition of NF-κB pathway in ovariectomized rats. Scientific Reports. 7(1). 15334–15334. 33 indexed citations
7.
Bi, Rui-Yun, et al.. (2017). Estradiol upregulates voltage-gated sodium channel 1.7 in trigeminal ganglion contributing to hyperalgesia of inflamed TMJ. PLoS ONE. 12(6). e0178589–e0178589. 49 indexed citations
8.
Kou, Xiaoxing, Chenshuang Li, Rui-Yun Bi, et al.. (2014). Estradiol-potentiated cadherin-11 in synovial membrane involves in temporomandibular joint inflammation in rats. The Journal of Steroid Biochemistry and Molecular Biology. 143. 444–450. 11 indexed citations
10.
Kou, Xiaoxing, et al.. (2013). Estrogen Aggravates Iodoacetate-induced Temporomandibular Joint Osteoarthritis. Journal of Dental Research. 92(10). 918–924. 56 indexed citations
11.
Kou, Xiaoxing, Rui-Yun Bi, Wen Xu, et al.. (2012). Hippocampal Nerve Growth Factor Potentiated by 17β-Estradiol and Involved in Allodynia of Inflamed TMJ in Rat. Journal of Pain. 13(6). 555–563. 14 indexed citations
12.
Wang, Xuedong, Xiaoxing Kou, Danqing He, et al.. (2012). Progression of Cartilage Degradation, Bone Resorption and Pain in Rat Temporomandibular Joint Osteoarthritis Induced by Injection of Iodoacetate. PLoS ONE. 7(9). e45036–e45036. 116 indexed citations
13.
Kou, Xiaoxing, et al.. (2011). 17β-estradiol aggravates temporomandibular joint inflammation through the NF-κB pathway in ovariectomized rats. Arthritis & Rheumatism. 63(7). 1888–1897. 76 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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