Rosa M. Soler

4.7k total citations
43 papers, 1.9k citations indexed

About

Rosa M. Soler is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Rosa M. Soler has authored 43 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 19 papers in Cellular and Molecular Neuroscience and 15 papers in Genetics. Recurrent topics in Rosa M. Soler's work include Nerve injury and regeneration (16 papers), Neurogenetic and Muscular Disorders Research (15 papers) and RNA modifications and cancer (8 papers). Rosa M. Soler is often cited by papers focused on Nerve injury and regeneration (16 papers), Neurogenetic and Muscular Disorders Research (15 papers) and RNA modifications and cancer (8 papers). Rosa M. Soler collaborates with scholars based in Spain, United Kingdom and United States. Rosa M. Soler's co-authors include Joan X. Comella, Jorge Gálvez, Joaquim Egea, Xavier Dolcet, Ricardo Garcı́a, Jesus Vicente de Julián‐Ortiz, R. Garcia‐Domenech, Ana Garcerá, Mario Encinas and Dionisio Martín‐Zanca and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Neuron.

In The Last Decade

Rosa M. Soler

42 papers receiving 1.8k citations

Peers

Rosa M. Soler

CMN

CTM

GENET

Xiaoxiang Zhu United States

Karlheinz Baumann Germany

Nichole E. LaPointe United States

Geneviève Evin Australia

G. Gaviraghi Italy

Thomas L. Deckwerth United States

Sabrina Taliani Italy

Faraj Terro France

Patrick Horne Canada

Tony Giordano United States

Xiaoxiang Zhu United States

Rosa M. Soler

1.8k ×1.9

252 ×0.4

CMN

444 ×1.2

CTM

95 ×0.3

GENET

69 ×0.3

Citations per year, relative to Rosa M. Soler Rosa M. Soler (= 1×) peers Xiaoxiang Zhu

Countries citing papers authored by Rosa M. Soler

Since Specialization

Citations

This map shows the geographic impact of Rosa M. Soler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rosa M. Soler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rosa M. Soler more than expected).

Fields of papers citing papers by Rosa M. Soler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rosa M. Soler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rosa M. Soler. The network helps show where Rosa M. Soler may publish in the future.

Co-authorship network of co-authors of Rosa M. Soler

This figure shows the co-authorship network connecting the top 25 collaborators of Rosa M. Soler. A scholar is included among the top collaborators of Rosa M. Soler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rosa M. Soler. Rosa M. Soler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Torres, Pascual, et al.. (2024). TDP-43 dysfunction leads to bioenergetic failure and lipid metabolic rewiring in human cells. Redox Biology. 75. 103301–103301. 11 indexed citations

Calderó, Jordi, et al.. (2023). ERK MAPK signaling pathway inhibition as a potential target to prevent autophagy alterations in Spinal Muscular Atrophy motoneurons. Cell Death Discovery. 9(1). 113–113. 7 indexed citations

Comella, Joan X., et al.. (2021). Intracellular pathways involved in cell survival are deregulated in mouse and human spinal muscular atrophy motoneurons. Neurobiology of Disease. 155. 105366–105366. 9 indexed citations

García‐Morales, Victoria, Guillermo Rodríguez Bey, Germán Domínguez-Vías, et al.. (2019). Sp1-regulated expression of p11 contributes to motor neuron degeneration by membrane insertion of TASK1. Nature Communications. 10(1). 3784–3784. 23 indexed citations

Garcerá, Ana, et al.. (2018). Calpain Inhibition Increases SMN Protein in Spinal Cord Motoneurons and Ameliorates the Spinal Muscular Atrophy Phenotype in Mice. Molecular Neurobiology. 56(6). 4414–4427. 11 indexed citations

Piras, Antonio, Lorenzo Schiaffino, Marina Boido, et al.. (2017). Inhibition of autophagy delays motoneuron degeneration and extends lifespan in a mouse model of spinal muscular atrophy. Cell Death and Disease. 8(12). 3223–3223. 41 indexed citations

Arumugam, Saravanan, et al.. (2017). Regulation of Survival Motor Neuron Protein by the Nuclear Factor-Kappa B Pathway in Mouse Spinal Cord Motoneurons. Molecular Neurobiology. 55(6). 5019–5030. 11 indexed citations

Arumugam, Saravanan, Ana Garcerá, Rosa M. Soler, & Lucı́a Tabares. (2017). Smn-Deficiency Increases the Intrinsic Excitability of Motoneurons. Frontiers in Cellular Neuroscience. 11. 269–269. 10 indexed citations

Arumugam, Saravanan, et al.. (2016). Autophagy modulators regulate survival motor neuron protein stability in motoneurons. Experimental Neurology. 283(Pt A). 287–297. 21 indexed citations

10.

Garcerá, Ana, et al.. (2013). Survival motor neuron protein reduction deregulates autophagy in spinal cord motoneurons in vitro. Cell Death and Disease. 4(6). e686–e686. 31 indexed citations

11.

Moubarak, Rana S., Carme Solé, Marta Pascual, et al.. (2010). The Death Receptor Antagonist FLIP-L Interacts with Trk and Is Necessary for Neurite Outgrowth Induced by Neurotrophins. Journal of Neuroscience. 30(17). 6094–6105. 13 indexed citations

12.

Garcerá, Ana, et al.. (2009). Specific vulnerability of mouse spinal cord motoneurons to membrane depolarization. Journal of Neurochemistry. 110(6). 1842–1854. 24 indexed citations

13.

Martí‐Fàbregas, Joan, et al.. (2009). Clinical status of motoneuron disease does not correlate with serum neurotoxicity on cultured neurons. Acta Neurologica Scandinavica. 85(3). 219–223. 2 indexed citations

14.

Fernández-Gómez, Francisco-José, María Dolores Pastor, María Gómez-Lázaro, et al.. (2006). Pyruvate protects cerebellar granular cells from 6-hydroxydopamine-induced cytotoxicity by activating the Akt signaling pathway and increasing glutathione peroxidase expression. Neurobiology of Disease. 24(2). 296–307. 38 indexed citations

15.

Pérez‐García, M. Jose, Valentı́n Ceña, Yolanda de Pablo, et al.. (2004). Glial Cell Line-derived Neurotrophic Factor Increases Intracellular Calcium Concentration. Journal of Biological Chemistry. 279(7). 6132–6142. 77 indexed citations

16.

Dolcet, Xavier, Rosa M. Soler, Thomas W. Gould, et al.. (2001). Cytokines Promote Motoneuron Survival through the Janus Kinase-Dependent Activation of the Phosphatidylinositol 3-Kinase Pathway. Molecular and Cellular Neuroscience. 18(6). 619–631. 82 indexed citations

17.

Egea, Joaquim, Carme Espinet, Rosa M. Soler, et al.. (2000). Nerve Growth Factor Activation of the Extracellular Signal-Regulated Kinase Pathway Is Modulated by Ca ²⁺ and Calmodulin. Molecular and Cellular Biology. 20(6). 1931–1946. 47 indexed citations

18.

Dolcet, Xavier, Joaquim Egea, Rosa M. Soler, Dionisio Martín‐Zanca, & Joan X. Comella. (1999). Activation of Phosphatidylinositol 3‐Kinase, but Not Extracellular‐Regulated Kinases, Is Necessary to Mediate Brain‐Derived Neurotrophic Factor‐Induced Motoneuron Survival. Journal of Neurochemistry. 73(2). 521–531. 113 indexed citations

19.

Minichiello, Liliana, Franca Casagranda, Rosa M. Soler, et al.. (1998). Point Mutation in trkB Causes Loss of NT4-Dependent Neurons without Major Effects on Diverse BDNF Responses. Neuron. 21(2). 335–345. 168 indexed citations

20.

Gálvez, Jorge, R. Garcia‐Domenech, Jesus Vicente de Julián‐Ortiz, & Rosa M. Soler. (1995). Topological Approach to Drug Design. Journal of Chemical Information and Computer Sciences. 35(2). 272–284. 155 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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