Rosa M. Sancho
- Molecular Biology top 10%
- Neurology top 2%
- Cell Biology top 5%
- Physiology top 10%
- Cellular and Molecular Neuroscience top 10%
- Co-authors
- Christopher C.J. MillerKurt J. De VosSébastien PaillussonRadu StoicaGema Vizcay‐BarrenaChristopher E. ShawJacqueline C. MitchellBernard M. H. Law
- Topics
- Parkinson's Disease Mechanisms and Treatments (3 papers)Endoplasmic Reticulum Stress and Disease (2 papers)Amyotrophic Lateral Sclerosis Research (2 papers)
- Partner nations
- United KingdomUnited StatesCzechia
In The Last Decade
Rosa M. Sancho
10 papers receiving 1.2k citations
Hit Papers
Peers
Comparison fields: 5 of 83
- Molecular Biology 782
- Neurology 507
- Cell Biology 361
- Physiology 261
- Cellular and Molecular Neuroscience 256
Countries citing papers authored by Rosa M. Sancho
This map shows the geographic impact of Rosa M. Sancho's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rosa M. Sancho with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rosa M. Sancho more than expected).
Fields of papers citing papers by Rosa M. Sancho
This network shows the impact of papers produced by Rosa M. Sancho. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rosa M. Sancho. The network helps show where Rosa M. Sancho may publish in the future.
Co-authorship network of co-authors of Rosa M. Sancho
This figure shows the co-authorship network connecting the top 25 collaborators of Rosa M. Sancho. A scholar is included among the top collaborators of Rosa M. Sancho based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rosa M. Sancho. Rosa M. Sancho is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 221 | |
| 3 | 17 | |
| 4 | 1 | |
| 5 | 16 | |
| 6 | 1 | |
| 7 | ER–mitochondria associations are regulated by the VAPB–PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43breakdown → | 497 |
| 8 | 218 | |
| 9 | 1 | |
| 10 | 107 | |
| 11 | 106 |
About Rosa M. Sancho
Rosa M. Sancho is a scholar working on Neurology, Cell Biology and Critical Care and Intensive Care Medicine, having authored 11 papers that have together received 1.2k indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (3 papers), Endoplasmic Reticulum Stress and Disease (2 papers) and Amyotrophic Lateral Sclerosis Research (2 papers). The work is most often cited by research in Neurology (507 citations), Cell Biology (361 citations) and Cellular and Molecular Neuroscience (256 citations). Rosa M. Sancho has collaborated with scholars based in United Kingdom, United States and Czechia. Frequent co-authors include Christopher C.J. Miller, Kurt J. De Vos, Sébastien Paillusson, Radu Stoica, Gema Vizcay‐Barrena, Christopher E. Shaw, Jacqueline C. Mitchell, Bernard M. H. Law, Kirsten Harvey and Leonard Petrucelli. Their work appears in journals such as Journal of Biological Chemistry, Nature Communications and Human Molecular Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.