Ronald G. Haller
- Molecular Biology top 2%
- Rheumatology top 0.5%
- Physiology top 1%
- Clinical Biochemistry top 0.1%
- Cellular and Molecular Neuroscience top 2%
- Co-authors
- John VissingTanja TaivassaloDouglas EbertMarlei WaltonS. F. LewisFanny MochelSteven F. LewisJames P. Knöchel
- Topics
- Glycogen Storage Diseases and Myoclonus (49 papers)Mitochondrial Function and Pathology (41 papers)Metabolism and Genetic Disorders (39 papers)
- Partner nations
- United StatesDenmarkCanada
In The Last Decade
Ronald G. Haller
134 papers receiving 6.2k citations
Peers
Comparison fields: 5 of 130
- Molecular Biology 3.2k
- Rheumatology 1.6k
- Physiology 1.5k
- Clinical Biochemistry 1.4k
- Cellular and Molecular Neuroscience 1.0k
Countries citing papers authored by Ronald G. Haller
This map shows the geographic impact of Ronald G. Haller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald G. Haller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald G. Haller more than expected).
Fields of papers citing papers by Ronald G. Haller
This network shows the impact of papers produced by Ronald G. Haller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald G. Haller. The network helps show where Ronald G. Haller may publish in the future.
Co-authorship network of co-authors of Ronald G. Haller
This figure shows the co-authorship network connecting the top 25 collaborators of Ronald G. Haller. A scholar is included among the top collaborators of Ronald G. Haller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ronald G. Haller. Ronald G. Haller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 30 | |
| 3 | 13 | |
| 4 | 133 | |
| 5 | Myopathy with Deficiency of ISCU | 7 |
| 6 | 16 | |
| 7 | 10 | |
| 8 | 55 | |
| 9 | 89 | |
| 10 | 39 | |
| 11 | 28 | |
| 12 | 1 | |
| 13 | 119 | |
| 14 | 74 | |
| 15 | 40 | |
| 16 | 157 | |
| 17 | 53 | |
| 18 | 68 | |
| 19 | 37 | |
| 20 | Hypophosphatemia and rhabdomyolysis. | 8 |
About Ronald G. Haller
Ronald G. Haller is a scholar working on Clinical Biochemistry, Rheumatology and Cellular and Molecular Neuroscience, having authored 135 papers that have together received 6.3k indexed citations. Recurring topics across this work include Glycogen Storage Diseases and Myoclonus (49 papers), Mitochondrial Function and Pathology (41 papers) and Metabolism and Genetic Disorders (39 papers). The work is most often cited by research in Clinical Biochemistry (1.4k citations), Rheumatology (1.6k citations) and Physiology (1.5k citations). Ronald G. Haller has collaborated with scholars based in United States, Denmark and Canada. Frequent co-authors include John Vissing, Tanja Taivassalo, Douglas Ebert, Marlei Walton, S. F. Lewis, Fanny Mochel, Steven F. Lewis, James P. Knöchel, Phil Wyrick and Tracey A. Rouault. Their work appears in journals such as Science, New England Journal of Medicine and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.