Robert Godemann

1.9k total citations · 1 hit paper
9 papers, 1.4k citations indexed

About

Robert Godemann is a scholar working on Molecular Biology, Physiology and Cell Biology. According to data from OpenAlex, Robert Godemann has authored 9 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 4 papers in Physiology and 3 papers in Cell Biology. Recurrent topics in Robert Godemann's work include Alzheimer's disease research and treatments (4 papers), Protein Structure and Dynamics (3 papers) and Microtubule and mitosis dynamics (3 papers). Robert Godemann is often cited by papers focused on Alzheimer's disease research and treatments (4 papers), Protein Structure and Dynamics (3 papers) and Microtubule and mitosis dynamics (3 papers). Robert Godemann collaborates with scholars based in Germany, United Kingdom and United States. Robert Godemann's co-authors include Eckhard Mandelkow�, Susanne Illenberger, Karsten Stamer, Bernhard Trinczek, Andreas Ebneth, Jacek Biernat, Eva‐Maria Mandelkow, Qingyi Zheng‐Fischhöfer, Ute Preuß and Karlheinz Baumann and has published in prestigious journals such as The Journal of Cell Biology, PLoS ONE and Biochemistry.

In The Last Decade

Robert Godemann

9 papers receiving 1.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Overexpression of Tau Protein Inhibits Kinesin-dependent Trafficking of Vesicles, Mitochondria, and Endoplasmic Reticulum: Implications for Alzheimer's Disease

1998 654 citations Andreas Ebneth, Robert Godemann et al. The Journal of Cell Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert Godemann Germany	7	881	802	414	338	180	9	1.4k
Della David Germany	16	1.0k 1.2×	1.1k 1.4×	400 1.0×	341 1.0×	209 1.2×	22	2.0k
Karsten Stamer Germany	4	1.1k 1.3×	864 1.1×	504 1.2×	491 1.5×	214 1.2×	7	1.7k
Yongjun Gu Canada	14	940 1.1×	907 1.1×	429 1.0×	505 1.5×	257 1.4×	14	1.7k
Qingyi Zheng‐Fischhöfer Germany	9	701 0.8×	696 0.9×	293 0.7×	281 0.8×	129 0.7×	9	1.1k
V.M.-Y. Lee United States	16	1.0k 1.2×	644 0.8×	241 0.6×	336 1.0×	176 1.0×	21	1.5k
Joel M. Litersky United States	11	860 1.0×	725 0.9×	444 1.1×	497 1.5×	164 0.9×	12	1.4k
Khalid Iqbal United States	11	1.1k 1.3×	889 1.1×	333 0.8×	430 1.3×	250 1.4×	11	1.6k
Midori OGAWARA Japan	13	1.2k 1.3×	1.0k 1.3×	405 1.0×	295 0.9×	199 1.1×	30	1.9k
William H. Stoothoff United States	9	732 0.8×	592 0.7×	230 0.6×	351 1.0×	177 1.0×	9	1.3k
K S Kosik United States	11	1.4k 1.5×	1.1k 1.3×	536 1.3×	484 1.4×	250 1.4×	16	2.1k

Countries citing papers authored by Robert Godemann

Since Specialization

Citations

This map shows the geographic impact of Robert Godemann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Godemann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Godemann more than expected).

Fields of papers citing papers by Robert Godemann

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Godemann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Godemann. The network helps show where Robert Godemann may publish in the future.

Co-authorship network of co-authors of Robert Godemann

This figure shows the co-authorship network connecting the top 25 collaborators of Robert Godemann. A scholar is included among the top collaborators of Robert Godemann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert Godemann. Robert Godemann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

9 of 9 papers shown

1.

Feng, Jianwen A., Patrick Lee, Kathy Barrett, et al.. (2019). Structure Based Design of Potent Selective Inhibitors of Protein Kinase D1 (PKD1). ACS Medicinal Chemistry Letters. 10(9). 1260–1265. 5 indexed citations

2.

Madden, J. Patrick, Robert Godemann, M. A. Smith, et al.. (2010). Fragment-based discovery and optimization of BACE1 inhibitors. Bioorganic & Medicinal Chemistry Letters. 20(17). 5329–5333. 37 indexed citations

3.

Law, Richard, Oliver Barker, John J. Barker, et al.. (2009). The multiple roles of computational chemistry in fragment-based drug design. Journal of Computer-Aided Molecular Design. 23(8). 459–473. 45 indexed citations

4.

Willuweit, Antje, Joachim Velden, Robert Godemann, et al.. (2009). Early-Onset and Robust Amyloid Pathology in a New Homozygous Mouse Model of Alzheimer's Disease. PLoS ONE. 4(11). e7931–e7931. 47 indexed citations

5.

Godemann, Robert, J. Patrick Madden, M. A. Smith, et al.. (2009). Fragment-Based Discovery of BACE1 Inhibitors Using Functional Assays. Biochemistry. 48(45). 10743–10751. 1 indexed citations

6.

Godemann, Robert, Jacek Biernat, Eva‐Maria Mandelkow, & Eckhard Mandelkow�. (1999). Phosphorylation of tau protein by recombinant GSK‐3β: pronounced phosphorylation at select Ser/Thr‐Pro motifs but no phosphorylation at Ser262 in the repeat domain. FEBS Letters. 454(1-2). 157–164. 96 indexed citations

7.

Zheng‐Fischhöfer, Qingyi, Jacek Biernat, Eva‐Maria Mandelkow, et al.. (1998). Sequential phosphorylation of Tau by glycogen synthase kinase‐3β and protein kinase A at Thr212 and Ser214 generates the Alzheimer‐specific epitope of antibody AT100 and requires a paired‐helical‐filament‐like conformation. European Journal of Biochemistry. 252(3). 542–552. 275 indexed citations

8.

Ebneth, Andreas, Robert Godemann, Karsten Stamer, et al.. (1998). Overexpression of Tau Protein Inhibits Kinesin-dependent Trafficking of Vesicles, Mitochondria, and Endoplasmic Reticulum: Implications for Alzheimer's Disease. The Journal of Cell Biology. 143(3). 777–794. 654 indexed citations breakdown →

9.

Illenberger, Susanne, Qingyi Zheng‐Fischhöfer, Ute Preuß, et al.. (1998). The Endogenous and Cell Cycle-dependent Phosphorylation of tau Protein in Living Cells: Implications for Alzheimer’s Disease. Molecular Biology of the Cell. 9(6). 1495–1512. 246 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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