Robert A. Copeland

10.3k total citations · 4 hit papers
66 papers, 5.0k citations indexed

About

Robert A. Copeland is a scholar working on Molecular Biology, Hematology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Robert A. Copeland has authored 66 papers receiving a total of 5.0k indexed citations (citations by other indexed papers that have themselves been cited), including 60 papers in Molecular Biology, 7 papers in Hematology and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in Robert A. Copeland's work include Epigenetics and DNA Methylation (39 papers), Cancer-related gene regulation (38 papers) and RNA modifications and cancer (12 papers). Robert A. Copeland is often cited by papers focused on Epigenetics and DNA Methylation (39 papers), Cancer-related gene regulation (38 papers) and RNA modifications and cancer (12 papers). Robert A. Copeland collaborates with scholars based in United States, United Kingdom and Japan. Robert A. Copeland's co-authors include Victoria M. Richon, Margaret Porter Scott, Roy M. Pollock, Sarah K. Knutson, Kevin W. Kuntz, Mikel P. Moyer, Heike Keilhack, Richard Chesworth, Michael E. Solomon and Tim J. Wigle and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Blood.

In The Last Decade

Robert A. Copeland

66 papers receiving 4.8k citations

Hit Papers

Durable tumor regression in genetically altered malignant... 2010 2026 2015 2020 2013 2013 2010 2018 100 200 300 400 500

Peers

Robert A. Copeland
Comparison fields: 5 of 122
  • Molecular Biology 4.3k
  • Oncology 508
  • Pathology and Forensic Medicine 465
  • Hematology 402
  • Cancer Research 355
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Citations per field, relative to Robert A. Copeland
Robert A. Copeland · 1×
Citations per year, relative to Robert A. Copeland
Robert A. Copeland · 1×

Countries citing papers authored by Robert A. Copeland

Since Specialization
Citations

This map shows the geographic impact of Robert A. Copeland's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert A. Copeland with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert A. Copeland more than expected).

Fields of papers citing papers by Robert A. Copeland

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert A. Copeland. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert A. Copeland. The network helps show where Robert A. Copeland may publish in the future.

Co-authorship network of co-authors of Robert A. Copeland

This figure shows the co-authorship network connecting the top 25 collaborators of Robert A. Copeland. A scholar is included among the top collaborators of Robert A. Copeland based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert A. Copeland. Robert A. Copeland is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 2
2 52
3 46
4 127
5 12
6 31
7 96
8 55
9 44
10
Potent inhibition of DOT1L as treatment of MLL-fusion leukemia breakdown →
546
11 116
12 151
13
Coordinated activities of wild-type plus mutant EZH2 drive tumor-associated hypertrimethylation of lysine 27 on histone H3 (H3K27) in human B-cell lymphomas breakdown →
500
14 372
15 7
16 103
17 3
18 7
19 11
20 111

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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