Randal M. Bugianesi

1.4k citations
16 papers · 773 indexed · h-index 15
Topics
Ion channel regulation and function (13 papers)Cardiac electrophysiology and arrhythmias (7 papers)Nicotinic Acetylcholine Receptors Study (7 papers)

In The Last Decade

Randal M. Bugianesi

16 papers receiving 748 citations

Peers

Randal M. Bugianesi
Comparison fields: 5 of 82
  • Molecular Biology 621
  • Cardiology and Cardiovascular Medicine 209
  • Cellular and Molecular Neuroscience 201
  • Genetics 89
  • Surgery 74
Replace Saverio Gentile with:
Saverio Gentile United States
Takaaki Ikemoto Japan
Christian Ried Germany
Núria Villalonga Spain
Ludovic Gillet France
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Michael A. Tones United States
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Citations per field
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Citations per year

Countries citing papers authored by Randal M. Bugianesi

Since Specialization
Citations

This map shows the geographic impact of Randal M. Bugianesi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Randal M. Bugianesi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Randal M. Bugianesi more than expected).

Fields of papers citing papers by Randal M. Bugianesi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Randal M. Bugianesi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Randal M. Bugianesi. The network helps show where Randal M. Bugianesi may publish in the future.

Co-authorship network of co-authors of Randal M. Bugianesi

This figure shows the co-authorship network connecting the top 25 collaborators of Randal M. Bugianesi. A scholar is included among the top collaborators of Randal M. Bugianesi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Randal M. Bugianesi. Randal M. Bugianesi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
#WorkIndexed citations
1 15
2 56
3 43
4 25
5 9
6 33
7 37
8 18
9 83
10 23
11 54
12 88
13 113
14 44
15 38
16 94

About Randal M. Bugianesi

Randal M. Bugianesi is a scholar working on Cardiology and Cardiovascular Medicine, Cellular and Molecular Neuroscience and Molecular Biology, having authored 16 papers that have together received 773 indexed citations. Recurring topics across this work include Ion channel regulation and function (13 papers), Cardiac electrophysiology and arrhythmias (7 papers) and Nicotinic Acetylcholine Receptors Study (7 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (201 citations), Cardiology and Cardiovascular Medicine (209 citations) and Molecular Biology (621 citations). Randal M. Bugianesi has collaborated with scholars based in United States, Austria and Japan. Frequent co-authors include Gregory J. Kaczorowski, María L. García, Robert S. Slaughter, John P. Felix, Hans‐Günther Knaus, Mitree M. Ponpipom, Jörg Mitterdorfer, Alexandra Koschak, Jeffrey Robbins and Mei‐Hwan Wu. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Physiology and Biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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