Alexandra Koschak

1.2k total citations
26 papers, 915 citations indexed

About

Alexandra Koschak is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Alexandra Koschak has authored 26 papers receiving a total of 915 indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 16 papers in Cellular and Molecular Neuroscience and 3 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Alexandra Koschak's work include Retinal Development and Disorders (14 papers), Ion channel regulation and function (11 papers) and Neuroscience and Neuropharmacology Research (9 papers). Alexandra Koschak is often cited by papers focused on Retinal Development and Disorders (14 papers), Ion channel regulation and function (11 papers) and Neuroscience and Neuropharmacology Research (9 papers). Alexandra Koschak collaborates with scholars based in Austria, Germany and Italy. Alexandra Koschak's co-authors include Jörg Striessnig, Jean‐Charles Hoda, Hanno J. Bolz, Anamika Singh, Tina Pangršič, Joshua H. Singer, Mathias Gebhart, Randal M. Bugianesi, Hans‐Günther Knaus and Jörg Mitterdorfer and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Journal of Neuroscience.

In The Last Decade

Alexandra Koschak

25 papers receiving 911 citations

Peers

Alexandra Koschak
Evanna Gleason United States
Alon Meir United Kingdom
Hannah I. Bishop United States
Eva Bosse Germany
Abram Akopian United States
Alexandra Koschak
Citations per year, relative to Alexandra Koschak Alexandra Koschak (= 1×) peers Georg Wietzorrek

Countries citing papers authored by Alexandra Koschak

Since Specialization
Citations

This map shows the geographic impact of Alexandra Koschak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alexandra Koschak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alexandra Koschak more than expected).

Fields of papers citing papers by Alexandra Koschak

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alexandra Koschak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alexandra Koschak. The network helps show where Alexandra Koschak may publish in the future.

Co-authorship network of co-authors of Alexandra Koschak

This figure shows the co-authorship network connecting the top 25 collaborators of Alexandra Koschak. A scholar is included among the top collaborators of Alexandra Koschak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alexandra Koschak. Alexandra Koschak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fernández‐Quintero, Monica L., et al.. (2023). Characterization of two pathological gating-charge substitutions in Cav1.4 L-type calcium channels. Channels. 17(1). 2192360–2192360. 1 indexed citations
2.
Seitter, Hartwig, et al.. (2023). A novel calcium channel Cavβ2 splice variant with unique properties predominates in the retina. Journal of Biological Chemistry. 299(4). 102972–102972.
3.
Babai, Norbert, et al.. (2021). Function of cone and cone-related pathways in CaV1.4 IT mice. Scientific Reports. 11(1). 2732–2732. 7 indexed citations
4.
Koschak, Alexandra, et al.. (2021). Cav1.4 dysfunction and congenital stationary night blindness type 2. Pflügers Archiv - European Journal of Physiology. 473(9). 1437–1454. 14 indexed citations
5.
Pangršič, Tina, Joshua H. Singer, & Alexandra Koschak. (2018). Voltage-Gated Calcium Channels: Key Players in Sensory Coding in the Retina and the Inner Ear. Physiological Reviews. 98(4). 2063–2096. 71 indexed citations
6.
Schwarzer, Christoph, Hartwig Seitter, Friedrich Fresser, et al.. (2018). Protein kinase N1 critically regulates cerebellar development and long-term function. Journal of Clinical Investigation. 128(5). 2076–2088. 12 indexed citations
7.
Seitter, Hartwig & Alexandra Koschak. (2017). Relevance of tissue specific subunit expression in channelopathies. Neuropharmacology. 132. 58–70. 12 indexed citations
8.
Messina, Andrea, Erik Dassi, Giovanni Provenzano, et al.. (2015). A New Splicing Isoform ofCacna2d4Mimicking the Effects of c.2451insC Mutation in the Retina: Novel Molecular and Electrophysiological Insights. Investigative Ophthalmology & Visual Science. 56(8). 4846–4846. 13 indexed citations
9.
Schicker, Klaus, Thomas Stockner, Christof Kugler, et al.. (2014). Spectrum of Cav1.4 dysfunction in congenital stationary night blindness type 2. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1838(8). 2053–2065. 26 indexed citations
10.
Obermair, Gerald J., Ruslan I. Stanika, Valentina Di Biase, et al.. (2014). Differential Neuronal Targeting of a New and Two Known Calcium Channel β4Subunit Splice Variants Correlates with Their Regulation of Gene Expression. Journal of Neuroscience. 34(4). 1446–1461. 35 indexed citations
11.
Kerov, Vasily, Simone B. Sartori, Gerald J. Obermair, et al.. (2013). Cav1.4 IT mouse as model for vision impairment in human congenital stationary night blindness type 2. Channels. 7(6). 503–513. 44 indexed citations
12.
Gebhart, Mathias, Annalisa Zuccotti, Niels Brandt, et al.. (2010). Modulation of Cav1.3 Ca2+ channel gating by Rab3 interacting molecule. Molecular and Cellular Neuroscience. 44(3). 246–259. 41 indexed citations
13.
Koschak, Alexandra. (2010). Impact of gating modulation in CaV1.3 L-type calcium channels. Channels. 4(6). 523–525. 10 indexed citations
14.
Striessnig, Jörg, Hanno J. Bolz, & Alexandra Koschak. (2010). Channelopathies in Cav1.1, Cav1.3, and Cav1.4 voltage-gated L-type Ca2+ channels. Pflügers Archiv - European Journal of Physiology. 460(2). 361–374. 104 indexed citations
15.
Ullrich, Nina D., Alexandra Koschak, & Kenneth T. MacLeod. (2007). Oestrogen directly inhibits the cardiovascular L-type Ca2+ channel Cav1.2. Biochemical and Biophysical Research Communications. 361(2). 522–527. 34 indexed citations
16.
Hoda, Jean‐Charles, et al.. (2006). Effects of congenital stationary night blindness type 2 mutations R508Q and L1364H on Cav1.4 L‐type Ca2+ channel function and expression. Journal of Neurochemistry. 96(6). 1648–1658. 28 indexed citations
17.
Singh, Anamika, Jean‐Charles Hoda, Mathias Gebhart, et al.. (2006). C-terminal modulator controls Ca2+-dependent gating of Cav1.4 L-type Ca2+ channels. Nature Neuroscience. 9(9). 1108–1116. 115 indexed citations
19.
Koschak, Alexandra, Michael Poteser, Martina J. Sinnegger, et al.. (2002). Functional Consequences of P/Q-type Ca2+Channel Cav2.1 Missense Mutations Associated with Episodic Ataxia Type 2 and Progressive Ataxia. Journal of Biological Chemistry. 277(9). 6960–6966. 84 indexed citations
20.
Koschak, Alexandra, Randal M. Bugianesi, Jörg Mitterdorfer, et al.. (1998). Subunit Composition of Brain Voltage-gated Potassium Channels Determined by Hongotoxin-1, a Novel Peptide Derived fromCentruroides limbatus Venom. Journal of Biological Chemistry. 273(5). 2639–2644. 113 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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