Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Allele-specific increase in basal transcription of the plasminogen-activator inhibitor 1 gene is associated with myocardial infarction.
1995603 citationsPer Eriksson, Anders Hamsten et al.profile →
Functional Polymorphism in the Regulatory Region of Gelatinase B Gene in Relation to Severity of Coronary Atherosclerosis
1999507 citationsPer Eriksson, Anders Hamsten et al.profile →
Author Peers
Peers are selected by citation overlap in the author's most active subfields.
citations ·
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This map shows the geographic impact of Per Eriksson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Per Eriksson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Per Eriksson more than expected).
This network shows the impact of papers produced by Per Eriksson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Per Eriksson. The network helps show where Per Eriksson may publish in the future.
Co-authorship network of co-authors of Per Eriksson
This figure shows the co-authorship network connecting the top 25 collaborators of Per Eriksson.
A scholar is included among the top collaborators of Per Eriksson based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Per Eriksson. Per Eriksson is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Liu, Lu, Peter Damberg, Anton Gisterå, et al.. (2020). Molecular Imaging of Inflammation in a Mouse Model of Atherosclerosis Using a Zirconium-89-Labeled Probe. SHILAP Revista de lepidopterología.1 indexed citations
Brokstad, Karl A., Lilian Vasaitis, Marika Kvarnström, et al.. (2015). Comparison between Multiplex and Routine Clinical Immunology Analysis of Antibodies Against SSA and SSB. Scandinavian Journal of Immunology. 81(5). 397–397.1 indexed citations
Nordmark, Gunnel, Elke Theander, Silke Appel, et al.. (2010). Gene Variants In Fam167A-Blk And Tnfsf4 Are Associated With Primary Sjogren'S Syndrome. Scandinavian Journal of Rheumatology. 39. 32–32.1 indexed citations
14.
Eriksson, Per, et al.. (2003). Co-exposure to a polybrominated diphenyl ether (PBDE 99) and an ortho-substituted PCB (PCB 52) enhances developmental neurotoxic effects. Organohalogen compounds. 61. 81–83.8 indexed citations
15.
Viberg, Henrik, et al.. (2003). Neurotoxicity of different polybrominated diphenyl ethers, including PBDE 209. Organohalogen compounds. 65. 9–11.1 indexed citations
16.
Eriksson, Per, et al.. (2002). A comparison on the developmental neurotoxic effects of hexabromocyclododecane, 2,2´,4,4´,5,5´-hexabromodiphenylether (PBDE 153) and 2,2´,4,4´,5,5´-hexachlorobiphenylether (PCB 153).. Organohalogen compounds. 57. 389–390.31 indexed citations
17.
Eriksson, Per. (2000). SKOGFORSK REVIEW OF SYSTEMS FOR LOGGING RESIDUES HANDLING IN SWEDEN. New Zealand journal of forestry science. 30. 88–93.5 indexed citations
18.
Eriksson, Per, Henrik Viberg, Eva Jakobsson, Ulrika Örn, & A Fredriksson. (1999). PBDE, 2,2',4,4'5-pentabromodiphenyl ether causes permanent neurotoxic effects during a defined period of neonatal brain development. Organohalogen compounds. 40. 333.24 indexed citations
19.
Ankarberg, Emma, A Fredriksson, & Per Eriksson. (1998). Interactive effects of PCB and nicotine administered during the neonatal brain development. Organohalogen compounds. 37. 93.3 indexed citations
20.
Eriksson, Per, et al.. (1996). A vldl response element in the promoter region of the human pai-1 gene implicated in the impaired fibrinolysis of hypertriglyceridaemia. Fibrinolysis & proteolysis. 10. 92.2 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.