Henrik Viberg
About
Henrik Viberg is a scholar working on Health, Toxicology and Mutagenesis, Developmental Neuroscience and Cellular and Molecular Neuroscience.
According to data from OpenAlex, Henrik Viberg has authored 59 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Health, Toxicology and Mutagenesis, 15 papers in Developmental Neuroscience and 12 papers in Cellular and Molecular Neuroscience. Recurrent topics in Henrik Viberg's work include Toxic Organic Pollutants Impact (32 papers), Effects and risks of endocrine disrupting chemicals (17 papers) and Anesthesia and Neurotoxicity Research (15 papers). Henrik Viberg is often cited by papers focused on Toxic Organic Pollutants Impact (32 papers), Effects and risks of endocrine disrupting chemicals (17 papers) and Anesthesia and Neurotoxicity Research (15 papers). Henrik Viberg collaborates with scholars based in Sweden, United States and Netherlands. Henrik Viberg's co-authors include Per Eriksson, Anders Fredriksson, Niclas Johansson, Iwa Lee, Torsten Gordh, Per Eriksson, A Fredriksson, Eva Jakobsson, Ulrika Örn and Sonja Buratovic and has published in prestigious journals such as Geophysical Research Letters, Environmental Health Perspectives and Behavioural Brain Research.
In The Last Decade
Henrik Viberg
58 papers receiving 3.2k citations
Peers
Henrik Viberg
Susan L. Schantz United States
Per Eriksson Sweden
Prasada Rao S. Kodavanti United States
Richard F. Seegal United States
Espen Mariussen Norway
Gennaro Giordano United States
Hellmuth Lilienthal Germany
María Teresa Colomina Spain
Sharon K. Sagiv United States
Ole Ladefoged Denmark
Citations per year, relative to Henrik Viberg Henrik Viberg (= 1×)
peers
Susan L. Schantz
Countries citing papers authored by Henrik Viberg
Since
Specialization
Citations
This map shows the geographic impact of Henrik Viberg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Henrik Viberg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Henrik Viberg more than expected).
Fields of papers citing papers by Henrik Viberg
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Henrik Viberg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Henrik Viberg. The network helps show where Henrik Viberg may publish in the future.
Co-authorship network of co-authors of Henrik Viberg
This figure shows the co-authorship network connecting the top 25 collaborators of Henrik Viberg. A scholar is included among the top collaborators of Henrik Viberg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Henrik Viberg. Henrik Viberg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Eriksson, Per, Niclas Johansson, Henrik Viberg, Sonja Buratovic, & Anders Fredriksson. (2022). Perfluorinated chemicals (PFOA) can, by interacting with highly brominated diphenyl ethers (PBDE 209) during a defined period of neonatal brain development, exacerbate neurobehavioural defects. Neurotoxicology and Teratology. 96. 107150–107150.
2.
Buratovic, Sonja, Bo Stenerlöw, Anders Fredriksson, et al.. (2018). Effects on adult cognitive function after neonatal exposure to clinically relevant doses of ionising radiation and ketamine in mice. British Journal of Anaesthesia. 120(3). 546–554.
3.
Lee, Iwa, et al.. (2015). Developmental neurotoxic effects of two pesticides: Behavior and neuroprotein studies on endosulfan and cypermethrin. Toxicology. 335. 1–10.
4.
Hallgren, Stefan & Henrik Viberg. (2015). Postnatal exposure to PFOS, but not PBDE 99, disturb dopaminergic gene transcription in the mouse CNS. Environmental Toxicology and Pharmacology. 41. 121–126.
5.
Lee, Iwa, Per Eriksson, Anders Fredriksson, Sonja Buratovic, & Henrik Viberg. (2015). Developmental neurotoxic effects of two pesticides: Behavior and biomolecular studies on chlorpyrifos and carbaryl. Toxicology and Applied Pharmacology. 288(3). 429–438.
6.
Hendriks, Hester S., Milou M.L. Dingemans, Henrik Viberg, et al.. (2014). Effects of neonatal exposure to the flame retardant tetrabromobisphenol-A, aluminum diethylphosphinate or zinc stannate on long-term potentiation and synaptic protein levels in mice. Archives of Toxicology. 89(12). 2345–2354.
7.
Viberg, Henrik, Per Eriksson, Torsten Gordh, & Anders Fredriksson. (2013). Paracetamol (Acetaminophen) Administration During Neonatal Brain Development Affects Cognitive Function and Alters Its Analgesic and Anxiolytic Response in Adult Male Mice. Toxicological Sciences. 138(1). 139–147.
8.
Lee, Iwa & Henrik Viberg. (2013). A single neonatal exposure to perfluorohexane sulfonate (PFHxS) affects the levels of important neuroproteins in the developing mouse brain. NeuroToxicology. 37. 190–196.
9.
Viberg, Henrik, Iwa Lee, & Per Eriksson. (2012). Adult dose-dependent behavioral and cognitive disturbances after a single neonatal PFHxS dose. Toxicology. 304. 185–191.
10.
Viberg, Henrik & Iwa Lee. (2012). A single exposure to bisphenol A alters the levels of important neuroproteins in adult male and female mice. NeuroToxicology. 33(5). 1390–1395.
11.
Johansson, Niclas, Per Eriksson, & Henrik Viberg. (2009). Neonatal Exposure to PFOS and PFOA in Mice Results in Changes in Proteins which are Important for Neuronal Growth and Synaptogenesis in the Developing Brain. Toxicological Sciences. 108(2). 412–418.
12.
Viberg, Henrik. (2009). Exposure to Polybrominated Diphenyl Ethers 203 and 206 during the Neonatal Brain Growth Spurt Affects Proteins Important for Normal Neurodevelopment in Mice. Toxicological Sciences. 109(2). 306–311.
13.
Johansson, Niclas, Per Eriksson, & Henrik Viberg. (2008). Neonatal exposure to perfluorooctane sulfonate (PFOS) and perfluorooctanoic acid (PFOA) affect developmental marker proteins, CaMKII, GAP-43, and synaptophysin levels in the mouse brain. Organohalogen compounds. 70. 749–751.
14.
Johansson, Niclas, Henrik Viberg, Anders Fredriksson, & Per Eriksson. (2008). Neonatal exposure to deca-brominated diphenyl ether (PBDE 209) causes dose–response changes in spontaneous behaviour and cholinergic susceptibility in adult mice. NeuroToxicology. 29(6). 911–919.
15.
Viberg, Henrik, Emma Pontén, Per Eriksson, Torsten Gordh, & Anders Fredriksson. (2008). Neonatal ketamine exposure results in changes in biochemical substrates of neuronal growth and synaptogenesis, and alters adult behavior irreversibly. Toxicology. 249(2-3). 153–159.
16.
Viberg, Henrik, et al.. (2003). Neurotoxicity of different polybrominated diphenyl ethers, including PBDE 209. Organohalogen compounds. 65. 9–11.
17.
Viberg, Henrik. (2003). Neonatal exposure to polybrominated diphenyl ether (PBDE 153) disrupts spontaneous behaviour, impairs learning and memory, and decreases hippocampal cholinergic receptors in adult mice. Toxicology and Applied Pharmacology. 192(2). 95–106.
18.
Eriksson, Per, et al.. (2002). A comparison on the developmental neurotoxic effects of hexabromocyclododecane, 2,2´,4,4´,5,5´-hexabromodiphenylether (PBDE 153) and 2,2´,4,4´,5,5´-hexachlorobiphenylether (PCB 153).. Organohalogen compounds. 57. 389–390.
19.
Eriksson, Per, Emma Ankarberg, Henrik Viberg, & Anders Fredriksson. (2001). The developing cholinergic system as target for environmental toxicants, nicotine and polychlorinated biphenyls (PCBs): Implications for neurotoxicological processes in mice. Neurotoxicity Research. 3(1). 37–51.
20.
Eriksson, Per, Henrik Viberg, Eva Jakobsson, Ulrika Örn, & A Fredriksson. (1999). PBDE, 2,2',4,4'5-pentabromodiphenyl ether causes permanent neurotoxic effects during a defined period of neonatal brain development. Organohalogen compounds. 40. 333.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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