A Fredriksson

400 total citations
17 papers, 338 citations indexed

About

A Fredriksson is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, A Fredriksson has authored 17 papers receiving a total of 338 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Molecular Biology, 3 papers in Cellular and Molecular Neuroscience and 3 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in A Fredriksson's work include Neurotransmitter Receptor Influence on Behavior (3 papers), Anesthesia and Neurotoxicity Research (2 papers) and Metabolism and Genetic Disorders (2 papers). A Fredriksson is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (3 papers), Anesthesia and Neurotoxicity Research (2 papers) and Metabolism and Genetic Disorders (2 papers). A Fredriksson collaborates with scholars based in Sweden, Brazil and United Kingdom. A Fredriksson's co-authors include Henrik Viberg, Per Eriksson, Per Eriksson, Trevor Archer, Trevor Archer, Iván Izquierdo, Nadja Schröder, Eva Jakobsson, Ulrika Örn and Svante B. Ross and has published in prestigious journals such as British Journal of Pharmacology, European Journal of Pharmacology and Toxicological Sciences.

In The Last Decade

A Fredriksson

16 papers receiving 324 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
A Fredriksson Sweden 8 188 91 56 51 38 17 338
Henrik Alm Sweden 9 133 0.7× 105 1.2× 37 0.7× 48 0.9× 92 2.4× 11 479
Tultul Nayyar United States 11 225 1.2× 114 1.3× 72 1.3× 76 1.5× 92 2.4× 25 508
Sonja Buratovic Sweden 11 119 0.6× 50 0.5× 53 0.9× 19 0.4× 125 3.3× 19 485
Emma Solà Spain 13 92 0.5× 35 0.4× 23 0.4× 36 0.7× 67 1.8× 31 310
Samir N. Kelada United States 9 143 0.8× 99 1.1× 64 1.1× 106 2.1× 192 5.1× 10 529
Chen-Ping Huang China 11 55 0.3× 50 0.5× 33 0.6× 118 2.3× 121 3.2× 13 429
J D Wang China 3 181 1.0× 57 0.6× 13 0.2× 77 1.5× 20 0.5× 7 358
Jingli Liu China 11 289 1.5× 33 0.4× 47 0.8× 11 0.2× 63 1.7× 18 463
Rima Woods United States 6 120 0.6× 32 0.4× 34 0.6× 104 2.0× 223 5.9× 6 471
Min Heui Yoo South Korea 8 52 0.3× 54 0.6× 21 0.4× 53 1.0× 108 2.8× 17 329

Countries citing papers authored by A Fredriksson

Since Specialization
Citations

This map shows the geographic impact of A Fredriksson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A Fredriksson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A Fredriksson more than expected).

Fields of papers citing papers by A Fredriksson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A Fredriksson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A Fredriksson. The network helps show where A Fredriksson may publish in the future.

Co-authorship network of co-authors of A Fredriksson

This figure shows the co-authorship network connecting the top 25 collaborators of A Fredriksson. A scholar is included among the top collaborators of A Fredriksson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A Fredriksson. A Fredriksson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Fredriksson, A, et al.. (2016). Etomidate exposure in early infant mice (P10) does not induce apoptosis or affect behaviour. Acta Anaesthesiologica Scandinavica. 60(5). 588–596. 8 indexed citations
3.
Fredriksson, A, et al.. (2006). Postnatal iron overload destroys NA-DA functional interactions. Journal of Neural Transmission. 114(2). 195–203. 7 indexed citations
4.
Fredriksson, A, Per Eriksson, & Trevor Archer. (2005). Postnatal iron-induced motor behaviour alterations following chronic neuroleptic administration in mice. Journal of Neural Transmission. 113(2). 137–150. 4 indexed citations
5.
Fredriksson, A, et al.. (2005). Influence of noradrenaline denervation on MPTP-induced deficits in mice. Journal of Neural Transmission. 113(9). 1119–1129. 35 indexed citations
6.
Viberg, Henrik, A Fredriksson, & Per Eriksson. (2002). Neonatal Exposure to the Brominated Flame Retardant 2,2`,4,4`,5-Pentabromodiphenyl Ether Causes Altered Susceptibility in the Cholinergic Transmitter System in the Adult Mouse. Toxicological Sciences. 67(1). 104–107. 164 indexed citations
7.
Fredriksson, A, Nadja Schröder, Per Eriksson, Iván Izquierdo, & Trevor Archer. (2001). Neonatal iron potentiates adult MPTP-induced neurodegenerative and functional deficits. Parkinsonism & Related Disorders. 7(2). 97–105. 39 indexed citations
8.
Archer, Trevor & A Fredriksson. (2000). Restoration and putative protectionof MPTP-induced functional deficits. In: Palomo T, Beninger RJ, Archer T (eds). Neurodegerative Brain Disorders. 1 indexed citations
9.
Eriksson, Per, Henrik Viberg, Eva Jakobsson, Ulrika Örn, & A Fredriksson. (1999). PBDE, 2,2',4,4'5-pentabromodiphenyl ether causes permanent neurotoxic effects during a defined period of neonatal brain development. Organohalogen compounds. 40. 333. 24 indexed citations
10.
Ankarberg, Emma, A Fredriksson, & Per Eriksson. (1998). Interactive effects of PCB and nicotine administered during the neonatal brain development. Organohalogen compounds. 37. 93. 3 indexed citations
11.
Archer, Trevor & A Fredriksson. (1998). Effects of acute/chronic, subthreshold/threshold doses of L-Dopa in MPTP-treated mice: In:T. Palomo, R.J.Beninger a T. Archer (eds) Interactive monoaminergic Basis of Brain Disorders.. 5 indexed citations
12.
Fredriksson, A & Trevor Archer. (1998). Effects of acute/chronic, subthreshold/threshold doses of L-Dopa in MPTP-treated mice: II Effects of MAO/COMT inhibitors. In: T.Palomo, R.J.Beninger a T.Archer (eds( Interactive monoaminergic Basis of. 1 indexed citations
13.
Eriksson, Per, Emma Ankarberg, & A Fredriksson. (1996). 82 Exposure to nicotine during a defi‐ ned period in neonatal life induces permanent changes in brain nicotinic receptors and response to nicotine in adult mice. International Journal of Developmental Neuroscience. 14(S1). 71–71. 1 indexed citations
14.
Fredriksson, A, et al.. (1996). Effects of dopamine agonists upon locomotor function in denervatedmice and rats: Comparison with cabergoline. In: Palomo T, Archer T, Beninger RJ, editors. Dopamine disease states.. 1 indexed citations
15.
Fredriksson, A, et al.. (1992). BEHAVIOURAL EFFECTS OF NEONATAL METALLIC MERCURY EXPOSURE IN RATS. Behavioural Pharmacology. 3(Supplement). 86–86. 3 indexed citations
16.
Archer, Trevor, et al.. (1986). The inhibition of the cage-leaving response?A model for studies of the serotonergic neurotransmission in the rat. Journal of Neural Transmission. 65(3-4). 193–210. 19 indexed citations
17.
Archer, Trevor, Christopher J. Fowler, A Fredriksson, et al.. (1985). Increased total activity in the rat after L‐tryptophan plus the monoamine oxidase‐A inhibitor amiflamine but not after L‐tryptophan plus clorgyline. British Journal of Pharmacology. 85(3). 581–590. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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