Pablo J. Sáez

4.3k total citations
44 papers, 2.6k citations indexed

About

Pablo J. Sáez is a scholar working on Molecular Biology, Cell Biology and Physiology. According to data from OpenAlex, Pablo J. Sáez has authored 44 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 13 papers in Cell Biology and 10 papers in Physiology. Recurrent topics in Pablo J. Sáez's work include Connexins and lens biology (13 papers), Cellular Mechanics and Interactions (8 papers) and Adenosine and Purinergic Signaling (6 papers). Pablo J. Sáez is often cited by papers focused on Connexins and lens biology (13 papers), Cellular Mechanics and Interactions (8 papers) and Adenosine and Purinergic Signaling (6 papers). Pablo J. Sáez collaborates with scholars based in Chile, France and United States. Pablo J. Sáez's co-authors include Juan C. Sáez, Kenji F. Shoji, Pascal Ezan, Christian Giaume, Nicolás Palacios‐Prado, Mauricio A. Retamal, Juan Orellana, Nicolas Froger, Pablo Vargas and Jean X. Jiang and has published in prestigious journals such as Nature Communications, Journal of Neuroscience and Blood.

In The Last Decade

Pablo J. Sáez

43 papers receiving 2.5k citations

Peers

Pablo J. Sáez

PHYSI

IMMUN

CMN

Susanne Feil Germany

Ingo Kurth Germany

François Natt Switzerland

Patricia E. Martin United Kingdom

Pilar Martínez‐Martínez Netherlands

Richard Rivera United States

Kenji Kontani Japan

Sharon L. Milgram United States

Robert S. Ames United States

Stuart M. Cahalan United States

Susanne Feil Germany

Pablo J. Sáez

2.4k ×1.6

806 ×1.5

PHYSI

671 ×1.9

IMMUN

263 ×0.8

681 ×2.4

CMN

Citations per year, relative to Pablo J. Sáez Pablo J. Sáez (= 1×) peers Susanne Feil

Countries citing papers authored by Pablo J. Sáez

Since Specialization

Citations

This map shows the geographic impact of Pablo J. Sáez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pablo J. Sáez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pablo J. Sáez more than expected).

Fields of papers citing papers by Pablo J. Sáez

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pablo J. Sáez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pablo J. Sáez. The network helps show where Pablo J. Sáez may publish in the future.

Co-authorship network of co-authors of Pablo J. Sáez

This figure shows the co-authorship network connecting the top 25 collaborators of Pablo J. Sáez. A scholar is included among the top collaborators of Pablo J. Sáez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pablo J. Sáez. Pablo J. Sáez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Sáez, Pablo J., et al.. (2025). Fluorescent probes to track complex membrane blebbing. Biosensors and Bioelectronics X. 23. 100584–100584.

Monzo, Pascale, et al.. (2024). Emergent seesaw oscillations during cellular directional decision-making. Nature Physics. 20(3). 501–511. 7 indexed citations

Zhovmer, Alexander S., Pablo J. Sáez, Xuefei Ma, et al.. (2024). Septins provide microenvironment sensing and cortical actomyosin partitioning in motile amoeboid T lymphocytes. Science Advances. 10(1). eadi1788–eadi1788. 8 indexed citations

Woo, Marcel S., Friederike Ufer, Jana K. Sonner, et al.. (2023). Calcium channel β3 subunit regulates ATP-dependent migration of dendritic cells. Science Advances. 9(38). eadh1653–eadh1653. 6 indexed citations

Levernier, Nicolas, Mabel San Roman, Pablo J. Sáez, et al.. (2021). Pinching the cortex of live cells reveals thickness instabilities caused by myosin II motors. Science Advances. 7(27). 16 indexed citations

ARCOS, Juan Manuel GARCIA, Mathieu Deygas, G. Nader, et al.. (2019). Reconstitution of cell migration at a glance. Journal of Cell Science. 132(4). 19 indexed citations

Kumari, Anita, Pablo J. Sáez, Mathieu Maurin, et al.. (2019). Actomyosin-driven force patterning controls endocytosis at the immune synapse. Nature Communications. 10(1). 2870–2870. 39 indexed citations

Cadart, Clotilde, Sylvain Monnier, Jacopo Grilli, et al.. (2018). Size control in mammalian cells involves modulation of both growth rate and cell cycle duration. Nature Communications. 9(1). 3275–3275. 153 indexed citations

Sáez, Pablo J., Juan C. Sáez, Ana‐Maria Lennon‐Duménil, & Pablo Vargas. (2018). Role of calcium permeable channels in dendritic cell migration. Current Opinion in Immunology. 52. 74–80. 19 indexed citations

10.

Villalobos-Labra, Roberto, Pablo J. Sáez, Mario Subiabre, et al.. (2018). Pre-pregnancy maternal obesity associates with endoplasmic reticulum stress in human umbilical vein endothelium. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1864(10). 3195–3210. 31 indexed citations

11.

Sáez, Pablo J., Pablo Vargas, Kenji F. Shoji, et al.. (2017). ATP promotes the fast migration of dendritic cells through the activity of pannexin 1 channels and P2X ₇ receptors. Science Signaling. 10(506). 130 indexed citations

12.

Bretou, Marine, Pablo J. Sáez, Doriane Sanséau, et al.. (2017). Lysosome signaling controls the migration of dendritic cells. Science Immunology. 2(16). 112 indexed citations

13.

Vargas, Pablo, et al.. (2017). Mechanisms for fast cell migration in complex environments. Current Opinion in Cell Biology. 48. 72–78. 34 indexed citations

14.

Obino, Dorian, Jheimmy Diaz, Jorge Ibañez-Vega, et al.. (2017). Vamp-7–dependent secretion at the immune synapse regulates antigen extraction and presentation in B-lymphocytes. Molecular Biology of the Cell. 28(7). 890–897. 22 indexed citations

15.

Balboa, Elisa, Juan Francisco Castro, Gonzalo I. Cancino, et al.. (2017). MLN64 induces mitochondrial dysfunction associated with increased mitochondrial cholesterol content. Redox Biology. 12. 274–284. 66 indexed citations

16.

Obino, Dorian, Francesca Farina, Odile Malbec, et al.. (2016). Actin nucleation at the centrosome controls lymphocyte polarity. Nature Communications. 7(1). 10969–10969. 99 indexed citations

17.

Westermeier, Francisco, Pablo J. Sáez, Roberto Villalobos-Labra, Luis Sobrevía, & Marcelo Farías. (2014). Programming of Fetal Insulin Resistance in Pregnancies with Maternal Obesity by ER Stress and Inflammation. BioMed Research International. 2014. 1–13. 48 indexed citations

18.

Shoji, Kenji F., Pablo J. Sáez, Paloma A. Harcha, Héctor L. Aguila, & Juan C. Sáez. (2014). Pannexin1 channels act downstream of P2X₇receptors in ATP-induced murine T-cell death. Channels. 8(2). 142–156. 44 indexed citations

19.

Orellana, Juan, Kenji F. Shoji, Verónica Abudara, et al.. (2011). Amyloid β-Induced Death in Neurons Involves Glial and Neuronal Hemichannels. Journal of Neuroscience. 31(13). 4962–4977. 248 indexed citations

20.

Mendoza‐Naranjo, Ariadna, Pablo J. Sáez, C. Christian Johansson, et al.. (2007). Functional Gap Junctions Facilitate Melanoma Antigen Transfer and Cross-Presentation between Human Dendritic Cells. The Journal of Immunology. 178(11). 6949–6957. 88 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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