P. Thomas

721 total citations
7 papers, 571 citations indexed

About

P. Thomas is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Pharmacology. According to data from OpenAlex, P. Thomas has authored 7 papers receiving a total of 571 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Cellular and Molecular Neuroscience, 4 papers in Molecular Biology and 1 paper in Pharmacology. Recurrent topics in P. Thomas's work include Genetic Neurodegenerative Diseases (5 papers), Mitochondrial Function and Pathology (3 papers) and Muscle Physiology and Disorders (2 papers). P. Thomas is often cited by papers focused on Genetic Neurodegenerative Diseases (5 papers), Mitochondrial Function and Pathology (3 papers) and Muscle Physiology and Disorders (2 papers). P. Thomas collaborates with scholars based in United States and United Kingdom. P. Thomas's co-authors include J. W. Neal, Jonathan M. Boutell, James A. Duce, Victoria Weston, P S Harper, A. Lesley Jones, Bryce L. Sopher, Albert R. La Spada, Ida E. Holm and Carol B. Ware and has published in prestigious journals such as Neuron, Human Molecular Genetics and The Journals of Gerontology Series A.

In The Last Decade

P. Thomas

7 papers receiving 557 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
P. Thomas United States	6	468	450	138	95	45	7	571
Isabel Onofre Portugal	10	390 0.8×	380 0.8×	148 1.1×	35 0.4×	57 1.3×	11	557
Jeannette Hübener‐Schmid Germany	15	557 1.2×	598 1.3×	251 1.8×	85 0.9×	51 1.1×	30	813
Davina J. Hensman Moss United Kingdom	9	348 0.7×	351 0.8×	295 2.1×	107 1.1×	47 1.0×	13	596
Kimberly Schadt United States	12	360 0.8×	284 0.6×	120 0.9×	83 0.9×	16 0.4×	23	431
Angela Marsili Italy	13	281 0.6×	245 0.5×	192 1.4×	38 0.4×	18 0.4×	19	486
Andrej Michalik Belgium	8	370 0.8×	366 0.8×	84 0.6×	21 0.2×	30 0.7×	11	463
José Leal Loureiro Portugal	12	364 0.8×	589 1.3×	226 1.6×	110 1.2×	54 1.2×	17	651
Elena D. Markova Russia	14	320 0.7×	318 0.7×	258 1.9×	31 0.3×	51 1.1×	28	560
Katherine A. Dick United States	5	479 1.0×	463 1.0×	158 1.1×	29 0.3×	44 1.0×	5	606
Stefania Magri Italy	13	296 0.6×	271 0.6×	115 0.8×	35 0.4×	71 1.6×	33	477

Countries citing papers authored by P. Thomas

Since Specialization

Citations

This map shows the geographic impact of P. Thomas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by P. Thomas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites P. Thomas more than expected).

Fields of papers citing papers by P. Thomas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by P. Thomas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by P. Thomas. The network helps show where P. Thomas may publish in the future.

Co-authorship network of co-authors of P. Thomas

This figure shows the co-authorship network connecting the top 25 collaborators of P. Thomas. A scholar is included among the top collaborators of P. Thomas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with P. Thomas. P. Thomas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Thomas, P., Catherine J. Spellicy, Mark J. Harding, et al.. (2021). The OPRD1 rs678849 variant influences outcome of disulfiram treatment for cocaine dependency in methadone-maintained patients. Psychiatric Genetics. 31(3). 88–94. 5 indexed citations

2.

Thomas, P., Gregory S. Fraley, Francisco Zapata, et al.. (2006). Loss of endogenous androgen receptor protein accelerates motor neuron degeneration and accentuates androgen insensitivity in a mouse model of X-linked spinal and bulbar muscular atrophy. Human Molecular Genetics. 15(14). 2225–2238. 84 indexed citations

3.

Thomas, P., Gregory S. Fraley, Vincent Damian, et al.. (2006). Loss of endogenous androgen receptor protein accelerates motor neuron degeneration and accentuates androgen insensitivity in a mouse model of X-linked spinal and bulbar muscular atrophy. Human Molecular Genetics. 15(19). 2972–2972. 6 indexed citations

4.

Sopher, Bryce L., P. Thomas, Michelle LaFevre-Bernt, et al.. (2004). Androgen Receptor YAC Transgenic Mice Recapitulate SBMA Motor Neuronopathy and Implicate VEGF164 in the Motor Neuron Degeneration. Neuron. 41(5). 687–699. 144 indexed citations

5.

Oeltgen, Peter R., et al.. (2004). Alternative Strategy for Stress Tolerance: Opioids. The Journals of Gerontology Series A. 59(5). B433–B440. 19 indexed citations

6.

Boutell, Jonathan M., P. Thomas, J. W. Neal, et al.. (1999). Aberrant Interactions of Transcriptional Repressor Proteins with the Huntington's Disease Gene Product, Huntingtin. Human Molecular Genetics. 8(9). 1647–1655. 248 indexed citations

7.

Singhrao, Sim K., P. Thomas, J.D. Wood, et al.. (1998). Huntingtin Protein Colocalizes with Lesions of Neurodegenerative Diseases: An Investigation in Huntington's, Alzheimer's, and Pick's Diseases. Experimental Neurology. 150(2). 213–222. 65 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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