Lee‐Way Jin

4.9k total citations · 1 hit paper
37 papers, 2.6k citations indexed

About

Lee‐Way Jin is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Lee‐Way Jin has authored 37 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Physiology, 19 papers in Molecular Biology and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Lee‐Way Jin's work include Alzheimer's disease research and treatments (22 papers), Mitochondrial Function and Pathology (4 papers) and Genetics and Neurodevelopmental Disorders (4 papers). Lee‐Way Jin is often cited by papers focused on Alzheimer's disease research and treatments (22 papers), Mitochondrial Function and Pathology (4 papers) and Genetics and Neurodevelopmental Disorders (4 papers). Lee‐Way Jin collaborates with scholars based in United States, Taiwan and Hungary. Lee‐Way Jin's co-authors include Izumi Maezawa, Frank R. Sharp, Xinhua Zhan, Boryana Stamova, Brett S. Phinney, Charles DeCarli, David G. Cook, James B. Leverenz, Thomas D. Bird and Inez Vincent and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Neuron.

In The Last Decade

Lee‐Way Jin

36 papers receiving 2.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Gram-negative bacterial molecules associate with Alzheimer disease pathology

2016 412 citations Xinhua Zhan, Boryana Stamova et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Lee‐Way Jin United States	23	1.4k	1.3k	423	382	309	37	2.6k
Dan Liu China	31	1.2k 0.9×	1.7k 1.3×	479 1.1×	633 1.7×	195 0.6×	80	3.6k
Sofie Lautrup Norway	15	1.3k 0.9×	1.7k 1.3×	562 1.3×	275 0.7×	376 1.2×	25	3.7k
Inés Moreno‐González United States	30	1.7k 1.2×	1.2k 0.9×	944 2.2×	607 1.6×	367 1.2×	57	3.2k
Sung Min Son South Korea	26	1.0k 0.8×	1.1k 0.9×	437 1.0×	309 0.8×	231 0.7×	41	2.6k
Dan Frenkel Israel	33	1.6k 1.2×	1.2k 1.0×	1.0k 2.4×	435 1.1×	234 0.8×	65	3.8k
Marc Gleichmann United States	27	1.2k 0.9×	2.0k 1.5×	580 1.4×	909 2.4×	341 1.1×	40	3.9k
Mireille Bélanger Canada	17	930 0.7×	1.3k 1.0×	926 2.2×	890 2.3×	355 1.1×	17	3.4k
Bin Ji Japan	27	1.1k 0.8×	783 0.6×	624 1.5×	540 1.4×	219 0.7×	82	2.5k
Seong Su Kang United States	30	1.1k 0.8×	1.1k 0.8×	501 1.2×	886 2.3×	552 1.8×	49	2.8k
Henrietta M. Nielsen Sweden	28	1.5k 1.1×	801 0.6×	1.0k 2.4×	423 1.1×	595 1.9×	45	2.8k

Countries citing papers authored by Lee‐Way Jin

Since Specialization

Citations

This map shows the geographic impact of Lee‐Way Jin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lee‐Way Jin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lee‐Way Jin more than expected).

Fields of papers citing papers by Lee‐Way Jin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lee‐Way Jin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lee‐Way Jin. The network helps show where Lee‐Way Jin may publish in the future.

Co-authorship network of co-authors of Lee‐Way Jin

This figure shows the co-authorship network connecting the top 25 collaborators of Lee‐Way Jin. A scholar is included among the top collaborators of Lee‐Way Jin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lee‐Way Jin. Lee‐Way Jin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Taha, Ameer Y., Qing Shen, Yurika Otoki, et al.. (2025). Air pollution and Alzheimer disease phenotype deplete esterified proresolving lipid mediator reserves in the brain. JCI Insight. 10(15).

Otoki, Yurika, Shunji Kato, Kiyotaka Nakagawa, et al.. (2021). Lipidomic Analysis of Postmortem Prefrontal Cortex Phospholipids Reveals Changes in Choline Plasmalogen Containing Docosahexaenoic Acid and Stearic Acid Between Cases With and Without Alzheimer’s Disease. NeuroMolecular Medicine. 23(1). 161–175. 21 indexed citations

Chang, Ching‐Pang, Thi Nguyen, Huimei Chen, et al.. (2021). Equilibrative nucleoside transporter 1 inhibition rescues energy dysfunction and pathology in a model of tauopathy. Acta Neuropathologica Communications. 9(1). 112–112. 14 indexed citations

Shih, Yao‐Hsiang, et al.. (2020). TDP-43 interacts with amyloid-β, inhibits fibrillization, and worsens pathology in a model of Alzheimer’s disease. Nature Communications. 11(1). 5950–5950. 69 indexed citations

Fagiolini, Michela, Annarita Patrizi, Jocelyn J. LeBlanc, et al.. (2020). Intellectual and Developmental Disabilities Research Centers: A Multidisciplinary Approach to Understand the Pathogenesis of Methyl-CpG Binding Protein 2-related Disorders. Neuroscience. 445. 190–206. 9 indexed citations

Her, Guor Mour, Yun‐Wen Chen, Pei‐Yi Wu, et al.. (2017). ErbB2 regulates autophagic flux to modulate the proteostasis of APP-CTFs in Alzheimer’s disease. Proceedings of the National Academy of Sciences. 114(15). E3129–E3138. 66 indexed citations

Chen, Chao-Yin, Jacopo Di Lucente, Cheng‐Chang Lien, et al.. (2017). Defective GABAergic neurotransmission in the nucleus tractus solitarius in Mecp2-null mice, a model of Rett syndrome. Neurobiology of Disease. 109(Pt A). 25–32. 22 indexed citations

Horiuchi, Makoto, et al.. (2016). CX3CR1 ablation ameliorates motor and respiratory dysfunctions and improves survival of a Rett syndrome mouse model. Brain Behavior and Immunity. 60. 106–116. 31 indexed citations

Jin, Lee‐Way, Makoto Horiuchi, Heike Wulff, et al.. (2015). Dysregulation of Glutamine Transporter SNAT1 in Rett Syndrome Microglia: A Mechanism for Mitochondrial Dysfunction and Neurotoxicity. Journal of Neuroscience. 35(6). 2516–2529. 73 indexed citations

10.

Altman, Robin, Sonny Ly, Jitka Petrlová, et al.. (2015). Protective spin-labeled fluorenes maintain amyloid beta peptide in small oligomers and limit transitions in secondary structure. Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics. 1854(12). 1860–1870. 8 indexed citations

11.

Bai, Zhouxian, Boryana Stamova, Huichun Xu, et al.. (2014). Distinctive RNA Expression Profiles in Blood Associated With Alzheimer Disease After Accounting for White Matter Hyperintensities. Alzheimer Disease & Associated Disorders. 28(3). 226–233. 40 indexed citations

12.

Hong, Hyun-Seok, Izumi Maezawa, Madhu S. Budamagunta, et al.. (2008). Candidate anti-Aβ fluorene compounds selected from analogs of amyloid imaging agents. Neurobiology of Aging. 31(10). 1690–1699. 23 indexed citations

13.

Ramos, Erin M., Eric B. Larson, Izumi Maezawa, et al.. (2006). Tumor Necrosis Factor α and Interleukin 10 Promoter Region Polymorphisms and Risk of Late-Onset Alzheimer Disease. Archives of Neurology. 63(8). 1165–1165. 70 indexed citations

14.

Jin, Lee‐Way, et al.. (2005). Polarimetric imaging of amyloid. Micron. 37(4). 324–338. 19 indexed citations

15.

Sopher, Bryce L., P. Thomas, Michelle LaFevre-Bernt, et al.. (2004). Androgen Receptor YAC Transgenic Mice Recapitulate SBMA Motor Neuronopathy and Implicate VEGF164 in the Motor Neuron Degeneration. Neuron. 41(5). 687–699. 144 indexed citations

16.

Jin, Lee‐Way, Izumi Maezawa, Inez Vincent, & Thomas D. Bird. (2004). Intracellular Accumulation of Amyloidogenic Fragments of Amyloid-β Precursor Protein in Neurons with Niemann-Pick Type C Defects Is Associated with Endosomal Abnormalities. American Journal Of Pathology. 164(3). 975–985. 180 indexed citations

17.

Cook, David G., James B. Leverenz, Pamela J. McMillan, et al.. (2003). Reduced Hippocampal Insulin-Degrading Enzyme in Late-Onset Alzheimer's Disease Is Associated with the Apolipoprotein E-ε4 Allele. American Journal Of Pathology. 162(1). 313–319. 277 indexed citations

18.

Vuletic, Simona, et al.. (2003). Widespread distribution of PLTP in human CNS: evidence for PLTP synthesis by glia and neurons, and increased levels in Alzheimer's disease. Journal of Lipid Research. 44(6). 1113–1123. 55 indexed citations

19.

Bu, Bitao, Hans H. Klünemann, Kinuko Suzuki, et al.. (2002). Niemann-Pick Disease Type C Yields Possible Clue for Why Cerebellar Neurons Do Not Form Neurofibrillary Tangles. Neurobiology of Disease. 11(2). 285–297. 53 indexed citations

20.

Maezawa, Izumi, et al.. (2002). Alterations of chaperone protein expression in presenilin mutant neurons in response to glutamate excitotoxicity. Pathology International. 52(9). 551–554. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact