J. MacMillan

1.4k total citations
36 papers, 1.1k citations indexed

About

J. MacMillan is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, J. MacMillan has authored 36 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Cellular and Molecular Neuroscience, 15 papers in Molecular Biology and 14 papers in Neurology. Recurrent topics in J. MacMillan's work include Genetic Neurodegenerative Diseases (13 papers), Mitochondrial Function and Pathology (11 papers) and Migraine and Headache Studies (9 papers). J. MacMillan is often cited by papers focused on Genetic Neurodegenerative Diseases (13 papers), Mitochondrial Function and Pathology (11 papers) and Migraine and Headache Studies (9 papers). J. MacMillan collaborates with scholars based in United Kingdom, Australia and New Zealand. J. MacMillan's co-authors include Peter S. Harper, Iain Fenton, P S Harper, Duncan J. Shaw, William Reardon, J Myring, Steve Crow, J. David Brook, Meena Upadhyaya and Russell G. Snell and has published in prestigious journals such as The Lancet, Annals of Neurology and The American Journal of Human Genetics.

In The Last Decade

J. MacMillan

35 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
J. MacMillan United Kingdom 16 648 537 389 151 126 36 1.1k
B Kremer Canada 16 716 1.1× 609 1.1× 519 1.3× 80 0.5× 158 1.3× 25 1.1k
Samantha Gillard United Kingdom 11 391 0.6× 314 0.6× 166 0.4× 77 0.5× 67 0.5× 14 1.0k
V. Collins Australia 15 196 0.3× 181 0.3× 125 0.3× 150 1.0× 40 0.3× 22 832
Hillary Lipe United States 24 945 1.5× 593 1.1× 600 1.5× 110 0.7× 85 0.7× 40 1.5k
R. H. Fryer United States 8 346 0.5× 277 0.5× 57 0.1× 46 0.3× 74 0.6× 18 756
Douglas E. Crompton Australia 18 362 0.6× 457 0.9× 143 0.4× 343 2.3× 611 4.8× 28 1.4k
James E. Hilbert United States 20 674 1.0× 590 1.1× 363 0.9× 34 0.2× 41 0.3× 29 1.1k
Rita Cittadella Italy 18 237 0.4× 274 0.5× 189 0.5× 141 0.9× 187 1.5× 36 1.0k
P Kraus Germany 10 289 0.4× 181 0.3× 350 0.9× 35 0.2× 75 0.6× 15 681
P. L. Murphy United States 13 134 0.2× 308 0.6× 296 0.8× 274 1.8× 77 0.6× 18 922

Countries citing papers authored by J. MacMillan

Since Specialization
Citations

This map shows the geographic impact of J. MacMillan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. MacMillan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. MacMillan more than expected).

Fields of papers citing papers by J. MacMillan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. MacMillan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. MacMillan. The network helps show where J. MacMillan may publish in the future.

Co-authorship network of co-authors of J. MacMillan

This figure shows the co-authorship network connecting the top 25 collaborators of J. MacMillan. A scholar is included among the top collaborators of J. MacMillan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. MacMillan. J. MacMillan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Menon, Shruti, Hannah C. Cox, Masashi Kuwahata, et al.. (2010). Association of a Notch 3 gene polymorphism with migraine susceptibility. Cephalalgia. 31(3). 264–270. 20 indexed citations
2.
Luciano, Michelle, Emma Hine, Margaret J. Wright, et al.. (2006). Effects of SCA1, MJD, and DPRLA triplet repeat polymorphisms on cognitive phenotypes in a normal population of adolescent twins. American Journal of Medical Genetics Part B Neuropsychiatric Genetics. 144B(1). 95–100. 2 indexed citations
3.
Lea, Rod A., et al.. (2005). Analysis of chromosome 1 microsatellite markers and the FHM2-ATP1A2gene mutations in migraine pedigrees. Neurological Research. 27(6). 647–652. 12 indexed citations
4.
Lea, Rod A., Dale R. Nyholt, Claire Bellis, et al.. (2005). A genome-wide scan provides evidence for loci influencing a severe heritable form of common migraine. Neurogenetics. 6(2). 67–72. 36 indexed citations
5.
Fedulov, Alexey V., et al.. (2005). Serum TGF-Beta 1 and TNF-Alpha Levels and Cardiac Fibrosis in Experimental Chronic Renal Failure. Immunological Investigations. 34(2). 143–152. 14 indexed citations
6.
Fedulov, Alexey V., et al.. (2005). Serum TGF-Beta 1 and TNF-Alpha Levels and Cardiac Fibrosis in Experimental Chronic Renal Failure. Immunological Investigations. 34(2). 143–152. 4 indexed citations
7.
Curtain, Robert P., Rod A. Lea, Sharon Quinlan, et al.. (2004). Investigation of the low-density lipoprotein receptor gene and cholesterol as a risk factor for migraine. Journal of the Neurological Sciences. 227(1). 95–100. 11 indexed citations
8.
Henderson, Robert D., et al.. (2003). Adult onset Krabbe disease may mimic motor neurone disease. Journal of Clinical Neuroscience. 10(5). 638–639. 22 indexed citations
9.
Tan, K. Meng, et al.. (2001). CADASIL (Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leucoencephalopathy):an Australian perspective. Journal of Clinical Neuroscience. 8(5). 404–406. 4 indexed citations
10.
MacMillan, J. & Gregory J. Anderson. (2000). Detection and functional analysis of mutations in Jagged1. The American Journal of Human Genetics. 67(4). 401–401. 1 indexed citations
11.
Singhrao, Sim K., P. Thomas, J.D. Wood, et al.. (1998). Huntingtin Protein Colocalizes with Lesions of Neurodegenerative Diseases: An Investigation in Huntington's, Alzheimer's, and Pick's Diseases. Experimental Neurology. 150(2). 213–222. 65 indexed citations
12.
Bêcher, Mark W., David C. Rubinsztein, Jayne Leggo, et al.. (1997). Dentatorubral and pallidoluysian atrophy (DRPLA) Clinical and neuropathological findings in genetically confirmed north american and european pedigrees. Movement Disorders. 12(4). 519–530. 72 indexed citations
13.
MacMillan, J., et al.. (1997). Novel mutation of the myelin P0 gene in a CMT1B family. Human Mutation. 9(1). 74–77. 9 indexed citations
14.
Thomas, Lois H, et al.. (1995). Obtaining Patients' Views of Nursing Care to Inform the Development of a Patient Satisfaction Scale. International Journal for Quality in Health Care. 7(2). 153–163. 59 indexed citations
15.
MacMillan, J. & P S Harper. (1994). Clinical genetics in neurological disease.. Journal of Neurology Neurosurgery & Psychiatry. 57(1). 7–15. 15 indexed citations
16.
MacMillan, J. & Niall Quinn. (1993). 15th International World Federation of Neurology Workshop on Huntington's Disease, 31 August-3 September 1993, Boston, Massachusetts, USA.. Journal of Medical Genetics. 30(12). 1039–1041. 4 indexed citations
17.
Upadhyaya, Meena, Susan Roberts, James M. Farnham, et al.. (1993). Charcot-Marie-Tooth disease 1A (CMT1A) associated with a maternal duplication of chromosome 17p11.2?12. Human Genetics. 91(4). 392–4. 38 indexed citations
18.
MacMillan, J., Russell G. Snell, Audrey Tyler, et al.. (1993). Molecular analysis and clinical correlations of the Huntington's disease mutation. The Lancet. 342(8877). 954–958. 77 indexed citations
19.
MacMillan, J., J Myring, H G Harley, et al.. (1992). Molecular analysis for the myotonic dystrophy mutation in neuromuscular disorders. Neuromuscular Disorders. 2(5-6). 405–411. 1 indexed citations
20.
MacMillan, J. & Peter S. Harper. (1991). Single‐gene neurological disorders in south wales: An epidemiological study. Annals of Neurology. 30(3). 411–414. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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