J. MacMillan
Impact in
-
- Genetic Neurodegenerative Diseases
- Hereditary Neurological Disorders
- Neurology top 5%
- Neurological disorders and treatments
- Parkinson's Disease Mechanisms and Treatments
Papers in ⓘ
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- Genetic Neurodegenerative Diseases 13
- Hereditary Neurological Disorders 6
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- Mitochondrial Function and Pathology 11
- Co-authors
- Peter S. Harper (11 shared papers)Iain Fenton (2 shared papers)P S Harper (2 shared papers)Duncan J. Shaw (2 shared papers)J Myring (3 shared papers)William Reardon (3 shared papers)Steve Crow (1 shared paper)J. David Brook (1 shared paper)
- Journals
- Journal of Medical Genetics (6 papers)Journal of Neurology Neurosurgery & Psychiatry (3 papers)Neurogenetics (3 papers)Immunological Investigations (2 papers)Human Mutation (2 papers)
- Partner nations
- United KingdomAustraliaNew Zealand
In The Last Decade
J. MacMillan
35 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 91
- Cellular and Molecular Neuroscience 648
- Neurology 389
- Neurology 101
- Psychiatry and Mental health 126
- Endocrine and Autonomic Systems 53
Countries citing papers authored by J. MacMillan
This map shows the geographic impact of J. MacMillan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. MacMillan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. MacMillan more than expected).
Fields of papers citing papers by J. MacMillan
This network shows the impact of papers produced by J. MacMillan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. MacMillan. The network helps show where J. MacMillan may publish in the future.
Co-authors
The 25 scholars most cited alongside J. MacMillan, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 36 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Size of the unstable CTG repeat sequence in relation to phenotype and parental transmission in myotonic dystrophy. | 1993 | 303 |
| 2 | 1993 | 77 | |
| 3 | 1997 | 72 | |
| 4 | 1998 | 65 | |
| 5 | 2001 | 60 | |
| 6 | 1995 | 59 | |
| 7 | 2009 | 41 | |
| 8 | 1993 | 38 | |
| 9 | 2005 | 36 | |
| 10 | 1993 | 31 | |
| 11 | 1992 | 27 | |
| 12 | 1996 | 26 | |
| 13 | 1991 | 25 | |
| 14 | 2003 | 22 | |
| 15 | 2010 | 20 | |
| 16 | 1992 | 19 | |
| 17 | 1994 | 15 | |
| 18 | 2005 | 14 | |
| 19 | 1995 | 14 | |
| 20 | 1993 | 13 |
About J. MacMillan
J. MacMillan is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Neurology, Psychiatry and Mental health and Endocrine and Autonomic Systems, having authored 36 papers that have together received 1.1k indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (13 papers), Mitochondrial Function and Pathology (11 papers), Migraine and Headache Studies (9 papers), Neurological disorders and treatments (7 papers), Hereditary Neurological Disorders (6 papers), Neuroscience of respiration and sleep (5 papers), Cerebrovascular and genetic disorders (4 papers) and Neurogenetic and Muscular Disorders Research (3 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (648 citations), Neurology (389 citations), Neurology (101 citations), Psychiatry and Mental health (126 citations) and Endocrine and Autonomic Systems (53 citations). J. MacMillan has collaborated with scholars based in United Kingdom, Australia and New Zealand. Frequent co-authors include Peter S. Harper, Iain Fenton, P S Harper, Duncan J. Shaw, J Myring, William Reardon, Steve Crow, J. David Brook, Meena Upadhyaya and Rod A. Lea. Their work appears in journals such as Journal of Medical Genetics, Journal of Neurology Neurosurgery & Psychiatry, Neurogenetics, Immunological Investigations and Human Mutation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.