Núria Llecha
Impact in
- Developmental Neuroscience top 10%
- Neurogenesis and neuroplasticity mechanisms
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- Nerve injury and regeneration
- Neuroscience and Neuropharmacology Research
Papers in
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- Cell death mechanisms and regulation 8
- RNA Interference and Gene Delivery 2
- DNA Repair Mechanisms 1
- Oncology 4
- HER2/EGFR in Cancer Research 2
- Cancer-related Molecular Pathways 2
- Co-authors
- Joan X. ComellaMario EncinasMontse IglesiasVı́ctor J. YusteJacint BoixXavier Matías‐GuiuXavier DolcetMiguel F. Segura
In The Last Decade
Núria Llecha
15 papers receiving 873 citations
Peers
Comparison fields: 5 of 92
- Developmental Neuroscience 65
- Cellular and Molecular Neuroscience 191
- Molecular Biology 561
- Neurology 117
- Cancer Research 111
Countries citing papers authored by Núria Llecha
This map shows the geographic impact of Núria Llecha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Núria Llecha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Núria Llecha more than expected).
Fields of papers citing papers by Núria Llecha
This network shows the impact of papers produced by Núria Llecha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Núria Llecha. The network helps show where Núria Llecha may publish in the future.
Co-authors
The 25 scholars most cited alongside Núria Llecha, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 13 | |
| 2 | 2009 | 41 | |
| 3 | 2009 | 19 | |
| 4 | 2008 | 18 | |
| 5 | 2008 | 35 | |
| 6 | Expression of somatostatin receptors (SSTR) in melanoma cell lines. Effect of somatostatin analogues on their proliferation | 2007 | 1 |
| 7 | 2007 | 116 | |
| 8 | 2007 | 65 | |
| 9 | 2007 | 45 | |
| 10 | 2006 | 117 | |
| 11 | 2005 | 9 | |
| 12 | 2001 | 61 | |
| 13 | 1999 | 245 | |
| 14 | 1998 | 24 | |
| 15 | 1997 | 79 |
About Núria Llecha
Núria Llecha is a scholar working on Molecular Biology, Oncology, Cancer Research, Cell Biology and Pathology and Forensic Medicine, having authored 15 papers that have together received 888 indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (8 papers), RNA Interference and Gene Delivery (2 papers), Genetic factors in colorectal cancer (2 papers), Microtubule and mitosis dynamics (2 papers), HER2/EGFR in Cancer Research (2 papers), Cancer-related Molecular Pathways (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Developmental Neuroscience (65 citations), Cellular and Molecular Neuroscience (191 citations), Molecular Biology (561 citations), Neurology (117 citations) and Cancer Research (111 citations). Núria Llecha has collaborated with scholars based in Spain, Poland and Ireland. Frequent co-authors include Joan X. Comella, Mario Encinas, Montse Iglesias, Vı́ctor J. Yuste, Jacint Boix, Xavier Matías‐Guiu, Xavier Dolcet, Miguel F. Segura, José R. Bayascas and Ana Velasco. Their work appears in journals such as Journal of Neurochemistry, Cell Research, Journal of Neuroscience, Archives of Dermatological Research and Diagnostic Molecular Pathology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.