Noriko Sakaguchi
About
In The Last Decade
Noriko Sakaguchi
53 papers receiving 10.2k citations
Hit Papers
Peers
Comparison fields: 5 of 118
- Immunology 8.1k
- Oncology 1.8k
- Molecular Biology 1.3k
- Rheumatology 1.2k
- Genetics 965
Countries citing papers authored by Noriko Sakaguchi
This map shows the geographic impact of Noriko Sakaguchi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Noriko Sakaguchi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Noriko Sakaguchi more than expected).
Fields of papers citing papers by Noriko Sakaguchi
This network shows the impact of papers produced by Noriko Sakaguchi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Noriko Sakaguchi. The network helps show where Noriko Sakaguchi may publish in the future.
Co-authorship network of co-authors of Noriko Sakaguchi
This figure shows the co-authorship network connecting the top 25 collaborators of Noriko Sakaguchi. A scholar is included among the top collaborators of Noriko Sakaguchi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Noriko Sakaguchi. Noriko Sakaguchi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Autoimmune Th17 Cells Induced Synovial Stromal and Innate Lymphoid Cell Secretion of the Cytokine GM-CSF to Initiate and Augment Autoimmune Arthritis breakdown → | 135 |
| 2 | Pillars article: immunologic self-tolerance maintained by activated T cells expressing IL-2 receptor α-chains (CD25). Breakdown of a single mechanism of self-tolerance causes various autoimmune diseases. J. Immunol. 1995. | 107 |
| 3 | 165 | |
| 4 | 142 | |
| 5 | Preferential recruitment of CCR6-expressing Th17 cells to inflamed joints via CCL20 in rheumatoid arthritis and its animal model breakdown → | 749 |
| 6 | 30 | |
| 7 | 20 | |
| 8 | 162 | |
| 9 | 228 | |
| 10 | Altered thymic T-cell selection due to a mutation of the ZAP-70 gene causes autoimmune arthritis in mice breakdown → | 647 |
| 11 | 1 | |
| 12 | 48 | |
| 13 | Immunologic tolerance maintained by CD25+ CD4+ regulatory T cells: their common role in controlling autoimmunity, tumor immunity, and transplantation tolerance breakdown → | 1230 |
| 14 | Thymus and Autoimmunity: Production of CD25+CD4+ Naturally Anergic and Suppressive T Cells as a Key Function of the Thymus in Maintaining Immunologic Self-Tolerance breakdown → | 994 |
| 15 | 15 | |
| 16 | 12 | |
| 17 | Immunologic self-tolerance maintained by CD25+CD4+ naturally anergic and suppressive T cells: induction of autoimmune disease by breaking their anergic/suppressive state. breakdown → | 1265 |
| 18 | 7 | |
| 19 | 91 | |
| 20 | 95 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.