M Itoh

7.5k total citations · 2 hit papers
18 papers, 6.3k citations indexed

About

M Itoh is a scholar working on Epidemiology, Immunology and Infectious Diseases. According to data from OpenAlex, M Itoh has authored 18 papers receiving a total of 6.3k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Epidemiology, 5 papers in Immunology and 4 papers in Infectious Diseases. Recurrent topics in M Itoh's work include Virology and Viral Diseases (5 papers), Influenza Virus Research Studies (3 papers) and Respiratory viral infections research (3 papers). M Itoh is often cited by papers focused on Virology and Viral Diseases (5 papers), Influenza Virus Research Studies (3 papers) and Respiratory viral infections research (3 papers). M Itoh collaborates with scholars based in Japan, Switzerland and United States. M Itoh's co-authors include Shimon Sakaguchi, Takamune Takahashi, Yuhshi Kuniyasu, M Iwata, Masaaki Toda, Jun Shimizu, Noriko Sakaguchi, Hak Hotta, M. Homma and Yuji Isegawa and has published in prestigious journals such as The Journal of Immunology, Journal of Applied Physics and Journal of Virology.

In The Last Decade

M Itoh

18 papers receiving 6.2k citations

Hit Papers

Immunologic self-tolerance maintained by activated T cell... 1995 2026 2005 2015 1995 1998 1000 2.0k 3.0k 4.0k

Peers

M Itoh
Comparison fields: 5 of 111
  • Immunology 5.2k
  • Oncology 967
  • Epidemiology 553
  • Genetics 496
  • Molecular Biology 466
Replace Christophe Pannetier with:
Christophe Pannetier France
Maria‐Grazia Roncarolo Italy
David N. Posnett United States
Jennifer L. Cannons United States
David H. Presky United States
Zoltán Fehérvári United States
Martin E. Sanders United States
Charles R. Maliszewski United States
Michael Lohoff Germany
Andrew J. Caton United States
Christophe Pannetier France View profile →
Citations per field, relative to M Itoh
M Itoh · 1×
Citations per year, relative to M Itoh
M Itoh · 1×

Countries citing papers authored by M Itoh

Since Specialization
Citations

This map shows the geographic impact of M Itoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M Itoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M Itoh more than expected).

Fields of papers citing papers by M Itoh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M Itoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M Itoh. The network helps show where M Itoh may publish in the future.

Co-authorship network of co-authors of M Itoh

This figure shows the co-authorship network connecting the top 25 collaborators of M Itoh. A scholar is included among the top collaborators of M Itoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M Itoh. M Itoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
# Work Indexed citations
1 13
2 36
3 8
4 9
5 19
6 74
7
Immunologic self-tolerance maintained by CD25+CD4+ naturally anergic and suppressive T cells: induction of autoimmune disease by breaking their anergic/suppressive state. breakdown →
1265
8 65
9 67
10
[Structure, function and regulation of expression of influenza virus matrix M1 protein].
3
11 113
12
Immunologic self-tolerance maintained by activated T cells expressing IL-2 receptor α-chains (CD25). Breakdown of a single mechanism of self-tolerance causes various autoimmune diseases breakdown →
4578
13 10
14
[A study of Epstein-Barr virus-associated hemophagocytic syndrome successfully treated with VP16 and analysis of T cell receptor chain genes of the bone marrow cells].
1
15 18
16 11
17 10
18 14

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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