Neva West

967 total citations
8 papers, 757 citations indexed

About

Neva West is a scholar working on Cellular and Molecular Neuroscience, Neurology and Cognitive Neuroscience. According to data from OpenAlex, Neva West has authored 8 papers receiving a total of 757 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Cellular and Molecular Neuroscience, 5 papers in Neurology and 2 papers in Cognitive Neuroscience. Recurrent topics in Neva West's work include Parkinson's Disease Mechanisms and Treatments (5 papers), Nuclear Receptors and Signaling (3 papers) and Alzheimer's disease research and treatments (2 papers). Neva West is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (5 papers), Nuclear Receptors and Signaling (3 papers) and Alzheimer's disease research and treatments (2 papers). Neva West collaborates with scholars based in United States, Netherlands and Germany. Neva West's co-authors include Michael K. Lee, Ted M. Dawson, Olga Pletniková, Juan C. Troncoso, Laura Marsh, Donald L. Price, Tobias Hartmann, Wenxue Li, Emanuela Colla and Pekka Jäkälä and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Brain Research.

In The Last Decade

Neva West

8 papers receiving 741 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Neva West United States 6 557 323 276 160 106 8 757
Anna Raunio Finland 10 653 1.2× 296 0.9× 233 0.8× 161 1.0× 155 1.5× 15 841
Yi‐Min Sun China 18 477 0.9× 307 1.0× 225 0.8× 246 1.5× 127 1.2× 73 919
Niccolò Candelise Italy 13 512 0.9× 215 0.7× 143 0.5× 211 1.3× 156 1.5× 20 728
Krisztina K. Johansen Norway 16 703 1.3× 301 0.9× 238 0.9× 247 1.5× 194 1.8× 24 933
Hitoshi Takahashi Japan 9 467 0.8× 243 0.8× 342 1.2× 199 1.2× 179 1.7× 9 689
Ann‐Kathrin Hauser Germany 19 695 1.2× 379 1.2× 187 0.7× 164 1.0× 160 1.5× 33 892
V. M. -Y. Lee United States 8 455 0.8× 556 1.7× 275 1.0× 328 2.0× 256 2.4× 8 988
Pazit Bar‐On United States 9 423 0.8× 417 1.3× 308 1.1× 255 1.6× 99 0.9× 9 856
Valerie Cullen United States 10 641 1.2× 541 1.7× 188 0.7× 192 1.2× 122 1.2× 12 970
Lasse Pihlstrøm Norway 18 549 1.0× 222 0.7× 284 1.0× 310 1.9× 229 2.2× 49 904

Countries citing papers authored by Neva West

Since Specialization
Citations

This map shows the geographic impact of Neva West's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Neva West with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Neva West more than expected).

Fields of papers citing papers by Neva West

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Neva West. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Neva West. The network helps show where Neva West may publish in the future.

Co-authorship network of co-authors of Neva West

This figure shows the co-authorship network connecting the top 25 collaborators of Neva West. A scholar is included among the top collaborators of Neva West based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Neva West. Neva West is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Galvin, James E., Paul Aisen, Jessica B. Langbaum, et al.. (2021). Early Stages of Alzheimer's Disease: Evolving the Care Team for Optimal Patient Management. Frontiers in Neurology. 11. 592302–592302. 34 indexed citations
3.
Khodr, Christina E., Mohan K. Sapru, Ye Han, et al.. (2011). An alpha-synuclein AAV gene silencing vector ameliorates a behavioral deficit in a rat model of Parkinson's disease, but displays toxicity in dopamine neurons. Brain Research. 1395. 94–107. 86 indexed citations
4.
Thomas, Bobby, Allen S. Mandir, Neva West, et al.. (2011). Resistance to MPTP-Neurotoxicity in α-Synuclein Knockout Mice Is Complemented by Human α-Synuclein and Associated with Increased β-Synuclein and Akt Activation. PLoS ONE. 6(1). e16706–e16706. 51 indexed citations
5.
Glavaski‐Joksimovic, Aleksandra, Tamás Virág, Qin Chang, et al.. (2009). Reversal of Dopaminergic Degeneration in a Parkinsonian Rat following Micrografting of Human Bone Marrow-Derived Neural Progenitors. Cell Transplantation. 18(7). 801–814. 50 indexed citations
6.
Li, Wenxue, Neva West, Emanuela Colla, et al.. (2005). Aggregation promoting C-terminal truncation of α-synuclein is a normal cellular process and is enhanced by the familial Parkinson's disease-linked mutations. Proceedings of the National Academy of Sciences. 102(6). 2162–2167. 370 indexed citations
7.
Pletniková, Olga, Neva West, Michael K. Lee, et al.. (2004). Aβ deposition is associated with enhanced cortical α-synuclein lesions in Lewy body diseases. Neurobiology of Aging. 26(8). 1183–1192. 159 indexed citations
8.
West, Neva, et al.. (1986). Interdonor Incompatibility Due to Anti‐Kell Antibody Undetectable by Automated Antibody Screening. Vox Sanguinis. 50(3). 174–176. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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