Neil E. McCarthy

2.1k total citations · 1 hit paper
40 papers, 1.5k citations indexed

About

Neil E. McCarthy is a scholar working on Immunology, Genetics and Molecular Biology. According to data from OpenAlex, Neil E. McCarthy has authored 40 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Immunology, 15 papers in Genetics and 10 papers in Molecular Biology. Recurrent topics in Neil E. McCarthy's work include Inflammatory Bowel Disease (11 papers), Immune Cell Function and Interaction (9 papers) and T-cell and B-cell Immunology (8 papers). Neil E. McCarthy is often cited by papers focused on Inflammatory Bowel Disease (11 papers), Immune Cell Function and Interaction (9 papers) and T-cell and B-cell Immunology (8 papers). Neil E. McCarthy collaborates with scholars based in United Kingdom, Singapore and Australia. Neil E. McCarthy's co-authors include Andrew J. Stagg, Kevin Whelan, James O. Lindsay, Charlotte Hedin, Matthias Eberl, Siew C. Ng, Jane Benjamin, Alastair Forbes, Jeremy Sanderson and Stella C. Knight and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Immunology and Gastroenterology.

In The Last Decade

Neil E. McCarthy

40 papers receiving 1.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Effects of Low FODMAP Diet on Symptoms, Fecal Microbiome, and Markers of Inflammation in Patients With Quiescent Inflammatory Bowel Disease in a Randomized Trial

2019 252 citations Selina Cox, James O. Lindsay et al. Gastroenterology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Neil E. McCarthy United Kingdom	18	666	521	398	279	268	40	1.5k
Takehide Fujimoto Japan	13	628 0.9×	380 0.7×	236 0.6×	270 1.0×	157 0.6×	30	1.2k
Mark E. Gerich United States	18	945 1.4×	383 0.7×	390 1.0×	220 0.8×	80 0.3×	38	1.8k
Jonathan B. Meddings Canada	21	488 0.7×	281 0.5×	201 0.5×	324 1.2×	214 0.8×	38	1.7k
Shunsuke Komoto Japan	23	391 0.6×	250 0.5×	305 0.8×	219 0.8×	109 0.4×	80	1.3k
Maria Pastore Italy	21	324 0.5×	551 1.1×	204 0.5×	194 0.7×	284 1.1×	92	1.8k
Kyle L. Flannigan Canada	22	666 1.0×	138 0.3×	383 1.0×	147 0.5×	105 0.4×	38	1.6k
Qihong Zhao United States	14	776 1.2×	190 0.4×	356 0.9×	126 0.5×	94 0.4×	22	1.4k
Leiqi Xu China	14	957 1.4×	162 0.3×	284 0.7×	171 0.6×	96 0.4×	22	1.5k
Vincenza Di Leo Italy	20	573 0.9×	533 1.0×	268 0.7×	696 2.5×	315 1.2×	30	2.1k
Chiara Sorini Sweden	14	592 0.9×	276 0.5×	350 0.9×	97 0.3×	124 0.5×	20	1.1k

Countries citing papers authored by Neil E. McCarthy

Since Specialization

Citations

This map shows the geographic impact of Neil E. McCarthy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Neil E. McCarthy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Neil E. McCarthy more than expected).

Fields of papers citing papers by Neil E. McCarthy

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Neil E. McCarthy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Neil E. McCarthy. The network helps show where Neil E. McCarthy may publish in the future.

Co-authorship network of co-authors of Neil E. McCarthy

This figure shows the co-authorship network connecting the top 25 collaborators of Neil E. McCarthy. A scholar is included among the top collaborators of Neil E. McCarthy based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Neil E. McCarthy. Neil E. McCarthy is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Tyler, Christopher J., Simone Cuff, Robert Andrews, et al.. (2024). IL-21 conditions antigen-presenting human γδ T-cells to promote IL-10 expression in naïve and memory CD4+ T-cells. PubMed. 3(1). kyae008–kyae008. 3 indexed citations

Ruchti, Fiorella, et al.. (2024). γδ T cells respond directly and selectively to the skin commensal yeast Malassezia for IL-17-dependent fungal control. PLoS Pathogens. 20(1). e1011668–e1011668. 6 indexed citations

Savage, Michael P., et al.. (2023). Mucosal Immunity to Gut Fungi in Health and Inflammatory Bowel Disease. Journal of Fungi. 9(11). 1105–1105. 8 indexed citations

McCarthy, Neil E., et al.. (2022). pSILAC-Based Determination of Cellular Protein Sorting into Extracellular Vesicles. Methods in molecular biology. 2603. 43–58. 1 indexed citations

Hedin, Charlotte, Neil E. McCarthy, Petra Louis, et al.. (2021). Prebiotic fructans have greater impact on luminal microbiology and CD3+ T cells in healthy siblings than patients with Crohn's disease: A pilot study investigating the potential for primary prevention of inflammatory bowel disease. Clinical Nutrition. 40(8). 5009–5019. 17 indexed citations

McCarthy, Neil E., Andrew J. Stagg, Claire Price, et al.. (2020). Patients with gastrointestinal irritability after TGN1412-induced cytokine storm displayed selective expansion of gut-homing αβ and γδT cells. Cancer Immunology Immunotherapy. 70(4). 1143–1153. 2 indexed citations

Panoskaltsis, Nicki, Neil E. McCarthy, Andrew J. Stagg, et al.. (2020). Immune reconstitution and clinical recovery following anti-CD28 antibody (TGN1412)-induced cytokine storm. Cancer Immunology Immunotherapy. 70(4). 1127–1142. 11 indexed citations

Panoskaltsis, Nicki, Neil E. McCarthy, & Stella C. Knight. (2020). Myelopoiesis of acute inflammation: lessons from TGN1412-induced cytokine storm. Cancer Immunology Immunotherapy. 70(4). 1155–1160. 4 indexed citations

Cox, Selina, James O. Lindsay, Sébastien Fromentin, et al.. (2019). Effects of Low FODMAP Diet on Symptoms, Fecal Microbiome, and Markers of Inflammation in Patients With Quiescent Inflammatory Bowel Disease in a Randomized Trial. Gastroenterology. 158(1). 176–188.e7. 252 indexed citations breakdown →

10.

McCarthy, Neil E. & Matthias Eberl. (2018). Human γδ T-Cell Control of Mucosal Immunity and Inflammation. Frontiers in Immunology. 9. 985–985. 71 indexed citations

11.

Giles, Edward, Theodore J. Sanders, Neil E. McCarthy, et al.. (2016). Regulation of human intestinal T-cell responses by type 1 interferon-STAT1 signaling is disrupted in inflammatory bowel disease. Mucosal Immunology. 10(1). 184–193. 25 indexed citations

12.

Petty, Robert, Neil E. McCarthy, Rifca Le Dieu, & Jonathan R. Kerr. (2016). MicroRNAs hsa-miR-99b, hsa-miR-330, hsa-miR-126 and hsa-miR-30c: Potential Diagnostic Biomarkers in Natural Killer (NK) Cells of Patients with Chronic Fatigue Syndrome (CFS)/ Myalgic Encephalomyelitis (ME). PLoS ONE. 11(3). e0150904–e0150904. 36 indexed citations

13.

Hedin, Charlotte, Neil E. McCarthy, Petra Louis, et al.. (2014). Altered intestinal microbiota and blood T cell phenotype are shared by patients with Crohn's disease and their unaffected siblings. Gut. 63(10). 1578–1586. 125 indexed citations

14.

Sanders, Theodore J., Neil E. McCarthy, Edward Giles, et al.. (2014). Increased Production of Retinoic Acid by Intestinal Macrophages Contributes to Their Inflammatory Phenotype in Patients With Crohn's Disease. Gastroenterology. 146(5). 1278–1288.e2. 70 indexed citations

15.

McCarthy, Neil E., Charlotte Hedin, Anna Vossenkämper, et al.. (2012). OC-009 Human anti-microbial vδ2+ t-cells are novel intestinal lymphocytes with functional relevance in crohn's disease. Gut. 61(Suppl 2). A4.1–A4. 1 indexed citations

16.

Mann, Elizabeth R., David Bernardo, Hafid O. Al‐Hassi, et al.. (2011). Human Gut-Specific Homeostatic Dendritic Cells Are Generated from Blood Precursors by the Gut Microenvironment. Inflammatory Bowel Diseases. 18(7). 1275–1286. 24 indexed citations

17.

Benjamin, Jane, Charlotte Hedin, Siew C. Ng, et al.. (2011). Randomised, double-blind, placebo-controlled trial of fructo-oligosaccharides in active Crohn's disease. Gut. 60(7). 923–929. 252 indexed citations

18.

Ng, Siew C., Jane Benjamin, Neil E. McCarthy, et al.. (2010). Relationship between human intestinal dendritic cells, gut microbiota, and disease activity in Crohnʼs disease. Inflammatory Bowel Diseases. 17(10). 2027–2037. 93 indexed citations

19.

Bedford, P., Neil E. McCarthy, Philip W. Ind, et al.. (2008). Dendritic cells from control but not atopic donors respond to contact and respiratory sensitizer treatment in vitro with differential cytokine production and altered stimulatory capacity. Clinical & Experimental Allergy. 38(7). 1148–1159. 15 indexed citations

20.

McCarthy, Neil E., Hannah Jones, Robert J. Shiner, et al.. (2006). Inhaled allergen‐driven CD1c up‐regulation and enhanced antigen uptake by activated human respiratory‐tract dendritic cells in atopic asthma. Clinical & Experimental Allergy. 37(1). 72–82. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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