Christopher G. Mathew

53.5k total citations · 3 hit papers
246 papers, 14.2k citations indexed

About

Christopher G. Mathew is a scholar working on Genetics, Molecular Biology and Epidemiology. According to data from OpenAlex, Christopher G. Mathew has authored 246 papers receiving a total of 14.2k indexed citations (citations by other indexed papers that have themselves been cited), including 109 papers in Genetics, 105 papers in Molecular Biology and 38 papers in Epidemiology. Recurrent topics in Christopher G. Mathew's work include DNA Repair Mechanisms (51 papers), Inflammatory Bowel Disease (50 papers) and BRCA gene mutations in cancer (19 papers). Christopher G. Mathew is often cited by papers focused on DNA Repair Mechanisms (51 papers), Inflammatory Bowel Disease (50 papers) and BRCA gene mutations in cancer (19 papers). Christopher G. Mathew collaborates with scholars based in United Kingdom, United States and South Africa. Christopher G. Mathew's co-authors include Hans Joenje, Cathryn M. Lewis, Alastair Forbes, Jochen Hampe, Stefan Schreiber, Sheila Fisher, Martin Bobrow, Muddassar M. Mirza, John Edward Lennard-Jones and Johan P. de Winter and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Lancet.

In The Last Decade

Christopher G. Mathew

237 papers receiving 13.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Association between insertion mutation in NOD2 gene and Crohn's disease in German and British populations

2001 837 citations Jochen Hampe, Andrew Cuthbert et al. profile →
Mapping of a susceptibility locus for Crohn's disease on chromosome 16

1996 727 citations Christopher G. Mathew et al. profile →
The contribution of NOD2 gene mutations to the risk and site of disease in inflammatory bowel disease

2002 523 citations Andrew Cuthbert, Sheila Fisher et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Christopher G. Mathew United Kingdom	62	7.3k	6.4k	2.3k	2.1k	2.1k	246	14.2k
Karl V. Voelkerding United States	32	11.2k 1.5×	9.8k 1.5×	1.4k 0.6×	1.7k 0.8×	1.1k 0.5×	86	23.1k
John W. Belmont United States	60	6.6k 0.9×	5.3k 0.8×	2.7k 1.2×	1.3k 0.6×	2.4k 1.1×	217	14.4k
Magnus Nordenskjöld Sweden	57	4.8k 0.7×	3.7k 0.6×	1.4k 0.6×	769 0.4×	2.2k 1.1×	241	12.9k
Gilles Thomas United States	38	3.7k 0.5×	4.7k 0.7×	4.3k 1.9×	1.7k 0.8×	2.4k 1.1×	72	11.5k
Marshall S. Horwitz United States	58	6.9k 1.0×	7.3k 1.1×	2.9k 1.3×	737 0.3×	1.3k 0.6×	220	13.6k
Takashi Shimada Japan	57	6.9k 1.0×	5.7k 0.9×	919 0.4×	1.4k 0.7×	1.2k 0.6×	318	17.3k
Timothy A. Stewart United States	45	6.1k 0.8×	3.8k 0.6×	5.4k 2.3×	2.2k 1.0×	3.1k 1.5×	80	15.6k
Michel Vidaud France	75	5.9k 0.8×	1.6k 0.3×	1.5k 0.6×	1.4k 0.6×	6.0k 2.9×	269	17.4k
David A. Williams United States	74	11.0k 1.5×	4.7k 0.7×	5.5k 2.4×	2.2k 1.0×	1.0k 0.5×	338	21.2k
Audrey D. Goddard United States	49	9.8k 1.4×	2.1k 0.3×	3.9k 1.7×	894 0.4×	1.1k 0.5×	70	15.8k

Countries citing papers authored by Christopher G. Mathew

Since Specialization

Citations

This map shows the geographic impact of Christopher G. Mathew's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher G. Mathew with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher G. Mathew more than expected).

Fields of papers citing papers by Christopher G. Mathew

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher G. Mathew. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher G. Mathew. The network helps show where Christopher G. Mathew may publish in the future.

Co-authorship network of co-authors of Christopher G. Mathew

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher G. Mathew. A scholar is included among the top collaborators of Christopher G. Mathew based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher G. Mathew. Christopher G. Mathew is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Chen, Wenlong Carl, Chantal Babb de Villiers, Sang Hyuck Lee, et al.. (2025). Genome-wide association study identifies common variants associated with breast cancer in South African Black women. Nature Communications. 16(1). 3542–3542. 1 indexed citations

Brandenburg, Jean‐Tristan, et al.. (2024). Prevalence and Reclassification of Genetic Variants in South African Populations with Breast Cancer. Genes Chromosomes and Cancer. 63(9). e23275–e23275. 1 indexed citations

Muchengeti, Mazvita, Debbie Bradshaw, Wenlong Carl Chen, et al.. (2023). Kaposi sarcoma‐associated herpesvirus, HIV‐1 and Kaposi sarcoma risk in black South Africans diagnosed with cancer during antiretroviral treatment rollout. International Journal of Cancer. 152(10). 2081–2089. 4 indexed citations

Singh, Elvira, Wenlong Carl Chen, Debbie Bradshaw, et al.. (2022). Thirteen cancers associated with HIV infection in a Black South African cancer patient population (1995‐2016). International Journal of Cancer. 152(2). 183–194. 11 indexed citations

Singh, Elvira, Debbie Bradshaw, Wenlong Carl Chen, et al.. (2022). Usefulness of high‐risk HPV early oncoprotein (E6 and E7) serological markers in the detection of cervical cancer: A systematic review and meta‐analysis. Journal of Medical Virology. 95(1). e27900–e27900. 16 indexed citations

Boua, Palwendé Romuald, Jean‐Tristan Brandenburg, Ananyo Choudhury, et al.. (2022). Genetic associations with carotid intima-media thickness link to atherosclerosis with sex-specific effects in sub-Saharan Africans. Nature Communications. 13(1). 855–855. 13 indexed citations

Chen, Wenlong Carl, Elvira Singh, Mazvita Muchengeti, et al.. (2020). Johannesburg Cancer Study (JCS): contribution to knowledge and opportunities arising from 20 years of data collection in an African setting. Cancer Epidemiology. 65. 101701–101701. 15 indexed citations

Onoufriadis, Alexandros, Kristina L. Stone, Katrina M. de Lange, et al.. (2017). Exome Sequencing and Genotyping Identify a Rare Variant in NLRP7 Gene Associated With Ulcerative Colitis. Journal of Crohn s and Colitis. 12(3). 321–326. 15 indexed citations

Gravante, Gianpiero, et al.. (2014). "Left-sided" Chilaiditi sign? A large gastric perforation with secondary pancreatitis.. PubMed. 18(12). 1694–7. 1 indexed citations

10.

Elliott, Tim, Natalie J. Prescott, B N Hudspith, et al.. (2011). Autophagy gene polymorphisms influence the interaction of E coli and macrophages in Crohn's disease. Gut. 60(Suppl 1). A61.1–A61. 1 indexed citations

11.

Baburajan, Bijay, M Arenas, Natalie J. Prescott, et al.. (2007). The pharmacogenetics of folate and purine metabolic pathways in methotrexate therapy of inflammatory bowel disease.. Gut. 56. 1 indexed citations

12.

Tischkowitz, Marc, Najim Ameziane, Quinten Waisfisz, et al.. (2003). Bi-allelic silencing of the Fanconi anaemia gene FANCF in acute myeloid leukaemia. European Journal of Human Genetics. 11(3). 83–83. 7 indexed citations

13.

Fisher, Sheila, Anne Marie Moody, Christopher G. Mathew, & Cathryn M. Lewis. (2002). Efficient candidate gene study design for association studies in complex diseases. Genetic Epidemiology. 23(3). 1 indexed citations

14.

Hampe, Jochen, H Frenzel, Muddassar M. Mirza, et al.. (2001). Evidence for a NOD2 -independent susceptibility locus for inflammatory bowel disease on chromosome 16p. Proceedings of the National Academy of Sciences. 99(1). 321–326. 82 indexed citations

15.

Tischkowitz, Marc, Neil V. Morgan, S V Hodgson, et al.. (2001). Mutation analysis of the Fanconi Anaemia group A, C, E, F and G genes in sporadic acute myeloid leukaemia. Journal of Medical Genetics. 38.

16.

Wainwright, Linda, et al.. (2001). Molecular genetic analysis of Fanconi anaemia in Black South Africans.. The American Journal of Human Genetics. 69(4). 588–588. 1 indexed citations

17.

Allen, M H, Cathryn M. Lewis, Scott Fisher, et al.. (2000). Corneodesmosin (S) gene association with psoriasis vulgaris. Journal of Investigative Dermatology. 115(3). 576–576. 2 indexed citations

18.

Marinaki, Anthony M., Margaret Town, Fernando Moro, et al.. (1999). Exclusion of four candidate kidney disease loci by linkage analysis in familial juvenile hyperuricaemic nephropathy [FJHN]. Cellular & Molecular Biology Letters. 4(3). 2 indexed citations

19.

Whittock, Neil V., Roland G. Roberts, Christopher G. Mathew, & Stephen Abbs. (1997). Dystrophin Point Mutation Screening Using a Multiplexed Protein Truncation Test. Genetic Testing. 1(2). 115–123. 23 indexed citations

20.

Wallis, Gillian A., Peter Beighton, C. D. Boyd, & Christopher G. Mathew. (1986). Mutations linked to the pro alpha 2(I) collagen gene are responsible for several cases of osteogenesis imperfecta type I.. Journal of Medical Genetics. 23(5). 411–416. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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