Natalie L. Stephenson
- Biotechnology top 5%
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- Melanoma and MAPK Pathways 4
- Protein Kinase Regulation and GTPase Signaling 3
- PI3K/AKT/mTOR signaling in cancer 2
- Developmental Biology and Gene Regulation 2
- Fibroblast Growth Factor Research 1
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- Computational Drug Discovery Methods 3
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- Cancer Mechanisms and Therapy 2
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- Orthopaedic implants and arthroplasty 1
- Co-authors
- John BrognardJohanna M. AvisEleanor W. TrotterAndrew HudsonCrispin MillerAlexandra C. NewtonChristopher WirthEmily L. Kang
- Journals
- Cell (1 paper)Proceedings of the National Academy of Sciences (3 papers)Nature Communications (1 paper)
- Partner nations
- United KingdomUnited StatesAustralia
In The Last Decade
Natalie L. Stephenson
15 papers receiving 644 citations
Peers
Comparison fields: 5 of 83
- Biotechnology 118
- Molecular Biology 501
- Cancer Research 77
- Aging 8
- Cell Biology 69
Countries citing papers authored by Natalie L. Stephenson
This map shows the geographic impact of Natalie L. Stephenson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natalie L. Stephenson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natalie L. Stephenson more than expected).
Fields of papers citing papers by Natalie L. Stephenson
This network shows the impact of papers produced by Natalie L. Stephenson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natalie L. Stephenson. The network helps show where Natalie L. Stephenson may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Natalie L. Stephenson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2020 | 7 | |
| 2 | 2018 | 33 | |
| 3 | 2018 | 10 | |
| 4 | 2016 | 20 | |
| 5 | 2015 | 263 | |
| 6 | 2015 | 3 | |
| 7 | 2015 | 2 | |
| 8 | 2015 | 11 | |
| 9 | 2014 | 63 | |
| 10 | 2014 | 1 | |
| 11 | 2014 | 1 | |
| 12 | 2013 | 51 | |
| 13 | 2012 | 69 | |
| 14 | 2009 | 23 | |
| 15 | 2007 | 119 |
About Natalie L. Stephenson
Natalie L. Stephenson is a scholar working on Computational Theory and Mathematics, Molecular Biology and Immunology and Allergy, having authored 15 papers that have together received 676 indexed citations. Recurring topics across this work include Melanoma and MAPK Pathways (4 papers), Computational Drug Discovery Methods (3 papers), Protein Kinase Regulation and GTPase Signaling (3 papers), Cancer Mechanisms and Therapy (2 papers), PI3K/AKT/mTOR signaling in cancer (2 papers), Developmental Biology and Gene Regulation (2 papers), Orthopaedic implants and arthroplasty (1 paper) and Fibroblast Growth Factor Research (1 paper). The work is most often cited by research in Biotechnology (118 citations), Molecular Biology (501 citations) and Cancer Research (77 citations). Natalie L. Stephenson has collaborated with scholars based in United Kingdom, United States and Australia. Frequent co-authors include John Brognard, Johanna M. Avis, Eleanor W. Trotter, Andrew Hudson, Crispin Miller, Alexandra C. Newton, Christopher Wirth, Emily L. Kang, Tony Hunter and Corina E. Antal. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.