Xuecui Guo

1.5k total citations
13 papers, 1.2k citations indexed

About

Xuecui Guo is a scholar working on Molecular Biology, Oncology and Biotechnology. According to data from OpenAlex, Xuecui Guo has authored 13 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 8 papers in Oncology and 5 papers in Biotechnology. Recurrent topics in Xuecui Guo's work include Cancer-related Molecular Pathways (5 papers), Cancer Research and Treatments (4 papers) and Mast cells and histamine (3 papers). Xuecui Guo is often cited by papers focused on Cancer-related Molecular Pathways (5 papers), Cancer Research and Treatments (4 papers) and Mast cells and histamine (3 papers). Xuecui Guo collaborates with scholars based in Canada, United States and United Kingdom. Xuecui Guo's co-authors include Alea A. Mills, Hannes Vogel, William M. Keyes, John W. Schrader, Scott W. Lowe, Juergen Kast, Julian Vasilescu, Ying Wu, Wangzhi Li and Götz R. A. Ehrhardt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Genes & Development.

In The Last Decade

Xuecui Guo

13 papers receiving 1.1k citations

Peers

Countries citing papers authored by Xuecui Guo

Since Specialization

Citations

This map shows the geographic impact of Xuecui Guo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xuecui Guo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xuecui Guo more than expected).

Fields of papers citing papers by Xuecui Guo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xuecui Guo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xuecui Guo. The network helps show where Xuecui Guo may publish in the future.

Co-authorship network of co-authors of Xuecui Guo

This figure shows the co-authorship network connecting the top 25 collaborators of Xuecui Guo. A scholar is included among the top collaborators of Xuecui Guo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xuecui Guo. Xuecui Guo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

#	Work	Indexed citations
1	ΔNp63α Is an Oncogene that Targets Chromatin Remodeler Lsh to Drive Skin Stem Cell Proliferation and Tumorigenesis 2011 ·Cell stem cell ·William M. Keyes, Matteo Pecoraro, Victoria Aranda, (unknown), Wangzhi Li, Hannes Vogel, Xuecui Guo, (unknown), Tatyana V. Michurina, Grigori Enikolopov, Senthil K. Muthuswamy, Alea A. Mills	159
2	TAp63 induces senescence and suppresses tumorigenesis in vivo 2009 ·Nature Cell Biology ·Xuecui Guo, William M. Keyes, Cristian Papazoglu, Johannes Zuber, Wangzhi Li, Scott W. Lowe, Hannes Vogel, Alea A. Mills	206
3	p63, Cellular Senescence and Tumor Development 2007 ·Cell Cycle ·Xuecui Guo, Alea A. Mills	24
4	Expression of activated M-Ras in hemopoietic stem cells initiates leukemogenic transformation, immortalization and preferential generation of mast cells 2006 ·Oncogene ·Xuecui Guo, (unknown), John W. Schrader	14
5	p63heterozygous mutant mice are not prone to spontaneous or chemically induced tumors 2006 ·Proceedings of the National Academy of Sciences ·William M. Keyes, Hannes Vogel, Maranke I. Koster, Xuecui Guo, Yi Qi, (unknown), Dennis R. Roop, Allan Bradley, Alea A. Mills	70
6	Expression of a constitutively active mutant of M-Ras in normal bone marrow is sufficient for induction of a malignant mastocytosis/mast cell leukemia, distinct from the histiocytosis/monocytic leukemia induced by expression of activated H-Ras 2005 ·Oncogene ·Xuecui Guo, Kasmintan A. Schrader, (unknown), John W. Schrader	19
7	p63 deficiency activates a program of cellular senescence and leads to accelerated aging 2005 ·Genes & Development ·William M. Keyes, Ying Wu, Hannes Vogel, Xuecui Guo, Scott W. Lowe, Alea A. Mills	237
8	Identification of protein‐protein interactions using *in vivo* cross‐linking and mass spectrometry 2004 ·PROTEOMICS ·Julian Vasilescu, Xuecui Guo, Juergen Kast	182
9	Defining the Involvement of p38α MAPK in the Production of Anti- and Proinflammatory Cytokines Using an SB 203580-resistant Form of the Kinase 2003 ·Journal of Biological Chemistry ·Xuecui Guo, Robert E. Gerl, John W. Schrader	67
10	Activation of small GTPases of the Ras and Rho family by growth factors active on mast cells 2002 ·Molecular Immunology ·John W. Schrader, (unknown), Brock Grill, Xuecui Guo	3
11	Ras and relatives—job sharing and networking keep an old family together 2002 ·Experimental Hematology ·Annette Ehrhardt, Götz R. A. Ehrhardt, Xuecui Guo, John W. Schrader	147
12	The p38 mitogen-activated protein kinases can have opposing roles in the antigen-dependent or endotoxin-stimulated production of IL-12 and IFN-γ 2001 ·European Journal of Immunology ·Ruth A. Salmon, Xuecui Guo, Hung‐Sia Teh, John W. Schrader	23
13	The mechanism of induction of anergic cells by CTLA4Ig 2000 ·Europe PMC (PubMed Central) ·Houping Wang, (unknown), (unknown), Xuecui Guo	1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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