Nat F. Brown

2.9k total citations
30 papers, 1.7k citations indexed

About

Nat F. Brown is a scholar working on Endocrinology, Food Science and Ecology. According to data from OpenAlex, Nat F. Brown has authored 30 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Endocrinology, 18 papers in Food Science and 7 papers in Ecology. Recurrent topics in Nat F. Brown's work include Salmonella and Campylobacter epidemiology (18 papers), Vibrio bacteria research studies (16 papers) and Escherichia coli research studies (10 papers). Nat F. Brown is often cited by papers focused on Salmonella and Campylobacter epidemiology (18 papers), Vibrio bacteria research studies (16 papers) and Escherichia coli research studies (10 papers). Nat F. Brown collaborates with scholars based in Canada, Australia and United Kingdom. Nat F. Brown's co-authors include B. Brett Finlay, Brian K. Coombes, Mark E. Wickham, John H. Brumell, Erin C. Boyle, Yanet Valdez, Bruce A. Vallance, Leonard J. Foster, Michael J. Lowden and B. Brett Finlay and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The EMBO Journal.

In The Last Decade

Nat F. Brown

29 papers receiving 1.7k citations

Peers

Countries citing papers authored by Nat F. Brown

Since Specialization

Citations

This map shows the geographic impact of Nat F. Brown's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nat F. Brown with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nat F. Brown more than expected).

Fields of papers citing papers by Nat F. Brown

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nat F. Brown. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nat F. Brown. The network helps show where Nat F. Brown may publish in the future.

Co-authorship network of co-authors of Nat F. Brown

This figure shows the co-authorship network connecting the top 25 collaborators of Nat F. Brown. A scholar is included among the top collaborators of Nat F. Brown based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nat F. Brown. Nat F. Brown is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Interactome disassembly during apoptosis occurs independent of caspase cleavage 2017 ·Molecular Systems Biology ·Nichollas E. Scott, Lindsay D. Rogers, Anna Prudova, Nat F. Brown, Nikolaus Fortelny, Christopher M. Overall, Leonard J. Foster	43
2	Global Impact of Salmonella Pathogenicity Island 2-secreted Effectors on the Host Phosphoproteome 2013 ·Molecular & Cellular Proteomics ·Koshi Imami, Amit P. Bhavsar, Hongbing Yu, Nat F. Brown, Lindsay D. Rogers, B. Brett Finlay, Leonard J. Foster	34
3	The Salmonella Type III Effector SspH2 Specifically Exploits the NLR Co-chaperone Activity of SGT1 to Subvert Immunity 2013 ·PLoS Pathogens ·Amit P. Bhavsar, Nat F. Brown, (unknown), Marcel Wiermer, Dale D. O. Martin, Karolynn Hsu, Koshi Imami, Colin J.D. Ross, Michael R. Hayden, Leonard J. Foster, Xin Li, Phil Hieter, B. Brett Finlay	80
4	Potential origins and horizontal transfer of type III secretion systems and effectors 2011 ·Mobile Genetic Elements ·Nat F. Brown, B. Brett Finlay	28
5	Salmonella Phage ST64B Encodes a Member of the SseK/NleB Effector Family 2011 ·PLoS ONE ·Nat F. Brown, Brian K. Coombes, (unknown), Mark E. Wickham, Michael J. Lowden, Ohad Gal‐Mor, David L. Goode, Erin C. Boyle, (unknown), B. Brett Finlay	51
6	Inhibition of the PtdIns(5) kinase PIKfyve disrupts intracellular replication of Salmonella 2010 ·The EMBO Journal ·Markus C. Kerr, Jack T. H. Wang, (unknown), Nicholas Hamilton, Liam Town, Darren L. Brown, Frédéric A. Meunier, Nat F. Brown, Jennifer L. Stow, Rohan D. Teasdale	71
7	Oral infection of mice with Salmonella entericaserovar Typhimurium causes meningitis and infection of the brain 2007 ·BMC Infectious Diseases ·Mark E. Wickham, Nat F. Brown, John Provias, B. Brett Finlay, Brian K. Coombes	38
8	SopD acts cooperatively with SopB during Salmonella enterica serovar Typhimurium invasion 2007 ·Cellular Microbiology ·Malina A. Bakowski, Judith T. Cirulis, Nat F. Brown, B. Brett Finlay, John H. Brumell	61
9	Virulence Is Positively Selected by Transmission Success between Mammalian Hosts 2007 ·Current Biology ·Mark E. Wickham, Nat F. Brown, Erin C. Boyle, Brian K. Coombes, B. Brett Finlay	50
10	Salmonella enterica serovar Typhimurium effectors SopB, SopE, SopE2 and SipA disrupt tight junction structure and function 2006 ·Cellular Microbiology ·Erin C. Boyle, Nat F. Brown, B. Brett Finlay	153
11	Bacterial Genetic Determinants of Non‐O157 STEC Outbreaks and Hemolytic‐Uremic Syndrome after Infection 2006 ·The Journal of Infectious Diseases ·Mark E. Wickham, Claudia Lupp, Mariola Mascarenhas, Alejandra Vázquez, Brian K. Coombes, Nat F. Brown, Bryan Coburn, Wanyin Deng, José L. Puente, Mohamed A. Karmali, B. Brett Finlay	84
12	Crossing the Line: Selection and Evolution of Virulence Traits 2006 ·PLoS Pathogens ·Nat F. Brown, Mark E. Wickham, Brian K. Coombes, B. Brett Finlay	72
13	Negative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid 2005 ·Proceedings of the National Academy of Sciences ·Brian K. Coombes, Mark E. Wickham, Michael J. Lowden, Nat F. Brown, B. Brett Finlay	81
14	Salmonella Pathogenicity Island 2 Is Expressed Priorto Penetrating the Intestine 2005 ·PLoS Pathogens ·Nat F. Brown, Bruce A. Vallance, Brian K. Coombes, Yanet Valdez, Bryan Coburn, B. Brett Finlay	101
15	Genetic and Molecular Analysis of GogB, a Phage-encoded Type III-secreted Substrate in Salmonella enterica Serovar Typhimurium with Autonomous Expression from its Associated Phage 2005 ·Journal of Molecular Biology ·Brian K. Coombes, Mark E. Wickham, Nat F. Brown, Sébastien Lemire, Lionello Bossi, William Hsiao, Fiona S. L. Brinkman, B. Brett Finlay	51
16	Expression and Secretion of Salmonella Pathogenicity Island-2 Virulence Genes in Response to Acidification Exhibit Differential Requirements of a Functional Type III Secretion Apparatus and SsaL 2004 ·Journal of Biological Chemistry ·Brian K. Coombes, Nat F. Brown, Yanet Valdez, John H. Brumell, B. Brett Finlay	154
17	The related effector proteins SopD and SopD2 from Salmonella enterica serovar Typhimurium contribute to virulence during systemic infection of mice 2004 ·Molecular Microbiology ·Xiuju Jiang, Olivia W. Rossanese, Nat F. Brown, (unknown), Jorge E. Galán, B. Brett Finlay, John H. Brumell	81
18	SseA is required for translocation of Salmonella pathogenicity island-2 effectors into host cells 2003 ·Microbes and Infection ·Brian K. Coombes, Nat F. Brown, (unknown), Bruce A. Vallance, B. Brett Finlay	32
19	SopD2 is a Novel Type III Secreted Effector of Salmonella typhimurium That Targets Late Endocytic Compartments Upon Delivery Into Host Cells 2003 ·Traffic ·John H. Brumell, (unknown), Nat F. Brown, Bruce A. Vallance, Leigh A. Knodler, B. Brett Finlay	102
20	Identification of new transposable genetic elements inBurkholderia pseudomalleiusing subtractive hybridisation 2000 ·FEMS Microbiology Letters ·Nat F. Brown, Ala E. Lew, Ifor R. Beacham	12

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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