Millet Treinin

3.3k total citations
44 papers, 2.5k citations indexed

About

Millet Treinin is a scholar working on Aging, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Millet Treinin has authored 44 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Aging, 22 papers in Molecular Biology and 17 papers in Cellular and Molecular Neuroscience. Recurrent topics in Millet Treinin's work include Genetics, Aging, and Longevity in Model Organisms (24 papers), Circadian rhythm and melatonin (15 papers) and Nicotinic Acetylcholine Receptors Study (13 papers). Millet Treinin is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (24 papers), Circadian rhythm and melatonin (15 papers) and Nicotinic Acetylcholine Receptors Study (13 papers). Millet Treinin collaborates with scholars based in Israel, United States and United Kingdom. Millet Treinin's co-authors include Martin Chalfie, David M. Miller, Orgad Laub, David H. Hall, William R Schafer, Andrew Fire, Yosef Gruenbaum, Jun Liu, Marios Chatzigeorgiou and P. Spann and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Millet Treinin

43 papers receiving 2.5k citations

Peers

Millet Treinin

AGING

CMN

EAS

PHYSI

Massimo A. Hilliard Australia

Peter J. Roy Canada

Naoki Hisamoto Japan

Laurent Ségalat France

Karen L. Thijssen Netherlands

Christelle Gally France

William W. Ja United States

David M. Miller United States

Joseph A. Dent Canada

Demetrios K. Vassilatis United States

Massimo A. Hilliard Australia

Millet Treinin

1.0k ×0.7

1.3k ×1.3

AGING

750 ×1.3

CMN

579 ×1.1

EAS

315 ×1.1

PHYSI

Citations per year, relative to Millet Treinin Millet Treinin (= 1×) peers Massimo A. Hilliard

Countries citing papers authored by Millet Treinin

Since Specialization

Citations

This map shows the geographic impact of Millet Treinin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Millet Treinin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Millet Treinin more than expected).

Fields of papers citing papers by Millet Treinin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Millet Treinin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Millet Treinin. The network helps show where Millet Treinin may publish in the future.

Co-authorship network of co-authors of Millet Treinin

This figure shows the co-authorship network connecting the top 25 collaborators of Millet Treinin. A scholar is included among the top collaborators of Millet Treinin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Millet Treinin. Millet Treinin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ben-Ami, Hagit Cohen, et al.. (2021). Conserved nicotine-activated neuroprotective pathways involve mitochondrial stress. iScience. 24(3). 102140–102140. 11 indexed citations

Thakur, Ganesh A., et al.. (2021). Suppression of neuroinflammation by an allosteric agonist and positive allosteric modulator of the α7 nicotinic acetylcholine receptor GAT107. Journal of Neuroinflammation. 18(1). 99–99. 26 indexed citations

Cohen, Emiliano, et al.. (2020). GTL-1, a Calcium Activated TRPM Channel, Enhances Nociception. Frontiers in Pharmacology. 10. 1567–1567. 1 indexed citations

Rabinowitch, Ithai, Patrick Laurent, Denise S. Walker, et al.. (2016). Neuropeptide-Driven Cross-Modal Plasticity following Sensory Loss in Caenorhabditis elegans. PLoS Biology. 14(1). e1002348–e1002348. 25 indexed citations

Liewald, Jana, et al.. (2016). RIC-3 phosphorylation enables dual regulation of excitation and inhibition ofCaenorhabditis elegansmuscle. Molecular Biology of the Cell. 27(19). 2994–3003. 11 indexed citations

Rabinowitch, Ithai, et al.. (2014). Rewiring neural circuits by the insertion of ectopic electrical synapses in transgenic C. elegans. Nature Communications. 5(1). 4442–4442. 36 indexed citations

Nagy, Stanislav, et al.. (2013). The Caenorhabditis elegans interneuron ALA is (also) a high-threshold mechanosensor. BMC Neuroscience. 14(1). 156–156. 28 indexed citations

Husson, Steven, Wagner Steuer Costa, Sebastian Wabnig, et al.. (2012). Optogenetic Analysis of a Nociceptor Neuron and Network Reveals Ion Channels Acting Downstream of Primary Sensors. Current Biology. 22(9). 743–752. 60 indexed citations

Hall, David H. & Millet Treinin. (2011). How does morphology relate to function in sensory arbors?. Trends in Neurosciences. 34(9). 443–451. 39 indexed citations

10.

Oren‐Suissa, Meital, David H. Hall, Millet Treinin, Gidi Shemer, & Benjamin Podbilewicz. (2010). The Fusogen EFF-1 Controls Sculpting of Mechanosensory Dendrites. Science. 328(5983). 1285–1288. 130 indexed citations

11.

Chatzigeorgiou, Marios, Sungjae Yoo, Joseph D. Watson, et al.. (2010). Specific roles for DEG/ENaC and TRP channels in touch and thermosensation in C. elegans nociceptors. Nature Neuroscience. 13(7). 861–868. 193 indexed citations

12.

Smith, Cody J., Marios Chatzigeorgiou, Dror G. Feitelson, et al.. (2010). C. elegans multi-dendritic sensory neurons: Morphology and function. Molecular and Cellular Neuroscience. 46(1). 308–317. 122 indexed citations

13.

Biala, Yoav, Jana Liewald, Hagit Cohen Ben-Ami, Alexander Gottschalk, & Millet Treinin. (2008). The Conserved RIC-3 Coiled-Coil Domain Mediates Receptor-specific Interactions with Nicotinic Acetylcholine Receptors. Molecular Biology of the Cell. 20(5). 1419–1427. 18 indexed citations

14.

Treinin, Millet. (2008). RIC‐3 and nicotinic acetylcholine receptors: Biogenesis, properties, and diversity. Biotechnology Journal. 3(12). 1539–1547. 35 indexed citations

15.

Stetina, Stephen E. Von, Millet Treinin, & David M. Miller. (2005). The Motor Circuit. International review of neurobiology. 69. 125–167. 65 indexed citations

16.

Yassin, Lina M., et al.. (2001). Characterization of the DEG-3/DES-2 Receptor: A Nicotinic Acetylcholine Receptor That Mutates to Cause Neuronal Degeneration. Molecular and Cellular Neuroscience. 17(3). 589–599. 66 indexed citations

17.

Khatchatouriants, Artium, et al.. (2000). GFP Is a Selective Non-Linear Optical Sensor of Electrophysiological Processes in Caenorhabditis elegans. Biophysical Journal. 79(5). 2345–2352. 33 indexed citations

18.

Liu, Jun, Dieter Riemer, Millet Treinin, et al.. (2000). Essential Roles forCaenorhabditis elegansLamin Gene in Nuclear Organization, Cell Cycle Progression, and Spatial Organization of Nuclear Pore Complexes. Molecular Biology of the Cell. 11(11). 3937–3947. 345 indexed citations

19.

Treinin, Millet & Dror G. Feitelson. (1993). Unequal Cell Division as a Driving Force During Differentiation. Journal of Theoretical Biology. 160(1). 85–95.

20.

Treinin, Millet & Orgad Laub. (1987). Identification of a Promoter Element Located Upstream from the Hepatitis B Virus X Gene. Molecular and Cellular Biology. 7(1). 545–548. 44 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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