Sarah Halevi

632 citations
8 papers · 499 indexed · h-index 7
Topics
Ion channel regulation and function (3 papers)Genetics, Aging, and Longevity in Model Organisms (3 papers)Photoreceptor and optogenetics research (2 papers)
Partner nations
IsraelSpainUnited States

In The Last Decade

Sarah Halevi

8 papers receiving 481 citations

Peers

Sarah Halevi
Comparison fields: 5 of 54
  • Molecular Biology 375
  • Cellular and Molecular Neuroscience 155
  • Aging 116
  • Insect Science 90
  • Pharmacology 85
Replace Diego Rayes with:
Diego Rayes Argentina
C.D. Johnson United States
Philip Brackley United Kingdom
J P Walrond United States
Georgia Rapti Germany
Katherine J. Burton United States
Anna O. Burdina United States
Jill S. Wentzell United States
Kisang Kwon South Korea
Marlène Cassar United States
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Citations per field
00.5×1.5×
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Citations per year

Countries citing papers authored by Sarah Halevi

Since Specialization
Citations

This map shows the geographic impact of Sarah Halevi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah Halevi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah Halevi more than expected).

Fields of papers citing papers by Sarah Halevi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah Halevi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah Halevi. The network helps show where Sarah Halevi may publish in the future.

Co-authorship network of co-authors of Sarah Halevi

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah Halevi. A scholar is included among the top collaborators of Sarah Halevi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah Halevi. Sarah Halevi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
#WorkIndexed citations
1 153
2 203
3 10
4 66
5
Antibodies from ALS patients inhibit dopamine release mediated by L- type calcium channels [6] (multiple letters)
7
6 47
7 11
8 2

About Sarah Halevi

Sarah Halevi is a scholar working on Aging, Cellular and Molecular Neuroscience and Endocrine and Autonomic Systems, having authored 8 papers that have together received 499 indexed citations. Recurring topics across this work include Ion channel regulation and function (3 papers), Genetics, Aging, and Longevity in Model Organisms (3 papers) and Photoreceptor and optogenetics research (2 papers). The work is most often cited by research in Aging (116 citations), Cellular and Molecular Neuroscience (155 citations) and Endocrine and Autonomic Systems (54 citations). Sarah Halevi has collaborated with scholars based in Israel, Spain and United States. Frequent co-authors include Lina M. Yassin, Millet Treinin, Manuel Criado, F. Sala, Salvador Sala, Boaz Gillo, Tamar Kahan, Daphné Atlas, Michael Trus and Dror Tobi. Their work appears in journals such as Journal of Biological Chemistry, The EMBO Journal and Biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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