Mikael Lindberg

1.3k total citations
19 papers, 1.1k citations indexed

About

Mikael Lindberg is a scholar working on Molecular Biology, Neurology and Physiology. According to data from OpenAlex, Mikael Lindberg has authored 19 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 5 papers in Neurology and 2 papers in Physiology. Recurrent topics in Mikael Lindberg's work include Amyotrophic Lateral Sclerosis Research (5 papers), Parkinson's Disease Mechanisms and Treatments (4 papers) and Ubiquitin and proteasome pathways (3 papers). Mikael Lindberg is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (5 papers), Parkinson's Disease Mechanisms and Treatments (4 papers) and Ubiquitin and proteasome pathways (3 papers). Mikael Lindberg collaborates with scholars based in Sweden, United States and Germany. Mikael Lindberg's co-authors include Mikael Oliveberg, Lena Tibell, Peter M. Andersen, Arne Holmgren, Roberth Byström, Niklas Boknäs, Annika E. Wallberg, P. Andreas Jonsson, Stefan L. Marklund and Thomas Brännström and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Nature Communications.

In The Last Decade

Mikael Lindberg

17 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mikael Lindberg Sweden 14 619 519 191 173 173 19 1.1k
Lisa J. Whitson United States 10 483 0.8× 275 0.5× 111 0.6× 119 0.7× 169 1.0× 10 749
Basant K. Patel India 15 501 0.8× 855 1.6× 178 0.9× 235 1.4× 259 1.5× 27 1.2k
Eiichi Tokuda Japan 18 452 0.7× 241 0.5× 139 0.7× 117 0.7× 175 1.0× 28 769
Se Hui Sohn United States 5 376 0.6× 188 0.4× 98 0.5× 99 0.6× 166 1.0× 8 567
Rachel L. Redler United States 10 343 0.6× 291 0.6× 99 0.5× 67 0.4× 139 0.8× 15 612
Marjatta Son United States 12 351 0.6× 253 0.5× 118 0.6× 98 0.6× 153 0.9× 20 647
Tania Massignan Italy 19 261 0.4× 1.0k 1.9× 99 0.5× 341 2.0× 291 1.7× 28 1.4k
Joy Mitra United States 19 497 0.8× 777 1.5× 221 1.2× 63 0.4× 108 0.6× 39 1.2k
Pavana M. Hegde United States 22 464 0.7× 1.1k 2.2× 224 1.2× 62 0.4× 117 0.7× 30 1.5k
Jeffrey A. Cohlberg United States 12 294 0.5× 555 1.1× 52 0.3× 77 0.4× 265 1.5× 18 1.1k

Countries citing papers authored by Mikael Lindberg

Since Specialization
Citations

This map shows the geographic impact of Mikael Lindberg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mikael Lindberg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mikael Lindberg more than expected).

Fields of papers citing papers by Mikael Lindberg

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mikael Lindberg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mikael Lindberg. The network helps show where Mikael Lindberg may publish in the future.

Co-authorship network of co-authors of Mikael Lindberg

This figure shows the co-authorship network connecting the top 25 collaborators of Mikael Lindberg. A scholar is included among the top collaborators of Mikael Lindberg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mikael Lindberg. Mikael Lindberg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Lindberg, Mikael, et al.. (2019). Bax to the future – A novel, high-yielding approach for purification and expression of full-length Bax protein for structural studies. Protein Expression and Purification. 158. 20–26. 7 indexed citations
2.
Zhang, Jin, Christin Grundström, Kristoffer Brännström, et al.. (2018). Interspecies Variation between Fish and Human Transthyretins in Their Binding of Thyroid-Disrupting Chemicals. Environmental Science & Technology. 52(20). 11865–11874. 15 indexed citations
3.
Karlberg, T., P. Hornyak, A.F. Pinto, et al.. (2018). 14-3-3 proteins activate Pseudomonas exotoxins-S and -T by chaperoning a hydrophobic surface. Nature Communications. 9(1). 3785–3785. 40 indexed citations
4.
Lindberg, Mikael. (2017). Styrning och ledning.
5.
Hall, Michael, Christin Grundström, A. Begum, et al.. (2016). Structural basis for glutathione-mediated activation of the virulence regulatory protein PrfA in Listeria. Proceedings of the National Academy of Sciences. 113(51). 14733–14738. 67 indexed citations
6.
Lindberg, Mikael, et al.. (2015). Mastermind-Like 1 Is Ubiquitinated: Functional Consequences for Notch Signaling. PLoS ONE. 10(7). e0134013–e0134013. 10 indexed citations
7.
Lindberg, Mikael, et al.. (2012). GCN5 acetylates and regulates the stability of the oncoprotein E2A-PBX1 in acute lymphoblastic leukemia. Leukemia. 27(3). 578–585. 54 indexed citations
8.
Lindberg, Mikael, et al.. (2011). Ubiquitination of Notch1 is regulated by MAML1-mediated p300 acetylation of Notch1. Biochemical and Biophysical Research Communications. 416(3-4). 300–306. 25 indexed citations
9.
Lindberg, Mikael, et al.. (2010). SUMO modification regulates the transcriptional activity of MAML1. The FASEB Journal. 24(7). 2396–2404. 20 indexed citations
10.
Dancy, Beverley M., et al.. (2009). The transcriptional coactivator MAML1 regulates p300 autoacetylation and HAT activity. Nucleic Acids Research. 37(9). 2996–3006. 53 indexed citations
11.
Lindberg, Mikael, et al.. (2009). GSK3  is a negative regulator of the transcriptional coactivator MAML1. Nucleic Acids Research. 37(20). 6691–6700. 27 indexed citations
12.
Jonsson, P. Andreas, Karin S. Graffmo, Peter M. Andersen, et al.. (2005). Disulphide-reduced superoxide dismutase-1 in CNS of transgenic amyotrophic lateral sclerosis models. Brain. 129(2). 451–464. 196 indexed citations
13.
Lindberg, Mikael. (2005). Memory recognition for monolingual and bilingual speakers. KTH Publication Database DiVA (KTH Royal Institute of Technology).
14.
Lindberg, Mikael, Roberth Byström, Niklas Boknäs, Peter M. Andersen, & Mikael Oliveberg. (2005). Systematically perturbed folding patterns of amyotrophic lateral sclerosis (ALS)-associated SOD1 mutants. Proceedings of the National Academy of Sciences. 102(28). 9754–9759. 197 indexed citations
15.
Lindberg, Mikael, et al.. (2004). Coexpression of yeast copper chaperone (yCCS) and CuZn-superoxide dismutases in Escherichia coli yields protein with high copper contents. Protein Expression and Purification. 37(2). 311–319. 29 indexed citations
16.
Lindberg, Mikael, et al.. (2004). Folding of human superoxide dismutase: Disulfide reduction prevents dimerization and produces marginally stable monomers. Proceedings of the National Academy of Sciences. 101(45). 15893–15898. 158 indexed citations
17.
Lindberg, Mikael, Lena Tibell, & Mikael Oliveberg. (2002). Common denominator of Cu/Zn superoxide dismutase mutants associated with amyotrophic lateral sclerosis: Decreased stability of the apo state. Proceedings of the National Academy of Sciences. 99(26). 16607–16612. 176 indexed citations
18.
19.
Lindberg, Mikael, et al.. (2001). AXE 810—The evolution continues. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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