Michael Rapé
- Cell Biology top 0.1%
- Microtubule and mitosis dynamics 24
- Endoplasmic Reticulum Stress and Disease 8
- Molecular Biology top 0.2%
- Ubiquitin and proteasome pathways 66
- Protein Degradation and Inhibitors 18
- Glycosylation and Glycoproteins Research 9
- Histone Deacetylase Inhibitors Research 6
- Oncology top 0.5%
- Cancer-related Molecular Pathways 12
- Aging top 2%
- Epidemiology top 1%
- Autophagy in Disease and Therapy 14
- Co-authors
- David KomanderRichard G. YauYihong YeStefan JentschMarc W. KirschnerKatherine E. WickliffeAdam J. WilliamsonThorsten Hoppe
- Partner nations
- United StatesGermanyCanada
In The Last Decade
Michael Rapé
79 papers receiving 12.8k citations
Hit Papers
Peers
Comparison fields: 5 of 127
- Cell Biology 3.6k
- Molecular Biology 11.4k
- Oncology 3.3k
- Aging 119
- Epidemiology 2.2k
Countries citing papers authored by Michael Rapé
This map shows the geographic impact of Michael Rapé's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Rapé with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Rapé more than expected).
Fields of papers citing papers by Michael Rapé
This network shows the impact of papers produced by Michael Rapé. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Rapé. The network helps show where Michael Rapé may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Michael Rapé, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 3 | |
| 3 | 2021 | 22 | |
| 4 | 2020 | 38 | |
| 5 | 2019 | 113 | |
| 6 | 2017 | 36 | |
| 7 | 2016 | 98 | |
| 8 | 2014 | 63 | |
| 9 | 2014 | 373 | |
| 10 | Ubiquitin-dependent regulation of COPII coat size and functionbreakdown → | 2012 | 257 |
| 11 | 2011 | 75 | |
| 12 | 2011 | 231 | |
| 13 | 2010 | 14 | |
| 14 | 2009 | 244 | |
| 15 | 2009 | 15 | |
| 16 | 2006 | 234 | |
| 17 | 2004 | 49 | |
| 18 | 2002 | 92 | |
| 19 | 2001 | 130 | |
| 20 | 1998 | 36 |
About Michael Rapé
Michael Rapé is a scholar working on Cell Biology, Molecular Biology and Oncology, having authored 81 papers that have together received 12.9k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (66 papers), Microtubule and mitosis dynamics (24 papers), Protein Degradation and Inhibitors (18 papers), Autophagy in Disease and Therapy (14 papers), Cancer-related Molecular Pathways (12 papers), Glycosylation and Glycoproteins Research (9 papers), Endoplasmic Reticulum Stress and Disease (8 papers) and Histone Deacetylase Inhibitors Research (6 papers). The work is most often cited by research in Cell Biology (3.6k citations), Molecular Biology (11.4k citations) and Oncology (3.3k citations). Michael Rapé has collaborated with scholars based in United States, Germany and Canada. Frequent co-authors include David Komander, Richard G. Yau, Yihong Ye, Stefan Jentsch, Marc W. Kirschner, Katherine E. Wickliffe, Adam J. Williamson, Thorsten Hoppe, Ling Song and Sashank Reddy. Their work appears in journals such as Cell, Nature, Molecular Cell, Trends in Cell Biology and Current Opinion in Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.