Katherine E. Wickliffe

4.8k citations
22 papers · 3.7k indexed · 3 hit papers · h-index 20
Topics
Cell death mechanisms and regulation (9 papers)Ubiquitin and proteasome pathways (9 papers)Cancer-related Molecular Pathways (6 papers)

In The Last Decade

Katherine E. Wickliffe

21 papers receiving 3.6k citations

Hit Papers

Activity of Protein Kinase RIPK3 Determines Whether Cells...2012202620162021201420192012100200300400500

Peers

Katherine E. Wickliffe
Comparison fields: 5 of 111
  • Molecular Biology 2.9k
  • Immunology 1.3k
  • Oncology 613
  • Epidemiology 595
  • Cell Biology 591
Replace Dan T. Bergstralh with:
Dan T. Bergstralh United States
Isabelle S. Lucet Australia
Tamás Oravecz United States
Marc Van de Craen Belgium
Nicolas Bidère France
Rossana Zaru United Kingdom
Emmanuel Di Valentin Belgium
Margaret Hugunin United States
Taoyong Chen China
Vivien R. Sutton Australia
Katherine E. Wickliffe relative to Dan T. Bergstralh United States Dan T. Bergstralh's profile →
Citations per field
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Dan T. Bergstralh · 1×
Citations per year

Countries citing papers authored by Katherine E. Wickliffe

Since Specialization
Citations

This map shows the geographic impact of Katherine E. Wickliffe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katherine E. Wickliffe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katherine E. Wickliffe more than expected).

Fields of papers citing papers by Katherine E. Wickliffe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katherine E. Wickliffe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katherine E. Wickliffe. The network helps show where Katherine E. Wickliffe may publish in the future.

Co-authorship network of co-authors of Katherine E. Wickliffe

This figure shows the co-authorship network connecting the top 25 collaborators of Katherine E. Wickliffe. A scholar is included among the top collaborators of Katherine E. Wickliffe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katherine E. Wickliffe. Katherine E. Wickliffe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 8
3 21
4
Cleavage of RIPK1 by caspase-8 is crucial for limiting apoptosis and necroptosisbreakdown →
407
5 289
6 27
7 307
8 62
9
Activity of Protein Kinase RIPK3 Determines Whether Cells Die by Necroptosis or Apoptosisbreakdown →
531
10 63
11
Ubiquitin-dependent regulation of COPII coat size and functionbreakdown →
257
12 27
13 231
14 143
15 321
16 244
17 28
18 451
19 64
20 19

About Katherine E. Wickliffe

Katherine E. Wickliffe is a scholar working on Cell Biology, Oncology and Molecular Biology, having authored 22 papers that have together received 3.7k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (9 papers), Ubiquitin and proteasome pathways (9 papers) and Cancer-related Molecular Pathways (6 papers). The work is most often cited by research in Immunology (1.3k citations), Molecular Biology (2.9k citations) and Cell Biology (591 citations). Katherine E. Wickliffe has collaborated with scholars based in United States, France and Australia. Frequent co-authors include Michael Rapé, Vishva M. Dixit, Kim Newton, Joshua D. Webster, Debra L. Dugger, Merone Roose‐Girma, Allie Maltzman, Adam J. Williamson, László G. Kömüves and Stephen H. Leppla. Their work appears in journals such as Nature, Science and Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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