Martin Rechsteiner
- Cell Biology top 0.1%
- Endoplasmic Reticulum Stress and Disease 25
- Molecular Biology top 0.2%
- Ubiquitin and proteasome pathways 78
- Glycosylation and Glycoproteins Research 30
- Biochemical and Molecular Research 14
- RNA modifications and cancer 14
- Oncology top 0.5%
- Peptidase Inhibition and Analysis 32
- Aging top 1%
- Immunology top 1%
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- Genetics and Neurodevelopmental Disorders 12
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- Enzyme Structure and Function 11
- Co-authors
- Scott W. RogersG PrattWolfgang DubielR HoughQuinn L. DeverauxVicença UstrellLaura HoffmanClaudio Realini
- Partner nations
- United StatesGermanyJapan
In The Last Decade
Martin Rechsteiner
148 papers receiving 14.1k citations
Hit Papers
Peers
Comparison fields: 5 of 131
- Cell Biology 3.9k
- Molecular Biology 12.3k
- Oncology 3.0k
- Aging 187
- Immunology 1.7k
Countries citing papers authored by Martin Rechsteiner
This map shows the geographic impact of Martin Rechsteiner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Rechsteiner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Rechsteiner more than expected).
Fields of papers citing papers by Martin Rechsteiner
This network shows the impact of papers produced by Martin Rechsteiner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Rechsteiner. The network helps show where Martin Rechsteiner may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Martin Rechsteiner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | The ubiquitin-proteasome system and disease | 2008 | 1 |
| 2 | 2008 | 35 | |
| 3 | 2006 | 29 | |
| 4 | 2006 | 7 | |
| 5 | Ubiquitin and the chemistry of life | 2005 | 6 |
| 6 | 2004 | 9 | |
| 7 | 2004 | 83 | |
| 8 | 2002 | 118 | |
| 9 | 2001 | 90 | |
| 10 | 2000 | 50 | |
| 11 | 2000 | 22 | |
| 12 | 1999 | 9 | |
| 13 | 1997 | 142 | |
| 14 | 1997 | 65 | |
| 15 | 1996 | 26 | |
| 16 | 1996 | 6 | |
| 17 | 1994 | 36 | |
| 18 | 1991 | 8 | |
| 19 | 1990 | 6 | |
| 20 | 1988 | 121 |
About Martin Rechsteiner
Martin Rechsteiner is a scholar working on Cell Biology, Molecular Biology and Oncology, having authored 148 papers that have together received 14.5k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (78 papers), Peptidase Inhibition and Analysis (32 papers), Glycosylation and Glycoproteins Research (30 papers), Endoplasmic Reticulum Stress and Disease (25 papers), Biochemical and Molecular Research (14 papers), RNA modifications and cancer (14 papers), Genetics and Neurodevelopmental Disorders (12 papers) and Enzyme Structure and Function (11 papers). The work is most often cited by research in Cell Biology (3.9k citations), Molecular Biology (12.3k citations) and Oncology (3.0k citations). Martin Rechsteiner has collaborated with scholars based in United States, Germany and Japan. Frequent co-authors include Scott W. Rogers, G Pratt, Wolfgang Dubiel, R Hough, Quinn L. Deveraux, Vicença Ustrell, Laura Hoffman, Claudio Realini, Cecile M. Pickart and Katherine Ferrell. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.