Michael G. Anderson

6.8k total citations
118 papers, 4.6k citations indexed

About

Michael G. Anderson is a scholar working on Ophthalmology, Molecular Biology and Epidemiology. According to data from OpenAlex, Michael G. Anderson has authored 118 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Ophthalmology, 46 papers in Molecular Biology and 22 papers in Epidemiology. Recurrent topics in Michael G. Anderson's work include Glaucoma and retinal disorders (49 papers), Retinal Development and Disorders (22 papers) and Retinal Diseases and Treatments (18 papers). Michael G. Anderson is often cited by papers focused on Glaucoma and retinal disorders (49 papers), Retinal Development and Disorders (22 papers) and Retinal Diseases and Treatments (18 papers). Michael G. Anderson collaborates with scholars based in United States, United Kingdom and Russia. Michael G. Anderson's co-authors include Simon W. M. John, Richard S. Smith, Richard T. Libby, Bo Chang, Wayne A. Johnson, Norman L. Hawes, Abbot F. Clark, О. В. Савинова, Ioan M Cosma and Adam Hedberg‐Buenz and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Journal of Clinical Investigation.

In The Last Decade

Michael G. Anderson

116 papers receiving 4.5k citations

Peers

Michael G. Anderson

OPHTH

CMN

RNMI

Peter Munro United Kingdom

Susanne Kohl Germany

Elena Vecino Spain

Jacque L. Duncan United States

Eric F. Wawrousek United States

Chris F. Inglehearn United Kingdom

Birgit Lorenz Germany

J. Fielding Hejtmancik United States

Stephen P. Daiger United States

Russell N. Van Gelder United States

Peter Munro United Kingdom

Michael G. Anderson

2.3k ×1.0

904 ×0.4

OPHTH

744 ×1.1

CMN

1.1k ×2.0

RNMI

447 ×0.9

Citations per year, relative to Michael G. Anderson Michael G. Anderson (= 1×) peers Peter Munro

Countries citing papers authored by Michael G. Anderson

Since Specialization

Citations

This map shows the geographic impact of Michael G. Anderson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael G. Anderson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael G. Anderson more than expected).

Fields of papers citing papers by Michael G. Anderson

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael G. Anderson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael G. Anderson. The network helps show where Michael G. Anderson may publish in the future.

Co-authorship network of co-authors of Michael G. Anderson

This figure shows the co-authorship network connecting the top 25 collaborators of Michael G. Anderson. A scholar is included among the top collaborators of Michael G. Anderson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael G. Anderson. Michael G. Anderson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Meyer, Kacie J., et al.. (2024). Minimal phenotypes in transgenic mice with the human LOXL1/LOXL1-AS1 locus associated with exfoliation glaucoma. Vision Research. 223. 108464–108464.

Meyer, Kacie J., John H. Fingert, & Michael G. Anderson. (2024). Lack of evidence for GWAS signals of exfoliation glaucoma working via monogenic loss-of-function mutation in the nearest gene. Human Molecular Genetics. 2 indexed citations

Meyer, Kacie J., et al.. (2023). Anterior chamber depth in mice is controlled by several quantitative trait loci. PLoS ONE. 18(8). e0286897–e0286897. 2 indexed citations

Jiang, Jingwei, John J. Reho, Sajag Bhattarai, et al.. (2021). Endothelial BBSome is essential for vascular, metabolic, and retinal functions. Molecular Metabolism. 53. 101308–101308. 10 indexed citations

Meyer, Kacie J., et al.. (2021). Quantification and image-derived phenotyping of retinal ganglion cell nuclei in the nee mouse model of congenital glaucoma. Experimental Eye Research. 212. 108774–108774. 2 indexed citations

Evans, Lucy, Shu Wu, Adam Hedberg‐Buenz, et al.. (2020). Modulation of Post-Traumatic Immune Response Using the IL-1 Receptor Antagonist Anakinra for Improved Visual Outcomes. Journal of Neurotrauma. 37(12). 1463–1480. 25 indexed citations

Harper, Matthew M., Danielle S. Rudd, Kacie J. Meyer, et al.. (2020). Identification of chronic brain protein changes and protein targets of serum auto-antibodies after blast-mediated traumatic brain injury. Heliyon. 6(2). e03374–e03374. 26 indexed citations

Anderson, Michael G., et al.. (2019). Correlation of retinal ganglion cell metrics made in vivo using SD-OCT versus ex vivo using quantitative histology in Diversity Outbred mice. Investigative Ophthalmology & Visual Science. 60(9). 5604–5604. 1 indexed citations

Dutca, Laura M., et al.. (2018). Effects of sustained daily latanoprost application on anterior chamber anatomy and physiology in mice. Scientific Reports. 8(1). 13088–13088.

10.

Meyer, Kacie J., et al.. (2018). Neprilysin inhibition promotes corneal wound healing. Scientific Reports. 8(1). 14385–14385. 8 indexed citations

11.

Gramlich, Oliver W., Qiong Ding, Michael G. Anderson, & Markus H. Kuehn. (2015). Adaptive immune responses in glaucoma promote IOP-independent RCG loss. Investigative Ophthalmology & Visual Science. 56(7). 1694–1694. 1 indexed citations

12.

Howell, Gareth R., Ileana Soto, Xianjun Zhu, et al.. (2012). Radiation treatment inhibits monocyte entry into the optic nerve head and prevents neuronal damage in a mouse model of glaucoma. Journal of Clinical Investigation. 122(4). 1246–1261. 183 indexed citations

13.

Zode, Gulab, Markus H. Kuehn, Darryl Nishimura, et al.. (2011). Reduction of ER stress via a chemical chaperone prevents disease phenotypes in a mouse model of primary open angle glaucoma. Journal of Clinical Investigation. 121(9). 3542–3553. 212 indexed citations

14.

Gallant, Tricia Bertram, et al.. (2011). Academic Integrity in a Mandatory Physics Lab: The Influence of Post-Graduate Aspirations and Grade Point Averages. Science and Engineering Ethics. 19(1). 219–235. 3 indexed citations

15.

Haraszti, Tamás, M. Grunze, & Michael G. Anderson. (2010). STXMPy: a new software package for automated region of interest selection and statistical analysis of XANES data. Chemistry Central Journal. 4(1). 11–11. 5 indexed citations

16.

Hedberg‐Buenz, Adam, et al.. (2010). Elevated Oxidative Membrane Damage Associated with Genetic Modifiers of Lyst-Mutant Phenotypes. PLoS Genetics. 6(7). e1001008–e1001008. 25 indexed citations

17.

Howell, Gareth R., Richard T. Libby, Jeffrey K. Marchant, et al.. (2007). Absence of glaucoma in DBA/2J mice homozygous for wild-type versions of Gpnmb and Tyrp1. BMC Genetics. 8(1). 45–45. 114 indexed citations

18.

Anderson, Michael G., Charles F. Hinderliter, & James R. Misanin. (2005). The effects of chronic water deprivation on metabolic rate and long-trace taste-aversion conditioning in rats. Neurobiology of Learning and Memory. 85(3). 199–205. 8 indexed citations

19.

Anderson, Michael G., et al.. (2003). A case of severe monkeypox virus disease in an American child: emerging infections and changing professional values. The Pediatric Infectious Disease Journal. 22(12). 1093–1096. 84 indexed citations

20.

Anderson, Michael G., Richard S. Smith, Norman L. Hawes, et al.. (2001). Mutations in genes encoding melanosomal proteins cause pigmentary glaucoma in DBA/2J mice. Nature Genetics. 30(1). 81–85. 361 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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