Polly J. Ferguson

13.5k total citations · 1 hit paper
95 papers, 6.2k citations indexed

About

Polly J. Ferguson is a scholar working on Rheumatology, Molecular Biology and Surgery. According to data from OpenAlex, Polly J. Ferguson has authored 95 papers receiving a total of 6.2k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Rheumatology, 34 papers in Molecular Biology and 31 papers in Surgery. Recurrent topics in Polly J. Ferguson's work include Osteomyelitis and Bone Disorders Research (41 papers), Infectious Diseases and Tuberculosis (24 papers) and Inflammasome and immune disorders (23 papers). Polly J. Ferguson is often cited by papers focused on Osteomyelitis and Bone Disorders Research (41 papers), Infectious Diseases and Tuberculosis (24 papers) and Inflammasome and immune disorders (23 papers). Polly J. Ferguson collaborates with scholars based in United States, Canada and Germany. Polly J. Ferguson's co-authors include Frank T. Saulsbury, Thaddeus E. Kelly, Hans D. Ochs, Craig L. Bennett, Mary E. Brunkow, Phillip F. Chance, Fred Ramsdell, Luke Whitesell, Hatem El‐Shanti and T. Shawn Sato and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Journal of Clinical Investigation.

In The Last Decade

Polly J. Ferguson

91 papers receiving 6.1k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) is caused by mutations of FOXP3

2001 2.5k citations Polly J. Ferguson, Frank T. Saulsbury et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Polly J. Ferguson United States	34	3.1k	1.9k	1.7k	1.4k	677	95	6.2k
Christian M. Hedrich Germany	47	2.3k 0.8×	2.6k 1.4×	1.4k 0.8×	1.4k 1.0×	301 0.4×	162	5.7k
Timothy R. D. J. Radstake Netherlands	39	2.5k 0.8×	1.5k 0.8×	1.3k 0.8×	357 0.3×	406 0.6×	174	5.4k
Giuliana Guggino Italy	40	2.5k 0.8×	1.5k 0.8×	1.1k 0.6×	607 0.4×	338 0.5×	158	5.1k
Thierry Martin France	33	2.2k 0.7×	1.3k 0.7×	1.3k 0.8×	478 0.3×	295 0.4×	152	5.2k
Michèlle Rosenzwajg France	39	3.2k 1.0×	1.0k 0.5×	959 0.6×	585 0.4×	593 0.9×	103	6.0k
Jordi Yagüe Spain	41	2.4k 0.8×	851 0.5×	2.3k 1.4×	585 0.4×	334 0.5×	146	5.8k
Maxime Bréban France	42	2.4k 0.8×	3.5k 1.9×	796 0.5×	666 0.5×	557 0.8×	172	5.5k
Seza Özen Türkiye	50	3.2k 1.0×	1.9k 1.0×	5.7k 3.4×	1.7k 1.2×	768 1.1×	335	9.5k
Michael Mähler United States	47	1.9k 0.6×	2.6k 1.4×	1.1k 0.7×	598 0.4×	853 1.3×	190	6.3k
Ivona Aksentijevich United States	44	4.3k 1.4×	1.3k 0.7×	5.9k 3.6×	949 0.7×	560 0.8×	144	8.4k

Countries citing papers authored by Polly J. Ferguson

Since Specialization

Citations

This map shows the geographic impact of Polly J. Ferguson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Polly J. Ferguson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Polly J. Ferguson more than expected).

Fields of papers citing papers by Polly J. Ferguson

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Polly J. Ferguson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Polly J. Ferguson. The network helps show where Polly J. Ferguson may publish in the future.

Co-authorship network of co-authors of Polly J. Ferguson

This figure shows the co-authorship network connecting the top 25 collaborators of Polly J. Ferguson. A scholar is included among the top collaborators of Polly J. Ferguson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Polly J. Ferguson. Polly J. Ferguson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Nuruzzaman, Farzana, T. Shawn Sato, Jennifer Stimec, et al.. (2025). Preliminary validation of a web-based MRI scoring system for children with chronic nonbacterial osteomyelitis (ChRonic nonbacterial Osteomyelitis Magnetic Resonance Imaging Scoring: CROMRIS). Pediatric Rheumatology. 23(1). 85–85.

Ferguson, Polly J., et al.. (2025). Gorham-Stout disease of the craniovertebral junction causing basilar impression and Chiari malformation type I: illustrative case. Journal of Neurosurgery Case Lessons. 9(6).

Kim, Sujin, et al.. (2024). Accuracy and Performance Characteristics of Administrative Codes for the Diagnosis of Autoinflammatory Syndromes. JCR Journal of Clinical Rheumatology. 31(2). 60–64.

Rodriguez, Saul, Shaunik Sharma, Kyle R. Jackson, et al.. (2024). Neuroprotective effects of naltrexone in a mouse model of post-traumatic seizures. Scientific Reports. 14(1). 13507–13507. 5 indexed citations

Hedrich, Christian M., Michael W. Beresford, Fatma Dedeoğlu, et al.. (2023). Gathering expert consensus to inform a proposed trial in chronic nonbacterial osteomyelitis (CNO). Clinical Immunology. 251. 109344–109344. 10 indexed citations

Luo, Zili, Michael S. Chimenti, John T. Harty, et al.. (2023). Selective neuroimmune modulation by type I interferon drives neuropathology and neurologic dysfunction following traumatic brain injury. Acta Neuropathologica Communications. 11(1). 134–134. 8 indexed citations

Chimenti, Michael S., et al.. (2021). Traumatic brain injury results in unique microglial and astrocyte transcriptomes enriched for type I interferon response. Journal of Neuroinflammation. 18(1). 151–151. 59 indexed citations

Sato, T. Shawn & Polly J. Ferguson. (2021). Whole-body MRI Imaging Is an Essential Tool in Diagnosing and Monitoring Patients With Sterile Osteomyelitis. The Journal of Rheumatology. 48(5). 635–637. 3 indexed citations

Nuruzzaman, Farzana, Yongdong Zhao, & Polly J. Ferguson. (2021). Chronic Nonbacterial Osteomyelitis. Rheumatic Disease Clinics of North America. 47(4). 691–705. 10 indexed citations

10.

Evans, Lucy, Shu Wu, Adam Hedberg‐Buenz, et al.. (2020). Modulation of Post-Traumatic Immune Response Using the IL-1 Receptor Antagonist Anakinra for Improved Visual Outcomes. Journal of Neurotrauma. 37(12). 1463–1480. 25 indexed citations

11.

Li, Suzanne C., Kathryn S. Torok, C. Egla Rabinovich, et al.. (2019). Initial Results from a Pilot Comparative Effectiveness Study of 3 Methotrexate-based Consensus Treatment Plans for Juvenile Localized Scleroderma. The Journal of Rheumatology. 47(8). 1242–1252. 20 indexed citations

12.

Newell, Elizabeth A., et al.. (2019). A Mouse Model for Juvenile, Lateral Fluid Percussion Brain Injury Reveals Sex-Dependent Differences in Neuroinflammation and Functional Recovery. Journal of Neurotrauma. 37(4). 635–646. 24 indexed citations

13.

Cox, Allison, Yongdong Zhao, & Polly J. Ferguson. (2017). Chronic Recurrent Multifocal Osteomyelitis and Related Diseases—Update on Pathogenesis. Current Rheumatology Reports. 19(4). 18–18. 33 indexed citations

14.

Khanna, Geetika & Polly J. Ferguson. (2006). MRI of Diabetic Cheiroarthropathy. American Journal of Roentgenology. 188(1). W94–W95. 15 indexed citations

15.

Su, Kaihong, Xiaoli Li, Jeffrey C. Edberg, et al.. (2004). A Promoter Haplotype of the Immunoreceptor Tyrosine-Based Inhibitory Motif-Bearing FcγRIIb Alters Receptor Expression and Associates with Autoimmunity. II. Differential Binding of GATA4 and Yin-Yang1 Transcription Factors and Correlated Receptor Expression and Function. The Journal of Immunology. 172(11). 7192–7199. 89 indexed citations

16.

Su, Kaihong, Jianming Wu, Jeffrey C. Edberg, et al.. (2004). A Promoter Haplotype of the Immunoreceptor Tyrosine-Based Inhibitory Motif-Bearing FcγRIIb Alters Receptor Expression and Associates with Autoimmunity. I. Regulatory FCGR2B Polymorphisms and Their Association with Systemic Lupus Erythematosus. The Journal of Immunology. 172(11). 7186–7191. 142 indexed citations

17.

Ferguson, Polly J., et al.. (1997). The Primate Erythrocyte Complement Receptor (CR1) as a Privileged Site: Binding of Immunoglobulin G to Erythrocyte CR1 Does Not Target Erythrocytes for Phagocytosis. Blood. 89(3). 1068–1077. 35 indexed citations

18.

Ferguson, Polly J., Frank T. Saulsbury, Scott F. Dowell, et al.. (1996). Prevalence of human parvovirus B19 infection in children with Henoch‐Schönlein purpura. Arthritis & Rheumatism. 39(5). 880–881. 30 indexed citations

19.

Ferguson, Polly J. & Frank T. Saulsbury. (1996). SUCCESSFUL TREATMENT OF CHRONIC MYCOBACTERIUM ABSCESSUS OTITIS MEDIA WITH CLARITHROMYCIN. The Pediatric Infectious Disease Journal. 15(4). 384–385. 10 indexed citations

20.

Taylor, Ronald P. & Polly J. Ferguson. (1995). Primate Erythrocyte (E) Complement Receptor (CR1) as an Anchor Site for Bispecific-Based Therapies to Clear Pathogens or Autoantibodies Safely from the Circulation. Journal of Hematotherapy. 4(5). 357–362. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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