Bo Chang

11.7k total citations · 2 hit papers
162 papers, 9.0k citations indexed

About

Bo Chang is a scholar working on Molecular Biology, Ophthalmology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Bo Chang has authored 162 papers receiving a total of 9.0k indexed citations (citations by other indexed papers that have themselves been cited), including 130 papers in Molecular Biology, 51 papers in Ophthalmology and 32 papers in Cellular and Molecular Neuroscience. Recurrent topics in Bo Chang's work include Retinal Development and Disorders (85 papers), Retinal Diseases and Treatments (37 papers) and Photoreceptor and optogenetics research (26 papers). Bo Chang is often cited by papers focused on Retinal Development and Disorders (85 papers), Retinal Diseases and Treatments (37 papers) and Photoreceptor and optogenetics research (26 papers). Bo Chang collaborates with scholars based in United States, China and Japan. Bo Chang's co-authors include Norman L. Hawes, John R. Heckenlively, Muriel T. Davisson, Steven Nusinowitz, R.E. Hurd, Simon W. M. John, Thomas H. Roderick, Richard S. Smith, Michael G. Anderson and William W. Hauswirth and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Bo Chang

155 papers receiving 8.9k citations

Hit Papers

Retinal degeneration mutants in the mouse 1999 2026 2008 2017 2002 1999 200 400 600

Peers

Bo Chang
Comparison fields: 5 of 140
  • Molecular Biology 7.3k
  • Ophthalmology 3.3k
  • Cellular and Molecular Neuroscience 2.1k
  • Genetics 1.3k
  • Cell Biology 973
Replace Mathias W. Seeliger with:
Mathias W. Seeliger Germany
Richard G. Weleber United States
Bernd Wissinger Germany
Paul A. Sieving United States
Arthur A. Bergen Netherlands
Shomi S. Bhattacharya United Kingdom
Graham E. Holder United Kingdom
Stephen H. Tsang United States
Christian Grimm Switzerland
John R. Heckenlively United States
Mathias W. Seeliger Germany View profile →
Citations per field, relative to Bo Chang
Bo Chang · 1×
Citations per year, relative to Bo Chang
Bo Chang · 1×

Countries citing papers authored by Bo Chang

Since Specialization
Citations

This map shows the geographic impact of Bo Chang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bo Chang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bo Chang more than expected).

Fields of papers citing papers by Bo Chang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bo Chang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bo Chang. The network helps show where Bo Chang may publish in the future.

Co-authorship network of co-authors of Bo Chang

This figure shows the co-authorship network connecting the top 25 collaborators of Bo Chang. A scholar is included among the top collaborators of Bo Chang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bo Chang. Bo Chang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 0
2 5
3 2
4
Survey of Common Genetic Eye Diseases in Mouse Strains
1
5
Loss of Lysophosphatidylcholine Acyltransferase 1 (LPCAT1) Leads to Photoreceptor Degeneration in rd11 Mice
1
6
Gene therapy following subretinal AAV5 vector delivery is not affected by a previous intravitreal AAV5 vector administration in the partner eye.
43
7
Long Term Rescue Following AAV-Mediated Cone Targeting Gene Therapy to Cpfl5 Mice, a Model for Human Achromatopsia 2 With Cnga3 Mutation
1
8
AAV-Mediated Gene Therapy Restores Cone System Functions in cpfl5 Mouse, a Model of Human Achromatopsia With CNGA3 Mutation
2
9
Oligonucleotide Microarray Based Expression Analysis of the Cpfl1 Mutant - a Mouse Model of Cone and Cone-Rod Dystrophies
1
10
Supernormal Dark-Adapted Electroretinogram b-wave in Hyperpolarization-Activated Cyclic Nucleotide-Gated Channel 1 (HCN1) Knockout Mice
1
11
Retinal Degeneration in the Rd16 Mouse Is Associated With a Deletion in a Novel Centrosomal Protein CEP290/NPHP6 That Interacts With Retinitis Pigmentosa GTPase Regulator (RPGR)
0
12
Retinal degeneration 12 (rd12): a new, spontaneously arising mouse model for human Leber congenital amaurosis (LCA).
182
13
Long Term Vision Restoration by Gene Therapy in the rd12 Mouse Model of RPE65 Leber Congenital Amaurosis
1
14
Cone Photoreceptor Function Loss–3 (Cpfl3), a New Mouse Model of Achromatopsia Due to Missense Mutation in GNAT2
1
15
Selective Loss Of ERG b–Wave Caused By An Autosomal Recessive Mutation In Mice.
2
16
Developmental Analysis of the rd10 Mouse
1
17
Comparison of Electroretinographic Responses across Eleven Normal Inbred Mouse Strains
2
18
Detect Rod- and Cone-driven Signals with Scotopic Flicker Electroretinogram (ERG) in Mice
1
19
A Point Mutation in the Rpe65 Gene Causes Retinal Degeneration (rd12) in Mice
6
20
A New Mouse Model of Retinal Degeneration (rd11)
3

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026