Michael D. Kalos

26.8k citations
117 papers · 22.5k · 14 hit papers · h-index 52

Impact in

  • Oncology top 0.05%
    • CAR-T cell therapy research
    • Cancer Immunotherapy and Biomarkers
  • Immunology top 0.1%
    • Immune Cell Function and Interaction
    • Immunotherapy and Immune Responses

Papers in

    • CAR-T cell therapy research 73
    • Cancer Immunotherapy and Biomarkers 16
    • Immunotherapy and Immune Responses 29
    • Immune Cell Function and Interaction 19

Michael D. Kalos

115 papers receiving 21.8k citations

Michael D. Kalos's Hit Papers

Disruption of TET2 promotes the therapeutic efficacy of CD19-targeted T cells 2018 · 736 citations
7360+5+10Years since publication10002.0k3.0k

Peers

Michael D. Kalos
Comparison fields: 5 of 141
  • Oncology 16.4k
  • Immunology 8.4k
  • Genetics 5.1k
  • Virology 659
  • Hematology 1.3k
Replace Simon Francis Lacey with:
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Claudio Bordignon Italy
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Chiara Bonini Italy
Donald B. Kohn United States
Jan Joseph Melenhorst United States
Michael D. Kalos relative to Simon Francis Lacey United States Simon Francis Lacey's profile →
Citations per field
00.5×1.5×2.0×
Simon Francis Lacey · 1×
Citations per year

Countries citing papers authored by Michael D. Kalos

Since Specialization
Citations

This map shows the geographic impact of Michael D. Kalos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael D. Kalos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael D. Kalos more than expected).

Fields of papers citing papers by Michael D. Kalos

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael D. Kalos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael D. Kalos. The network helps show where Michael D. Kalos may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael D. Kalos, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael D. Kalos Line = papers co-authored together Michael D. Kalos links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 117 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Chimeric Antigen Receptor–Modified T Cells in Chronic Lymphoid Leukemia
Hit paper breakdown →
20113013
2
Chimeric Antigen Receptor–Modified T Cells for Acute Lymphoid Leukemia
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20132979
3
T Cells with Chimeric Antigen Receptors Have Potent Antitumor Effects and Can Establish Memory in Patients with Advanced Leukemia
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20112026
4
Gene Editing of CCR5 in Autologous CD4 T Cells of Persons Infected with HIV
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20141105
5
Cardiovascular toxicity and titin cross-reactivity of affinity-enhanced T cells in myeloma and melanoma
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2013978
6
Mesothelin-Specific Chimeric Antigen Receptor mRNA-Engineered T Cells Induce Antitumor Activity in Solid Malignancies
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2013769
7
Disruption of TET2 promotes the therapeutic efficacy of CD19-targeted T cells
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2018736
8
NY-ESO-1–specific TCR–engineered T cells mediate sustained antigen-specific antitumor effects in myeloma
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2015725
9
Identification of a Titin-Derived HLA-A1–Presented Peptide as a Cross-Reactive Target for Engineered MAGE A3–Directed T Cells
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2013579
10
Decade-Long Safety and Function of Retroviral-Modified Chimeric Antigen Receptor T Cells
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2012556
11
Cytokine release syndrome after blinatumomab treatment related to abnormal macrophage activation and ameliorated with cytokine-directed therapy
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2013545
12
T Cells Expressing Chimeric Antigen Receptors Can Cause Anaphylaxis in Humans
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2013521
13
Dual CD19 and CD123 targeting prevents antigen-loss relapses after CD19-directed immunotherapies
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2016498
14 2010458
15 2014415
16 2013400
17
The CDK4/6 Inhibitor Abemaciclib Induces a T Cell Inflamed Tumor Microenvironment and Enhances the Efficacy of PD-L1 Checkpoint Blockade
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2018365
18 2010322
19 2018287
20 2012275

About Michael D. Kalos

Michael D. Kalos is a scholar working on Oncology, Immunology, Genetics, Genetics and Hematology, having authored 117 papers that have together received 22.5k indexed citations. Recurring topics across this work include CAR-T cell therapy research (73 papers), Immunotherapy and Immune Responses (29 papers), Immune Cell Function and Interaction (19 papers), Virus-based gene therapy research (18 papers), Cancer Immunotherapy and Biomarkers (16 papers), Viral Infectious Diseases and Gene Expression in Insects (8 papers), Chronic Lymphocytic Leukemia Research (8 papers) and Acute Lymphoblastic Leukemia research (8 papers). The work is most often cited by research in Oncology (16.4k citations), Immunology (8.4k citations), Genetics (5.1k citations), Virology (659 citations) and Hematology (1.3k citations). Michael D. Kalos has collaborated with scholars based in United States, Netherlands and Switzerland. Frequent co-authors include Carl H. June, Bruce L. Levine, David L. Porter, Adam Bagg, Stephan A. Grupp, Anne Chew, Sharyn I. Katz, David Maxwell Barrett, David T. Teachey and Susan R. Rheingold. Their work appears in journals such as Blood, Clinical Cancer Research, Journal for ImmunoTherapy of Cancer, Cancer Immunology Research and Science Translational Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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