Matti Haltia
- Molecular Biology top 1%
- Physiology top 0.2%
- Neurology top 0.5%
- Neurology top 0.5%
- Cell Biology top 0.5%
- Co-authors
- Juha E. JääskeläinenAnders PaetauPirkko SantavuoriRaimo SulkavaJuhani RapolaJ. PaloAntti ServoJaana Tyynelä
- Topics
- Lysosomal Storage Disorders Research (43 papers)Alzheimer's disease research and treatments (32 papers)Amyloidosis: Diagnosis, Treatment, Outcomes (22 papers)
- Cited by
- NeurologyPhysiology
- Partner nations
- FinlandUnited StatesSweden
In The Last Decade
Matti Haltia
202 papers receiving 10.5k citations
Hit Papers
Peers
Comparison fields: 5 of 152
- Molecular Biology 4.6k
- Physiology 4.2k
- Neurology 2.2k
- Neurology 1.6k
- Cell Biology 1.5k
Countries citing papers authored by Matti Haltia
This map shows the geographic impact of Matti Haltia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matti Haltia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matti Haltia more than expected).
Fields of papers citing papers by Matti Haltia
This network shows the impact of papers produced by Matti Haltia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matti Haltia. The network helps show where Matti Haltia may publish in the future.
Co-authorship network of co-authors of Matti Haltia
This figure shows the co-authorship network connecting the top 25 collaborators of Matti Haltia. A scholar is included among the top collaborators of Matti Haltia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matti Haltia. Matti Haltia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 47 | |
| 2 | 168 | |
| 3 | 32 | |
| 4 | 62 | |
| 5 | 76 | |
| 6 | Ezrin expression in stromal cells of capillary hemangioblastoma. An immunohistochemical survey of brain tumors. | 71 |
| 7 | Vascular growth factors and receptors in capillary hemangioblastomas and hemangiopericytomas | 16 |
| 8 | 87 | |
| 9 | 407 | |
| 10 | 79 | |
| 11 | STORAGE OF SAPOSIN-A AND SAPOSIN-D IN INFANTILE NEURONAL CEROID-LIPOFUSCINOSIS | 2 |
| 12 | 191 | |
| 13 | 15 | |
| 14 | 15 | |
| 15 | 53 | |
| 16 | 120 | |
| 17 | 40 | |
| 18 | 56 | |
| 19 | 17 | |
| 20 | 6 |
About Matti Haltia
Matti Haltia is a scholar working on Physiology, Neurology and Neurology, having authored 204 papers that have together received 10.9k indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (43 papers), Alzheimer's disease research and treatments (32 papers) and Amyloidosis: Diagnosis, Treatment, Outcomes (22 papers). The work is most often cited by research in Neurology (1.6k citations), Physiology (4.2k citations) and Neurology (2.2k citations). Matti Haltia has collaborated with scholars based in Finland, United States and Sweden. Frequent co-authors include Juha E. Jääskeläinen, Anders Paetau, Pirkko Santavuori, Raimo Sulkava, Juhani Rapola, J. Palo, Antti Servo, Jaana Tyynelä, Hannu Somer and Auli Verkkoniemi. Their work appears in journals such as New England Journal of Medicine, Journal of Clinical Investigation and Nature Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.