Matthew P. Coghlan

4.1k total citations · 3 hit papers
33 papers, 2.7k citations indexed

About

Matthew P. Coghlan is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Surgery. According to data from OpenAlex, Matthew P. Coghlan has authored 33 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 16 papers in Endocrinology, Diabetes and Metabolism and 14 papers in Surgery. Recurrent topics in Matthew P. Coghlan's work include Diabetes Treatment and Management (14 papers), Pancreatic function and diabetes (13 papers) and Metabolism, Diabetes, and Cancer (12 papers). Matthew P. Coghlan is often cited by papers focused on Diabetes Treatment and Management (14 papers), Pancreatic function and diabetes (13 papers) and Metabolism, Diabetes, and Cancer (12 papers). Matthew P. Coghlan collaborates with scholars based in United Kingdom, United States and Sweden. Matthew P. Coghlan's co-authors include Kyle W. Sloop, Ricardo J. Samms, Brendan Leighton, Stacey L. Corcoran, Gregory Murphy, Julie C. Holder, Neil Pearce, John Yates, Calum Sutherland and Pamela A. Lochhead and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Medicine.

In The Last Decade

Matthew P. Coghlan

30 papers receiving 2.7k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Selective small molecule inhibitors of glycogen synthase kinase-3 modulate glycogen metabolism and gene transcription

2000 771 citations Matthew P. Coghlan, Ainsley A. Culbert et al. Chemistry & Biology profile →
Tirzepatide is an imbalanced and biased dual GIP and GLP-1 receptor agonist

2020 314 citations Francis S. Willard, Jonathan D. Douros et al. JCI Insight profile →
How May GIP Enhance the Therapeutic Efficacy of GLP-1?

2020 276 citations Ricardo J. Samms, Matthew P. Coghlan et al. Trends in Endocrinology and Metabolism profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Matthew P. Coghlan United Kingdom	20	1.7k	905	733	395	325	33	2.7k
Julie S. Moyers United States	22	2.8k 1.6×	1.0k 1.1×	667 0.9×	695 1.8×	314 1.0×	37	4.0k
Adriano Maida Germany	21	1.1k 0.6×	967 1.1×	744 1.0×	670 1.7×	150 0.5×	25	2.5k
Masatoshi Kikuchi Japan	36	2.1k 1.2×	1.0k 1.1×	957 1.3×	790 2.0×	176 0.5×	77	3.8k
Masao Kakoki United States	31	1.1k 0.6×	477 0.5×	358 0.5×	689 1.7×	203 0.6×	53	3.0k
Alexander M. Efanov Sweden	23	1.2k 0.7×	469 0.5×	1.1k 1.4×	264 0.7×	174 0.5×	40	2.0k
Nadeeja Wijesekara Canada	25	1.2k 0.7×	519 0.6×	912 1.2×	853 2.2×	120 0.4×	33	2.8k
Kazuaki Nagashima Japan	31	1.7k 1.0×	913 1.0×	1.3k 1.7×	346 0.9×	159 0.5×	86	3.3k
Gil Leibowitz Israel	30	1.3k 0.8×	885 1.0×	1.5k 2.0×	579 1.5×	253 0.8×	53	3.0k
Catherine Pavoine France	29	1.5k 0.8×	302 0.3×	369 0.5×	527 1.3×	269 0.8×	58	2.9k
David A. Jacobson United States	31	1.2k 0.7×	606 0.7×	1.1k 1.6×	294 0.7×	119 0.4×	74	2.4k

Countries citing papers authored by Matthew P. Coghlan

Since Specialization

Citations

This map shows the geographic impact of Matthew P. Coghlan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew P. Coghlan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew P. Coghlan more than expected).

Fields of papers citing papers by Matthew P. Coghlan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew P. Coghlan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew P. Coghlan. The network helps show where Matthew P. Coghlan may publish in the future.

Co-authorship network of co-authors of Matthew P. Coghlan

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew P. Coghlan. A scholar is included among the top collaborators of Matthew P. Coghlan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew P. Coghlan. Matthew P. Coghlan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

White, Alex D., et al.. (2026). GLP ‐ 1R biased cAMP agonism maintains glycemic control with reduced malaise and emesis in preclinical mammalian models. Diabetes Obesity and Metabolism. 28(3). 2317–2328.

Bhatnagar, Pallav, et al.. (2025). Tirzepatide leads to weight reduction in people with obesity due to MC4R deficiency. Nature Medicine. 31(10). 3294–3296. 1 indexed citations

Coghlan, Matthew P., et al.. (2024). PULMONARY BLASTOMYCOSIS PRESENTING AS ACUTE RESPIRATORY DISTRESS SYNDROME. CHEST Journal. 166(4). A1044–A1045.

Stuart, J., et al.. (2024). FATAL CASE OF SEVERE ACUTE NECROTIZING PANCREATITIS IN THE SETTING OF RECENT GLP-1 RECEPTOR AGONIST INITIATION. CHEST Journal. 166(4). A2764–A2765.

Bowker, Nicholas, Stephen Burgess, Christopher N. Foley, et al.. (2021). Genetically Predicted Glucose-Dependent Insulinotropic Polypeptide (GIP) Levels and Cardiovascular Disease Risk Are Driven by Distinct Causal Variants in the GIPR Region. Diabetes. 70(11). 2706–2719. 16 indexed citations

Coghlan, Matthew P., Libbey S. O′Farrell, Aaron D. Showalter, et al.. (2021). 639-P: GIP Receptor Agonism Enhances Weight Loss from Either a Biased or an Unbiased GLP-1 Receptor Agonist in DIO Mice. Diabetes. 70(Supplement_1). 2 indexed citations

Willard, Francis S., Jonathan D. Douros, M Gabe, et al.. (2020). Tirzepatide is an imbalanced and biased dual GIP and GLP-1 receptor agonist. JCI Insight. 5(17). 314 indexed citations breakdown →

Samms, Ricardo J., Matthew P. Coghlan, & Kyle W. Sloop. (2020). How May GIP Enhance the Therapeutic Efficacy of GLP-1?. Trends in Endocrinology and Metabolism. 31(6). 410–421. 276 indexed citations breakdown →

Hornigold, David C., Victor Howard, Sarah Will, et al.. (2018). A GLP-1:CCK fusion peptide harnesses the synergistic effects on metabolism of CCK-1 and GLP-1 receptor agonism in mice. Appetite. 127. 334–340. 31 indexed citations

10.

Biggs, Emma K., Lihuan Liang, Jacqueline Naylor, et al.. (2017). Development and characterisation of a novel glucagon like peptide-1 receptor antibody. Diabetologia. 61(3). 711–721. 25 indexed citations

11.

Will, Sarah, David C. Hornigold, David Baker, et al.. (2017). Gut check on diabesity: leveraging gut mechanisms for the treatment of type 2 diabetes and obesity. Current Opinion in Pharmacology. 37. 10–15. 11 indexed citations

12.

Coghlan, Matthew P. & Brendan Leighton. (2008). Glucokinase activators in diabetes management. Expert Opinion on Investigational Drugs. 17(2). 145–167. 93 indexed citations

13.

McKerrecher, Darren, Craig Johnstone, Kurt G. Pike, et al.. (2006). Predictive blood glucose lowering efficacy by Glucokinase activators in high fat fed female Zucker rats. British Journal of Pharmacology. 149(3). 328–335. 67 indexed citations

14.

Pearce, Neil, Jonathan R.S. Arch, John C. Clapham, et al.. (2004). Development of glucose intolerance in male transgenic mice overexpressing human glycogen synthase kinase-3β on a muscle-specific promoter. Metabolism. 53(10). 1322–1330. 90 indexed citations

15.

MacAulay, Katrina, et al.. (2003). Use of lithium and SB‐415286 to explore the role of glycogen synthase kinase‐3 in the regulation of glucose transport and glycogen synthase. European Journal of Biochemistry. 270(18). 3829–3838. 55 indexed citations

16.

Aiston, Susan, Matthew P. Coghlan, & Loranne Agius. (2003). Inactivation of phosphorylase is a major component of the mechanism by which insulin stimulates hepatic glycogen synthesis. European Journal of Biochemistry. 270(13). 2773–2781. 48 indexed citations

17.

Lochhead, Pamela A., Matthew P. Coghlan, Simon Q.J. Rice, & Calum Sutherland. (2001). Inhibition of GSK-3 Selectively Reduces Glucose-6-Phosphatase and Phosphoenolpyruvate Carboxykinase Gene Expression. Diabetes. 50(5). 937–946. 185 indexed citations

18.

Coghlan, Matthew P., Ainsley A. Culbert, Darren A.E. Cross, et al.. (2000). Selective small molecule inhibitors of glycogen synthase kinase-3 modulate glycogen metabolism and gene transcription. Chemistry & Biology. 7(10). 793–803. 771 indexed citations breakdown →

19.

Behre, Gerhard, Alan J. Whitmarsh, Matthew P. Coghlan, et al.. (1999). c-Jun Is a JNK-independent Coactivator of the PU.1 Transcription Factor. Journal of Biological Chemistry. 274(8). 4939–4946. 151 indexed citations

20.

Sánchez‐Olea, Roberto, Francesco Emma, Matthew P. Coghlan, & Kevin Strange. (1998). Characterization of pICln phosphorylation state and a pICln-associated protein kinase. Biochimica et Biophysica Acta (BBA) - General Subjects. 1381(1). 49–60. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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