Stephanie Oldham

1.6k total citations · 1 hit paper
26 papers, 889 citations indexed

About

Stephanie Oldham is a scholar working on Epidemiology, Molecular Biology and Surgery. According to data from OpenAlex, Stephanie Oldham has authored 26 papers receiving a total of 889 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Epidemiology, 11 papers in Molecular Biology and 9 papers in Surgery. Recurrent topics in Stephanie Oldham's work include Liver Disease Diagnosis and Treatment (6 papers), Atherosclerosis and Cardiovascular Diseases (6 papers) and Pancreatic function and diabetes (6 papers). Stephanie Oldham is often cited by papers focused on Liver Disease Diagnosis and Treatment (6 papers), Atherosclerosis and Cardiovascular Diseases (6 papers) and Pancreatic function and diabetes (6 papers). Stephanie Oldham collaborates with scholars based in United States, United Kingdom and Finland. Stephanie Oldham's co-authors include Coleen A. McNamara, James L. Trevaskis, James C. Garmey, Michelle L. Boland, Amanda C. Doran, Silvia Guionaud, Christopher J. Rhodes, Daniel B. Harmon, Brandon B. Boland and Sarah Will and has published in prestigious journals such as Nature Communications, Circulation Research and Diabetes.

In The Last Decade

Stephanie Oldham

25 papers receiving 877 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Resolution of NASH and hepatic fibrosis by the GLP-1R and GCGR dual-agonist cotadutide via modulating mitochondrial function and lipogenesis

2020 200 citations Michelle L. Boland, Rhianna C. Laker et al. Nature Metabolism profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Stephanie Oldham United States	15	373	281	246	227	208	26	889
Agata Kubaszek Finland	10	257 0.7×	201 0.7×	191 0.8×	102 0.4×	201 1.0×	12	712
Nobuhiko Wada Japan	13	302 0.8×	210 0.7×	207 0.8×	53 0.2×	204 1.0×	26	671
Kyu Yong Cho Japan	15	158 0.4×	242 0.9×	403 1.6×	116 0.5×	75 0.4×	79	834
Weixia Jian China	16	191 0.5×	284 1.0×	85 0.3×	140 0.6×	140 0.7×	36	742
Florian Willecke Germany	13	152 0.4×	266 0.9×	107 0.4×	182 0.8×	80 0.4×	20	625
Michelle Kirby United States	12	471 1.3×	243 0.9×	293 1.2×	56 0.2×	335 1.6×	16	933
Ben A. Hall United States	7	157 0.4×	320 1.1×	169 0.7×	150 0.7×	126 0.6×	8	847
Péter Reismann Hungary	13	118 0.3×	215 0.8×	151 0.6×	124 0.5×	142 0.7×	44	654
Marco R. Schroeter Germany	14	158 0.4×	226 0.8×	94 0.4×	116 0.5×	181 0.9×	24	752
Linda L. Gibson United States	12	137 0.4×	219 0.8×	196 0.8×	111 0.5×	146 0.7×	15	1.0k

Countries citing papers authored by Stephanie Oldham

Since Specialization

Citations

This map shows the geographic impact of Stephanie Oldham's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephanie Oldham with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephanie Oldham more than expected).

Fields of papers citing papers by Stephanie Oldham

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stephanie Oldham. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephanie Oldham. The network helps show where Stephanie Oldham may publish in the future.

Co-authorship network of co-authors of Stephanie Oldham

This figure shows the co-authorship network connecting the top 25 collaborators of Stephanie Oldham. A scholar is included among the top collaborators of Stephanie Oldham based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stephanie Oldham. Stephanie Oldham is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Laker, Rhianna C., Shaun Egolf, Sarah Will, et al.. (2025). GLP-1R/GCGR dual agonism dissipates hepatic steatosis to restore insulin sensitivity and rescue pancreatic β-cell function in obese male mice. Nature Communications. 16(1). 4714–4714. 1 indexed citations

Wang, Yibin, Stephanie Oldham, David C. Hornigold, et al.. (2025). Roles for Prlhr/GPR10 and Npffr2/GPR74 in feeding responses to PrRP. Molecular Metabolism. 92. 102093–102093.

Liu, Cong, Milena Schönke, Joost M. Lambooij, et al.. (2023). FGF21 protects against hepatic lipotoxicity and macrophage activation to attenuate fibrogenesis in nonalcoholic steatohepatitis. eLife. 12. 46 indexed citations

Szydlowska, Marta, Stephanie Oldham, Michael Ford, et al.. (2023). Restoring polyamine levels by supplementation of spermidine modulates hepatic immune landscape in murine model of NASH. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1869(6). 166697–166697. 5 indexed citations

Oldham, Stephanie, et al.. (2023). Macronutrients effects on satiety and food intake in older and younger adults: A randomised controlled trial. Appetite. 189. 106982–106982. 6 indexed citations

Boland, Michelle L., Rhianna C. Laker, Arkadiusz Nawrocki, et al.. (2020). Resolution of NASH and hepatic fibrosis by the GLP-1R and GCGR dual-agonist cotadutide via modulating mitochondrial function and lipogenesis. Nature Metabolism. 2(5). 413–431. 200 indexed citations breakdown →

Oldham, Stephanie, Christian Rivera, Michelle L. Boland, & James L. Trevaskis. (2019). Incorporation of a Survivable Liver Biopsy Procedure in Mice to Assess Non-alcoholic Steatohepatitis (NASH) Resolution. Journal of Visualized Experiments. 3 indexed citations

Boland, Brandon B., Michael B. Mumphrey, Hao Zheng, et al.. (2019). Combined loss of GLP-1R and Y2R does not alter progression of high-fat diet-induced obesity or response to RYGB surgery in mice. Molecular Metabolism. 25. 64–72. 28 indexed citations

Oldham, Stephanie, et al.. (2019). Incorporation of a Survivable Liver Biopsy Procedure in Mice to Assess Non-alcoholic Steatohepatitis (NASH) Resolution. Journal of Visualized Experiments. 2 indexed citations

10.

Hornigold, David C., Victor Howard, Sarah Will, et al.. (2018). A GLP-1:CCK fusion peptide harnesses the synergistic effects on metabolism of CCK-1 and GLP-1 receptor agonism in mice. Appetite. 127. 334–340. 31 indexed citations

11.

Boland, Michelle L., Stephanie Oldham, Brandon B. Boland, et al.. (2018). Nonalcoholic steatohepatitis severity is defined by a failure in compensatory antioxidant capacity in the setting of mitochondrial dysfunction. World Journal of Gastroenterology. 24(16). 1748–1765. 46 indexed citations

12.

Boland, Brandon B., Victor Howard, Sarah Will, et al.. (2018). Coadministration of GLP-1 and Y2R Receptor Agonists Synergize to Promote Muscle Glucose Uptake and Induce Diabetic Remission. Diabetes. 67(Supplement_1). 1 indexed citations

13.

Jouihan, Hani, Sarah Will, Silvia Guionaud, et al.. (2017). Superior reductions in hepatic steatosis and fibrosis with co-administration of a glucagon-like peptide-1 receptor agonist and obeticholic acid in mice. Molecular Metabolism. 6(11). 1360–1370. 49 indexed citations

14.

Kaplan, Jennifer L., Melissa A. Marshall, Chantel McSkimming, et al.. (2015). Adipocyte progenitor cells initiate monocyte chemoattractant protein-1-mediated macrophage accumulation in visceral adipose tissue. Molecular Metabolism. 4(11). 779–794. 51 indexed citations

15.

Perry, Heather M., Stephanie Oldham, Shawn P. Fahl, et al.. (2013). Helix-Loop-Helix Factor Inhibitor of Differentiation 3 Regulates Interleukin-5 Expression and B-1a B Cell Proliferation. Arteriosclerosis Thrombosis and Vascular Biology. 33(12). 2771–2779. 48 indexed citations

16.

Bagavant, Harini, Yogesh Scindia, Dominika Nąckiewicz, et al.. (2011). Deficiency of a Transcriptional Regulator, Inhibitor of Differentiation 3, Induces Glomerulonephritis in Apolipoprotein E–Deficient Mice. American Journal Of Pathology. 179(2). 651–660. 11 indexed citations

17.

Cutchins, Alexis, Daniel B. Harmon, Jennifer L. Kirby, et al.. (2011). Inhibitor of Differentiation-3 Mediates High Fat Diet-Induced Visceral Fat Expansion. Arteriosclerosis Thrombosis and Vascular Biology. 32(2). 317–324. 29 indexed citations

18.

Doran, Amanda C., Allison B. Lehtinen, Nahum Meller, et al.. (2010). Id3 Is a Novel Atheroprotective Factor Containing a Functionally Significant Single-Nucleotide Polymorphism Associated With Intima–Media Thickness in Humans. Circulation Research. 106(7). 1303–1311. 39 indexed citations

19.

Meller, Nahum, Ajay Kadakkal, Rohit Malhotra, et al.. (2010). Increased 12/15-Lipoxygenase Enhances Cell Growth, Fibronectin Deposition, and Neointimal Formation in Response to Carotid Injury. Arteriosclerosis Thrombosis and Vascular Biology. 31(1). 110–116. 12 indexed citations

20.

Fischer, Jan A., et al.. (1971). Calcitonin Stimulation of Parathyroid Hormone Secretion in Vitro. Hormone and Metabolic Research. 3(3). 223–224. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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