Robert G. Tsushima

3.4k total citations
59 papers, 2.8k citations indexed

About

Robert G. Tsushima is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Robert G. Tsushima has authored 59 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 26 papers in Cardiology and Cardiovascular Medicine and 19 papers in Surgery. Recurrent topics in Robert G. Tsushima's work include Ion channel regulation and function (35 papers), Cardiac electrophysiology and arrhythmias (23 papers) and Pancreatic function and diabetes (19 papers). Robert G. Tsushima is often cited by papers focused on Ion channel regulation and function (35 papers), Cardiac electrophysiology and arrhythmias (23 papers) and Pancreatic function and diabetes (19 papers). Robert G. Tsushima collaborates with scholars based in Canada, United States and United Kingdom. Robert G. Tsushima's co-authors include Peter H. Backx, Herbert Y. Gaisano, Xiao‐Dong Gao, Michael B. Wheeler, Laura Sheu, Fuzhen Xia, Yuk‐Man Leung, Gavin Y. Oudit, Ronald A. Li and Anne Marie Salapatek and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Circulation Research.

In The Last Decade

Robert G. Tsushima

59 papers receiving 2.8k citations

Peers

Robert G. Tsushima
Kazuaki Nagashima Japan
Yoshiyuki Toya Japan
Junji Ishida Japan
Paula Q. Barrett United States
Lisa Hahner United States
Robert F. Spurney United States
Michael W. Roe United States
Robert Gros Canada
Christoph Korbmacher Germany
Laureano D. Asico United States
Kazuaki Nagashima Japan
Robert G. Tsushima
Citations per year, relative to Robert G. Tsushima Robert G. Tsushima (= 1×) peers Kazuaki Nagashima

Countries citing papers authored by Robert G. Tsushima

Since Specialization
Citations

This map shows the geographic impact of Robert G. Tsushima's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert G. Tsushima with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert G. Tsushima more than expected).

Fields of papers citing papers by Robert G. Tsushima

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert G. Tsushima. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert G. Tsushima. The network helps show where Robert G. Tsushima may publish in the future.

Co-authorship network of co-authors of Robert G. Tsushima

This figure shows the co-authorship network connecting the top 25 collaborators of Robert G. Tsushima. A scholar is included among the top collaborators of Robert G. Tsushima based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert G. Tsushima. Robert G. Tsushima is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Jie, Jason D. Bayer, Roozbeh Aschar‐Sobbi, et al.. (2018). Complex interactions in a novel SCN5A compound mutation associated with long QT and Brugada syndrome: Implications for Na+ channel blocking pharmacotherapy for de novo conduction disease. PLoS ONE. 13(5). e0197273–e0197273. 2 indexed citations
2.
Gonzalez, Ronald, et al.. (2011). Nesfatin-1 Exerts a Direct, Glucose-Dependent Insulinotropic Action on Mouse Islet Beta and MIN6 Cells. Journal of Endocrinology. 208(3). R9–R16. 94 indexed citations
3.
Mihic, Anton, Vijay S. Chauhan, Xiao‐Dong Gao, Gavin Y. Oudit, & Robert G. Tsushima. (2011). Trafficking Defect and Proteasomal Degradation Contribute to the Phenotype of a Novel KCNH2 Long QT Syndrome Mutation. PLoS ONE. 6(3). e18273–e18273. 15 indexed citations
4.
Kang, Youhou, Yi Zhang, Tao Liang, et al.. (2010). ATP Modulates Interaction of Syntaxin-1A with Sulfonylurea Receptor 1 to Regulate Pancreatic β-Cell KATP Channels. Journal of Biological Chemistry. 286(7). 5876–5883. 12 indexed citations
5.
6.
Oudit, Gavin Y., Zamaneh Kassiri, Mahomed Patel, et al.. (2007). Angiotensin II-mediated oxidative stress and inflammation mediate the age-dependent cardiomyopathy in ACE2 null mice. Cardiovascular Research. 75(1). 29–39. 199 indexed citations
7.
Leung, Yuk‐Man, Ishtiaq Ahmed, Laura Sheu, et al.. (2006). Insulin Regulates Islet α-Cell Function by Reducing KATP Channel Sensitivity to Adenosine 5′-Triphosphate Inhibition. Endocrinology. 147(5). 2155–2162. 61 indexed citations
8.
Leung, Yuk‐Man, Ishtiaq Ahmed, Laura Sheu, et al.. (2006). Two populations of pancreatic islet α-cells displaying distinct Ca2+ channel properties. Biochemical and Biophysical Research Communications. 345(1). 340–344. 15 indexed citations
9.
Leung, Yuk‐Man, Ishtiaq Ahmed, Laura Sheu, et al.. (2005). Electrophysiological Characterization of Pancreatic Islet Cells in the Mouse Insulin Promoter-Green Fluorescent Protein Mouse. Endocrinology. 146(11). 4766–4775. 57 indexed citations
10.
Kang, Youhou, Yuk‐Man Leung, Fuzhen Xia, et al.. (2004). Syntaxin-1A Inhibits Cardiac KATP Channels by Its Actions on Nucleotide Binding Folds 1 and 2 of Sulfonylurea Receptor 2A. Journal of Biological Chemistry. 279(45). 47125–47131. 36 indexed citations
11.
Leung, Yuk‐Man, Youhou Kang, Xiao‐Dong Gao, et al.. (2003). Syntaxin 1A Binds to the Cytoplasmic C Terminus of Kv2.1 to Regulate Channel Gating and Trafficking. Journal of Biological Chemistry. 278(19). 17532–17538. 110 indexed citations
12.
Huang, Xiaohang, Junzhi Ji, George G. Holz, et al.. (2002). Syntaxin-3 and syntaxin-1A inhibit L-type calcium channel activity, insulin biosynthesis and exocytosis in beta-cell lines. Diabetologia. 45(2). 231–241. 53 indexed citations
13.
MacDonald, Patrick E., Sabine Sewing, Jianli Wang, et al.. (2002). Inhibition of Kv2.1 Voltage-dependent K+Channels in Pancreatic β-Cells Enhances Glucose-dependent Insulin Secretion. Journal of Biological Chemistry. 277(47). 44938–44945. 150 indexed citations
14.
Wickenden, Alan D., et al.. (1999). Effect of Cd<sup>2+</sup> on Kv4.2 and Kv1.4 Expressed in <i>Xenopus</i> Oocytes and on the Transient Outward Currents in Rat and Rabbit Ventricular Myocytes. Cellular Physiology and Biochemistry. 9(1). 11–28. 33 indexed citations
15.
Tsushima, Robert G., Adolfo Borges, & Peter H. Backx. (1999). Inactivated state dependence of sodium channel modulation by β-scorpion toxin. Pflügers Archiv - European Journal of Physiology. 437(5). 661–668. 22 indexed citations
16.
Tsushima, Robert G., et al.. (1998). Preferential regulation of rabbit cardiac L‐type Ca2+ current by glycolytic derived ATP via a direct allosteric pathway. The Journal of Physiology. 511(1). 67–78. 19 indexed citations
17.
Tsushima, Robert G., et al.. (1997). Pore residues critical for mu-CTX binding to rat skeletal muscle Na+ channels revealed by cysteine mutagenesis. Biophysical Journal. 73(4). 1874–1884. 29 indexed citations
18.
Tsushima, Robert G., J. Kelly, & J. Andrew Wasserstrom. (1996). Characteristics of cocaine block of purified cardiac sarcoplasmic reticulum calcium release channels. Biophysical Journal. 70(3). 1263–1274. 18 indexed citations
19.
Tsushima, Robert G., et al.. (1990). Differential Effects of Purine/Xanthine Oxidase on the Electrophysiologic Characteristics of Ventricular Tissues. Journal of Cardiovascular Pharmacology. 16(1). 50–58. 11 indexed citations
20.
Tsushima, Robert G., et al.. (1989). Functional and electrophysiological effects of quinacrine on the response of ventricular tissues to hypoxia and reoxygenation. Canadian Journal of Physiology and Pharmacology. 67(8). 929–935. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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