Matthew Metcalf
- Cardiology and Cardiovascular Medicine top 10%
- Molecular Biology
- Cellular and Molecular Neuroscience top 10%
- Hematology top 5%
- Physiology
- Co-authors
- Nigel W. BunnettPeishen ZhaoAndrew CoopSue A. ThomasErika FriedmannFrances J. KelleyChi‐Wen KaoStephen S. Gottlieb
- Topics
- Pharmacological Receptor Mechanisms and Effects (6 papers)Neuropeptides and Animal Physiology (5 papers)Heart Failure Treatment and Management (4 papers)
- Partner nations
- United StatesUnited KingdomAustralia
In The Last Decade
Matthew Metcalf
18 papers receiving 765 citations
Peers
Comparison fields: 5 of 97
- Cardiology and Cardiovascular Medicine 249
- Molecular Biology 231
- Cellular and Molecular Neuroscience 188
- Hematology 158
- Physiology 84
Countries citing papers authored by Matthew Metcalf
This map shows the geographic impact of Matthew Metcalf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew Metcalf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew Metcalf more than expected).
Fields of papers citing papers by Matthew Metcalf
This network shows the impact of papers produced by Matthew Metcalf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew Metcalf. The network helps show where Matthew Metcalf may publish in the future.
Co-authorship network of co-authors of Matthew Metcalf
This figure shows the co-authorship network connecting the top 25 collaborators of Matthew Metcalf. A scholar is included among the top collaborators of Matthew Metcalf based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew Metcalf. Matthew Metcalf is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 20 | |
| 3 | 1 | |
| 4 | 0 | |
| 5 | 154 | |
| 6 | 10 | |
| 7 | 195 | |
| 8 | 3 | |
| 9 | 19 | |
| 10 | 28 | |
| 11 | 63 | |
| 12 | 6 | |
| 13 | 5 | |
| 14 | 39 | |
| 15 | 96 | |
| 16 | 93 | |
| 17 | 4 | |
| 18 | 5 | |
| 19 | 44 |
About Matthew Metcalf
Matthew Metcalf is a scholar working on Cellular and Molecular Neuroscience, Pharmacology and Cardiology and Cardiovascular Medicine, having authored 19 papers that have together received 786 indexed citations. Recurring topics across this work include Pharmacological Receptor Mechanisms and Effects (6 papers), Neuropeptides and Animal Physiology (5 papers) and Heart Failure Treatment and Management (4 papers). The work is most often cited by research in Hematology (158 citations), Cardiology and Cardiovascular Medicine (249 citations) and Cellular and Molecular Neuroscience (188 citations). Matthew Metcalf has collaborated with scholars based in United States, United Kingdom and Australia. Frequent co-authors include Nigel W. Bunnett, Peishen Zhao, Andrew Coop, Sue A. Thomas, Erika Friedmann, Frances J. Kelley, Chi‐Wen Kao, Stephen S. Gottlieb, D.J. Connolly and Harriet M. Syme. Their work appears in journals such as Journal of Biological Chemistry, The FASEB Journal and The American Journal of Cardiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.