Mary B. Gibbs

705 total citations
36 papers, 542 citations indexed

About

Mary B. Gibbs is a scholar working on Hematology, Physiology and Genetics. According to data from OpenAlex, Mary B. Gibbs has authored 36 papers receiving a total of 542 indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Hematology, 17 papers in Physiology and 10 papers in Genetics. Recurrent topics in Mary B. Gibbs's work include Blood groups and transfusion (23 papers), Erythrocyte Function and Pathophysiology (15 papers) and Diabetes and associated disorders (9 papers). Mary B. Gibbs is often cited by papers focused on Blood groups and transfusion (23 papers), Erythrocyte Function and Pathophysiology (15 papers) and Diabetes and associated disorders (9 papers). Mary B. Gibbs collaborates with scholars based in United States and China. Mary B. Gibbs's co-authors include R. Thomas Solis, Joseph H. Akeroyd, Joel Solomon, A. J. Bowdler, Dennis Goldfinger, Robert Silber, Creighton B. Wright, Chengyu Sheng, Chun Han and Quan Yuan and has published in prestigious journals such as Nature, Nature Communications and Blood.

In The Last Decade

Mary B. Gibbs

34 papers receiving 491 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mary B. Gibbs United States 14 179 142 138 92 64 36 542
Takanori Moriyama Japan 13 68 0.4× 33 0.2× 74 0.5× 13 0.1× 39 0.6× 40 489
P. Braquet France 13 36 0.2× 98 0.7× 98 0.7× 16 0.2× 45 0.7× 29 473
Beatriz E. Brito Venezuela 10 103 0.6× 40 0.3× 80 0.6× 35 0.4× 44 0.7× 18 399
Hiroyuki Hirahara Japan 10 22 0.1× 23 0.2× 158 1.1× 12 0.1× 19 0.3× 44 615
H. Dávid Germany 12 14 0.1× 65 0.5× 148 1.1× 9 0.1× 142 2.2× 65 486
Maria Stefaniotou Greece 17 21 0.1× 15 0.1× 233 1.7× 52 0.6× 67 1.0× 59 1.1k
Rahul Sharma India 12 53 0.3× 74 0.5× 163 1.2× 5 0.1× 39 0.6× 69 530
Junhua Lv China 14 81 0.5× 38 0.3× 762 5.5× 7 0.1× 72 1.1× 36 1.2k
Paula Persson Denmark 11 173 1.0× 56 0.4× 197 1.4× 3 0.0× 57 0.9× 15 620
Karine N. Traill Austria 11 14 0.1× 56 0.4× 168 1.2× 18 0.2× 73 1.1× 20 536

Countries citing papers authored by Mary B. Gibbs

Since Specialization
Citations

This map shows the geographic impact of Mary B. Gibbs's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mary B. Gibbs with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mary B. Gibbs more than expected).

Fields of papers citing papers by Mary B. Gibbs

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mary B. Gibbs. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mary B. Gibbs. The network helps show where Mary B. Gibbs may publish in the future.

Co-authorship network of co-authors of Mary B. Gibbs

This figure shows the co-authorship network connecting the top 25 collaborators of Mary B. Gibbs. A scholar is included among the top collaborators of Mary B. Gibbs based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mary B. Gibbs. Mary B. Gibbs is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chen, Yang, Mary B. Gibbs, Chengyu Sheng, et al.. (2021). Brain-specific lipoprotein receptors interact with astrocyte derived apolipoprotein and mediate neuron-glia lipid shuttling. Nature Communications. 12(1). 2408–2408. 33 indexed citations
2.
Poe, Amy R., Bei Wang, Maria L. Sapar, et al.. (2018). Robust CRISPR/Cas9-Mediated Tissue-Specific Mutagenesis Reveals Gene Redundancy and Perdurance inDrosophila. Genetics. 211(2). 459–472. 43 indexed citations
3.
Gibbs, Mary B., Caixia Long, Anna Kim, et al.. (2018). Transcriptional Regulation of Lipophorin Receptors Supports Neuronal Adaptation to Chronic Elevations of Activity. Cell Reports. 25(5). 1181–1192.e4. 11 indexed citations
4.
Sheng, Chengyu, Uzma Javed, Mary B. Gibbs, et al.. (2018). Experience-dependent structural plasticity targets dynamic filopodia in regulating dendrite maturation and synaptogenesis. Nature Communications. 9(1). 3362–3362. 23 indexed citations
5.
Solis, R. Thomas, et al.. (1974). Physical Characteristics of Microaggregates in Stored Blood. Transfusion. 14(6). 538–550. 44 indexed citations
6.
Solis, R. Thomas, Creighton B. Wright, Mary B. Gibbs, & Paul Stevens. (1974). Quantitative Studies of Microaggregate Formation in Vitro and in Vivo. CHEST Journal. 65(4). 44S–46S. 13 indexed citations
7.
Gibbs, Mary B., et al.. (1967). The Binding of Naturally Occurring Anti-B Isohemagglutinins to the B Antigens of Human Erythrocytes. The Journal of Immunology. 98(3). 461–472. 2 indexed citations
8.
Gibbs, Mary B. & Richard E. Rosenfield. (1966). Immunochemical Studies of the Rh System. IV. Hemagglutination Assay of Antigenic Expression Regulated by Interaction Between Paired Rh Genes. Transfusion. 6(5). 462–474. 5 indexed citations
9.
Gibbs, Mary B., et al.. (1965). Evaluation of Electronic Measurements of Hemagglutination for Quantitative Studies. The Journal of Immunology. 94(1). 62–66. 7 indexed citations
10.
Gibbs, Mary B.. (1965). EVALUATION OF ELECTRONIC MEASUREMENTS OF HEMAGGLUTINATION FOR QUANTITATIVE STUDIES. I. LIMITATIONS IN THE APPLICATION OF INSTRUMENT MEASUREMENTS.. PubMed. 94. 55–61. 5 indexed citations
11.
Gibbs, Mary B., et al.. (1965). EVALUATION OF ELECTRONIC MEASUREMENTS OF HEMAGGLUTINATION FOR QUANTITATIVE STUDIES. II. METHODS FOR ENUMERATION OF FREE CELLS IN AGGLUTINATION.. PubMed. 94. 62–6. 11 indexed citations
12.
Solomon, Joel, Mary B. Gibbs, & A. J. Bowdler. (1965). Methods in Quantitative Hemagglutination*. Vox Sanguinis. 10(1). 54–72. 17 indexed citations
13.
Gibbs, Mary B.. (1965). Evaluation of Electronic Measurements of Hemagglutination for Quantitative Studies. The Journal of Immunology. 94(1). 55–61. 3 indexed citations
14.
Gibbs, Mary B. & Elmer L. Becker. (1963). Quantitation of Hæmagglutination by Enumeration of Free Cells by an Electronic Counter. Nature. 198(4875). 90–90. 8 indexed citations
15.
Gibbs, Mary B., et al.. (1961). Quantitative Hemagglutination Inhibition Studies of Blood Group Substances. The Journal of Immunology. 87(4). 396–404. 10 indexed citations
16.
Gibbs, Mary B., et al.. (1961). Quantitative hemagglutination inhibition studies of blood group substances. II. Characterization of anti-A isohemagglutinins by their behavior with blood group A substances.. PubMed. 87. 405–14. 7 indexed citations
17.
Silber, Robert, et al.. (1961). Quantitative Hemagglutination Studies in the Rh Blood Group System. II. A Study of the D (Rho) Agglutinogen. Blood. 17(3). 291–302. 24 indexed citations
18.
Gibbs, Mary B. & Joseph H. Akeroyd. (1959). Quantitative Immunohematologic Studies of Hemagglutination. The Journal of Immunology. 82(6). 568–576. 9 indexed citations
19.
Gibbs, Mary B., et al.. (1959). On Counting Leukocytes by Electronic Means. American Journal of Clinical Pathology. 31(2_ts). 188–192. 11 indexed citations
20.
Gibbs, Mary B. & Joseph H. Akeroyd. (1959). Quantitative Immunohematologic Studies of Hemagglutination. The Journal of Immunology. 82(6). 577–584. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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