Martin Margittai
- Molecular Biology top 5%
- Physiology top 1%
- Cell Biology top 0.5%
- Cellular and Molecular Neuroscience top 2%
- Neurology top 5%
- Co-authors
- Ralf LangenReinhard JahnDirk FasshauerStefan PabstPaul D. DinkelMin ChenJeannie ChenWolfram Antonin
- Topics
- Alzheimer's disease research and treatments (21 papers)Cellular transport and secretion (14 papers)Prion Diseases and Protein Misfolding (13 papers)
- Cited by
- Cell BiologyPhysiologyBiophysics
- Journals
- CellProceedings of the National Academy of SciencesJournal of the American Chemical Society
- Partner nations
- United StatesGermanyChina
In The Last Decade
Martin Margittai
43 papers receiving 3.3k citations
Peers
Comparison fields: 5 of 106
- Molecular Biology 2.4k
- Physiology 1.5k
- Cell Biology 1.3k
- Cellular and Molecular Neuroscience 656
- Neurology 341
Countries citing papers authored by Martin Margittai
This map shows the geographic impact of Martin Margittai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Margittai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Margittai more than expected).
Fields of papers citing papers by Martin Margittai
This network shows the impact of papers produced by Martin Margittai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Margittai. The network helps show where Martin Margittai may publish in the future.
Co-authorship network of co-authors of Martin Margittai
This figure shows the co-authorship network connecting the top 25 collaborators of Martin Margittai. A scholar is included among the top collaborators of Martin Margittai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Margittai. Martin Margittai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 21 | |
| 3 | 2 | |
| 4 | 17 | |
| 5 | 35 | |
| 6 | 32 | |
| 7 | 4 | |
| 8 | 25 | |
| 9 | 6 | |
| 10 | 39 | |
| 11 | 32 | |
| 12 | 408 | |
| 13 | 59 | |
| 14 | 53 | |
| 15 | 149 | |
| 16 | 198 | |
| 17 | 38 | |
| 18 | 101 | |
| 19 | 109 | |
| 20 | 251 |
About Martin Margittai
Martin Margittai is a scholar working on Physiology, Cell Biology and Biophysics, having authored 43 papers that have together received 3.4k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (21 papers), Cellular transport and secretion (14 papers) and Prion Diseases and Protein Misfolding (13 papers). The work is most often cited by research in Cell Biology (1.3k citations), Physiology (1.5k citations) and Biophysics (261 citations). Martin Margittai has collaborated with scholars based in United States, Germany and China. Frequent co-authors include Ralf Langen, Reinhard Jahn, Dirk Fasshauer, Stefan Pabst, Paul D. Dinkel, Min Chen, Jeannie Chen, Wolfram Antonin, Burkhard Rammner and Tao Xu. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Journal of the American Chemical Society.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.