Mark Slevin

9.9k citations
223 papers · 7.3k indexed · 1 hit paper · h-index 44

Mark Slevin

214 papers receiving 7.1k citations

Hit Papers

Epidermoid anal cancer: Results from the UKCCCR randomise...5811996202620062016100200300400500

Peers

Mark Slevin
Comparison fields: 5 of 148
  • Neurology 827
  • Cell Biology 803
  • Biological Psychiatry 105
  • Developmental Neuroscience 168
  • Oncology 1.0k
Replace Maria B. Grant with:
Maria B. Grant United States
Fabrizio Montecucco Italy
Tao Wang China
Wolfgang G. Junger United States
Nikolaus Gaßler Germany
Duncan J. Stewart Canada
Yun Wang China
Carl J. Hauser United States
David J. Pinsky United States
Holger Jon Møller Denmark
Mark Slevin relative to Maria B. Grant United States Maria B. Grant's profile →
Citations per field
00.5×1.5×2.0×
Maria B. Grant · 1×
Citations per year

Countries citing papers authored by Mark Slevin

Since Specialization
Citations

This map shows the geographic impact of Mark Slevin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Slevin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Slevin more than expected).

Fields of papers citing papers by Mark Slevin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark Slevin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Slevin. The network helps show where Mark Slevin may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Mark Slevin, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mark Slevin Line = papers co-authored together Mark Slevin links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20252
2 20250
3 20250
4 20250
5 20252
6 20252
7 20252
8 20252
9 20241
10 202434
11 20233
12 20206
13 201817
14 201795
15 201630
16 200920
17 200989
18 200645
19 199333
20 19861

About Mark Slevin

Mark Slevin is a scholar working on Neurology, Cancer Research and Biological Psychiatry, having authored 223 papers that have together received 7.3k indexed citations. Recurring topics across this work include Angiogenesis and VEGF in Cancer (35 papers), Neuroinflammation and Neurodegeneration Mechanisms (20 papers), Protease and Inhibitor Mechanisms (18 papers), Adipokines, Inflammation, and Metabolic Diseases (18 papers), Acute Ischemic Stroke Management (14 papers), Cell Adhesion Molecules Research (12 papers), Atherosclerosis and Cardiovascular Diseases (11 papers) and Alzheimer's disease research and treatments (11 papers). The work is most often cited by research in Neurology (827 citations), Cell Biology (803 citations) and Biological Psychiatry (105 citations). Mark Slevin has collaborated with scholars based in United Kingdom, Spain and Romania. Frequent co-authors include Jerzy Krupiński, John Gaffney, Shant Kumar, Lina Badimón, Nessar Ahmed, Pat Kumar, Sabine Matou, S. Kumar, Patricia Kumar and Esther Peña. Their work appears in journals such as British Journal of Cancer, International Journal of Molecular Sciences, Frontiers in Immunology, Scientific Reports and Stroke.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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