María M. Municio

2.9k citations
16 papers · 2.7k · h-index 15

Impact in

Papers in

    • NF-κB Signaling Pathways 3
    • Protein Kinase Regulation and GTPase Signaling 12
    • Cell death mechanisms and regulation 4
    • Melanoma and MAPK Pathways 2

María M. Municio

16 papers receiving 2.7k citations

Peers

María M. Municio
Comparison fields: 5 of 86
  • Cancer Research 507
  • Aging 66
  • Molecular Biology 2.2k
  • Cell Biology 436
  • Immunology 370
Replace Christopher M. BELHAM with:
Christopher M. BELHAM United Kingdom
Yair N. Doza United Kingdom
Stephen R. James United Kingdom
Judy Wynne United Kingdom
Andreas Hecht Germany
Roy Katso United Kingdom
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Jean‐Marc Brondello France
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Citations per field
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Citations per year

Countries citing papers authored by María M. Municio

Since Specialization
Citations

This map shows the geographic impact of María M. Municio's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by María M. Municio with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites María M. Municio more than expected).

Fields of papers citing papers by María M. Municio

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by María M. Municio. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by María M. Municio. The network helps show where María M. Municio may publish in the future.

Co-authors

The 25 scholars most cited alongside María M. Municio, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with María M. Municio Line = papers co-authored together María M. Municio links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 1994435
2 1993352
3 1996336
4 1995248
5 1994218
6 1993214
7 1994208
8 1997158
9 1992152
10 1993122
11 1996110
12 199280
13 199439
14 199134
15 199233
16 19929

About María M. Municio

María M. Municio is a scholar working on Cancer Research, Molecular Biology, Genetics, Endocrinology, Diabetes and Metabolism and Aging, having authored 16 papers that have together received 2.7k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (12 papers), Cell death mechanisms and regulation (4 papers), NF-κB Signaling Pathways (3 papers), Cancer-related Molecular Pathways (2 papers), Coagulation, Bradykinin, Polyphosphates, and Angioedema (2 papers), Melanoma and MAPK Pathways (2 papers), Growth Hormone and Insulin-like Growth Factors (2 papers) and Reproductive Biology and Fertility (2 papers). The work is most often cited by research in Cancer Research (507 citations), Aging (66 citations), Molecular Biology (2.2k citations), Cell Biology (436 citations) and Immunology (370 citations). María M. Municio has collaborated with scholars based in Spain, Norway and France. Frequent co-authors include Maria Teresa Díaz-Meco, Jorge Moscat, Laura Sanz, José Lozano, Edurne Berra, Isabel M. Dominguez, Pilar Sánchez‐Gómez, Sonia Frutos, I. Domínguez Jiménez and Robert S. Chapkin. Their work appears in journals such as Molecular and Cellular Biology, Journal of Biological Chemistry, The EMBO Journal and Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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