Michael Peitz

5.3k total citations · 3 hit papers
57 papers, 3.7k citations indexed

About

Michael Peitz is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Michael Peitz has authored 57 papers receiving a total of 3.7k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 11 papers in Cellular and Molecular Neuroscience and 6 papers in Genetics. Recurrent topics in Michael Peitz's work include Pluripotent Stem Cells Research (26 papers), CRISPR and Genetic Engineering (18 papers) and RNA Interference and Gene Delivery (6 papers). Michael Peitz is often cited by papers focused on Pluripotent Stem Cells Research (26 papers), CRISPR and Genetic Engineering (18 papers) and RNA Interference and Gene Delivery (6 papers). Michael Peitz collaborates with scholars based in Germany, United States and United Kingdom. Michael Peitz's co-authors include Oliver Brüstle, Frank Edenhofer, Klaus Rajewsky, Kurt Pfannkuche, Christopher Sliwinski, Matthias Hebisch, Rudolph E. Tanzi, Doo Yeon Kim, Oliver Brüstle and Se Hoon Choi and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Michael Peitz

54 papers receiving 3.6k citations

Hit Papers

A three-dimensional human neural cell culture model of Al... 2011 2026 2016 2021 2014 2011 2015 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael Peitz Germany 25 2.6k 889 617 513 413 57 3.7k
Scott Noggle United States 27 2.7k 1.0× 581 0.7× 575 0.9× 397 0.8× 421 1.0× 53 3.4k
Jérôme Mertens United States 24 2.9k 1.1× 1.0k 1.2× 772 1.3× 324 0.6× 550 1.3× 39 4.0k
Wado Akamatsu Japan 35 2.6k 1.0× 929 1.0× 484 0.8× 243 0.5× 597 1.4× 89 3.7k
Gist F. Croft United States 20 3.3k 1.3× 626 0.7× 537 0.9× 586 1.1× 359 0.9× 27 4.1k
Mi‐Yoon Chang South Korea 30 2.9k 1.1× 1.2k 1.4× 437 0.7× 338 0.7× 607 1.5× 55 3.8k
Philipp Koch Germany 33 2.9k 1.1× 1.2k 1.4× 545 0.9× 544 1.1× 1.0k 2.5× 75 4.2k
Baoyang Hu China 26 2.4k 0.9× 768 0.9× 386 0.6× 396 0.8× 734 1.8× 73 3.4k
Shauna H. Yuan United States 19 1.5k 0.6× 568 0.6× 782 1.3× 360 0.7× 331 0.8× 34 2.4k
Naihe Jing China 37 3.6k 1.4× 522 0.6× 458 0.7× 249 0.5× 475 1.2× 147 4.7k

Countries citing papers authored by Michael Peitz

Since Specialization
Citations

This map shows the geographic impact of Michael Peitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Peitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Peitz more than expected).

Fields of papers citing papers by Michael Peitz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Peitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Peitz. The network helps show where Michael Peitz may publish in the future.

Co-authorship network of co-authors of Michael Peitz

This figure shows the co-authorship network connecting the top 25 collaborators of Michael Peitz. A scholar is included among the top collaborators of Michael Peitz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael Peitz. Michael Peitz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Müller, Stephan A., Pinja Kettunen, Šárka Lehtonen, et al.. (2025). Inflammation-induced lysosomal dysfunction in human iPSC-derived microglia is exacerbated by APOE 4/4 genotype. Journal of Neuroinflammation. 22(1). 147–147. 3 indexed citations
3.
Zellner, Andreas, Nils Braun, Thomas Bajaj, et al.. (2023). Loss of function of FIP200 in human pluripotent stem cell-derived neurons leads to axonal pathology and hyperactivity. Translational Psychiatry. 13(1). 143–143. 5 indexed citations
4.
Peitz, Michael, et al.. (2023). Bringing to light the physiological and pathological firing patterns of human induced pluripotent stem cell-derived neurons using optical recordings. Frontiers in Cellular Neuroscience. 16. 1039957–1039957. 4 indexed citations
5.
Wischhof, Lena, Michael Peitz, Oliver Brüstle, et al.. (2022). BCL7A ‐containing SWI/SNF/BAF complexes modulate mitochondrial bioenergetics during neural progenitor differentiation. The EMBO Journal. 41(23). e110595–e110595. 15 indexed citations
6.
Weykopf, Beatrice, Simone Haupt, Nicolas Wiest-Daesslé, et al.. (2022). High-content phenotyping of Parkinson's disease patient stem cell-derived midbrain dopaminergic neurons using machine learning classification. Stem Cell Reports. 17(10). 2349–2364. 12 indexed citations
7.
Manstein, Felix, Christina Kropp, Caroline Halloin, et al.. (2021). High Density Bioprocessing of Human Pluripotent Stem Cells by Metabolic Control and in Silico Modeling. Stem Cells Translational Medicine. 10(7). 1063–1080. 56 indexed citations
8.
Scholl, Catharina, Michael Steffens, Fabian Elgner, et al.. (2020). Impact of Zika Virus Infection on Human Neural Stem Cell MicroRNA Signatures. Viruses. 12(11). 1219–1219. 20 indexed citations
9.
Jäger, Andreas, et al.. (2020). A novel serum-free medium for the isolation, expansion and maintenance of stemness and tissue-specific markers of primary human periodontal ligament cells. Annals of Anatomy - Anatomischer Anzeiger. 231. 151517–151517. 7 indexed citations
10.
Boni, Laura de, Gilles Gasparoni, Sascha Tierling, et al.. (2018). DNA methylation alterations in iPSC- and hESC-derived neurons: potential implications for neurological disease modeling. Clinical Epigenetics. 10(1). 13–13. 32 indexed citations
11.
Peitz, Michael, et al.. (2018). Blood-derived integration-free iPS cell line UKBi011-A from a diagnosed male Alzheimer's disease patient with APOE ɛ4/ɛ4 genotype. Stem Cell Research. 29. 250–253. 7 indexed citations
12.
Falk, Anna, Vivi M. Heine, Adrian J. Harwood, et al.. (2016). Modeling psychiatric disorders: from genomic findings to cellular phenotypes. Molecular Psychiatry. 21(9). 1167–1179. 65 indexed citations
13.
Ruschel, Jörg, Farida Hellal, Kevin C. Flynn, et al.. (2015). Systemic administration of epothilone B promotes axon regeneration after spinal cord injury. Science. 348(6232). 347–352. 343 indexed citations breakdown →
14.
Kubaczka, Caroline, Claire E. Senner, Marcos J. Araúzo‐Bravo, et al.. (2015). Direct Induction of Trophoblast Stem Cells from Murine Fibroblasts. Cell stem cell. 17(5). 557–568. 69 indexed citations
15.
Qin, Jie, Stephanie Sontag, Qiong Lin, et al.. (2014). Cell Fusion Enhances Mesendodermal Differentiation of Human Induced Pluripotent Stem Cells. Stem Cells and Development. 23(23). 2875–2882. 5 indexed citations
16.
Kubaczka, Caroline, Claire E. Senner, Marcos J. Araúzo‐Bravo, et al.. (2014). Derivation and Maintenance of Murine Trophoblast Stem Cells under Defined Conditions. Stem Cell Reports. 2(2). 232–242. 84 indexed citations
17.
Peitz, Michael, et al.. (2014). Cell-permeant recombinant Nanog protein promotes pluripotency by inhibiting endodermal specification. Stem Cell Research. 12(3). 680–689. 18 indexed citations
18.
Hucke, Stephanie, Ildikò Rita Dunay, Kathrin Frenzel, et al.. (2012). Licensing of myeloid cells promotes central nervous system autoimmunity and is controlled by peroxisome proliferator-activated receptor γ. Brain. 135(5). 1586–1605. 48 indexed citations
19.
Hussein, Samer M. I., Nizar N. Batada, Sanna Vuoristo, et al.. (2011). Copy number variation and selection during reprogramming to pluripotency. Nature. 471(7336). 58–62. 693 indexed citations breakdown →
20.
Peitz, Michael, Kurt Pfannkuche, Klaus Rajewsky, & Frank Edenhofer. (2002). Ability of the hydrophobic FGF and basic TAT peptides to promote cellular uptake of recombinant Cre recombinase: A tool for efficient genetic engineering of mammalian genomes. Proceedings of the National Academy of Sciences. 99(7). 4489–4494. 275 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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