Lucette Faille

888 total citations
20 papers, 721 citations indexed

About

Lucette Faille is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Lucette Faille has authored 20 papers receiving a total of 721 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 9 papers in Cellular and Molecular Neuroscience and 9 papers in Neurology. Recurrent topics in Lucette Faille's work include Botulinum Toxin and Related Neurological Disorders (8 papers), Neurological disorders and treatments (7 papers) and Cellular transport and secretion (5 papers). Lucette Faille is often cited by papers focused on Botulinum Toxin and Related Neurological Disorders (8 papers), Neurological disorders and treatments (7 papers) and Cellular transport and secretion (5 papers). Lucette Faille collaborates with scholars based in France, Spain and Germany. Lucette Faille's co-authors include D. Angaut‐Petit, Jordi Molgó, Joan X. Comella, A Mallart, Nacira Tabti, Joseph J. McArdle, Jean-Marie Brigant, Roland Bournaud, A. Hoffmann and D. H. Hinzen and has published in prestigious journals such as Nature, Brain Research and Neuroscience.

In The Last Decade

Lucette Faille

20 papers receiving 697 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lucette Faille France 15 394 322 302 141 93 20 721
Rita Giuliano United States 14 273 0.7× 279 0.9× 268 0.9× 41 0.3× 163 1.8× 17 864
Jin-Chong Xu United States 10 235 0.6× 144 0.4× 277 0.9× 86 0.6× 73 0.8× 11 575
Matthias Bauer Germany 8 297 0.8× 456 1.4× 266 0.9× 124 0.9× 101 1.1× 8 722
Jeanette J. Norden United States 11 545 1.4× 89 0.3× 541 1.8× 179 1.3× 91 1.0× 16 912
Laurent Bogdanik United States 12 304 0.8× 413 1.3× 599 2.0× 130 0.9× 171 1.8× 19 1.1k
Isabelle Bardou France 15 177 0.4× 123 0.4× 193 0.6× 85 0.6× 83 0.9× 26 686
Michèle Aguera France 8 496 1.3× 245 0.8× 246 0.8× 185 1.3× 49 0.5× 11 762
Jesús F. Torres-Peraza Spain 13 543 1.4× 189 0.6× 429 1.4× 61 0.4× 44 0.5× 14 737
Katsuyuki Miyaguchi Japan 9 218 0.6× 103 0.3× 297 1.0× 132 0.9× 37 0.4× 12 528
Kyoko Ajiki Japan 11 283 0.7× 49 0.2× 318 1.1× 102 0.7× 94 1.0× 13 602

Countries citing papers authored by Lucette Faille

Since Specialization
Citations

This map shows the geographic impact of Lucette Faille's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lucette Faille with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lucette Faille more than expected).

Fields of papers citing papers by Lucette Faille

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lucette Faille. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lucette Faille. The network helps show where Lucette Faille may publish in the future.

Co-authorship network of co-authors of Lucette Faille

This figure shows the co-authorship network connecting the top 25 collaborators of Lucette Faille. A scholar is included among the top collaborators of Lucette Faille based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lucette Faille. Lucette Faille is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cifuentes-Díaz, Carmen, Lucette Faille, Danièle Goudou, et al.. (2002). Abnormal reinnervation of skeletal muscle in a tenascin-C-deficient mouse. Journal of Neuroscience Research. 67(1). 93–93. 1 indexed citations
2.
Cifuentes-Díaz, Carmen, Lucette Faille, Danièle Goudou, et al.. (2001). Abnormal reinnervation of skeletal muscle in a tenascin‐C‐deficient mouse. Journal of Neuroscience Research. 67(1). 93–99. 26 indexed citations
3.
Angaut‐Petit, D., et al.. (1998). Enhanced neurotransmitter release is associated with reduction of neuronal branching in a Drosophila mutant overexpressing frequenin. European Journal of Neuroscience. 10(2). 423–434. 32 indexed citations
4.
5.
Cifuentes-Díaz, Carmen, Elena Velasco, Frédéric A. Meunier, et al.. (1998). The peripheral nerve and the neuromuscular junction are affected in the tenascin-C-deficient mouse.. PubMed. 44(2). 357–79. 35 indexed citations
7.
Meunier, Frédéric A., Cesare Colasante, Lucette Faille, Myriam Gastard, & Jordi Molgó. (1996). Upregulation of calcitonin gene-related peptide at mouse motor nerve terminals poisoned with botulinum type-A toxin. Pflügers Archiv - European Journal of Physiology. 431(S6). R297–R298. 16 indexed citations
8.
Comella, Joan X., et al.. (1996). Nerve terminal sprouting in botulinum type-A treated mouse levator auris longus muscle. Neuromuscular Disorders. 6(3). 177–185. 70 indexed citations
9.
Molgó, Jordi, et al.. (1996). Synaptotagmin II immunoreactivity in normal and botulinum type-A treated mouse motor nerve terminals. Pflügers Archiv - European Journal of Physiology. 431(S6). R283–R284. 11 indexed citations
10.
Angaut‐Petit, D., Jordi Molgó, Lucette Faille, et al.. (1995). Mouse motor nerve terminal immunoreactivity to synaptotagmin II during sustained quantal transmitter release. Brain Research. 681(1-2). 213–217. 15 indexed citations
11.
Angaut‐Petit, D., Alberto Ferrús, & Lucette Faille. (1993). Plasticity of motor nerve terminals in Drosophila T(X, Y) V7 mutant: Effect of deregulation of the novel calcium-binding protein frequenin. Neuroscience Letters. 153(2). 227–231. 14 indexed citations
12.
Comella, Joan X., Jordi Molgó, & Lucette Faille. (1993). Sprouting of mammalian motor nerve terminals induced by in vivo injection of botulinum type-D toxin and the functional recovery of paralysed neuromuscular junctions. Neuroscience Letters. 153(1). 61–64. 28 indexed citations
13.
Faille, Lucette, et al.. (1992). Divalent cation changes in cerebellar purkinje cells after climbing fiber deafferentation. Journal of Physiology-Paris. 86(1-3). 39–45. 1 indexed citations
14.
Angaut‐Petit, D., Jordi Molgó, Joan X. Comella, Lucette Faille, & Nacira Tabti. (1990). Terminal sprouting in mouse neuromuscular junctions poisoned with botulinum type a toxin: Morphological and electrophysiological features. Neuroscience. 37(3). 799–808. 97 indexed citations
15.
Molgó, Jordi, Joan X. Comella, D. Angaut‐Petit, et al.. (1990). Presynaptic actions of botulinal neurotoxins at vertebrate neuromuscular junctions.. PubMed. 84(2). 152–66. 69 indexed citations
16.
Martin, Andrew R., Vikas V. Patel, Lucette Faille, & A Mallart. (1989). Presynaptic calcium currents recorded from calyciform nerve terminals in the lizard ciliary ganglion. Neuroscience Letters. 105(1-2). 14–18. 14 indexed citations
17.
Angaut‐Petit, D., et al.. (1987). The levator auris longus muscle of the mouse: A convenient preparation for studies of short- and long-term presynaptic effects of drugs or toxins. Neuroscience Letters. 82(1). 83–88. 70 indexed citations
18.
Angaut‐Petit, D. & Lucette Faille. (1987). Inability of regenerating mouse motor axons to innervate a denervated target. Neuroscience Letters. 75(2). 163–168. 8 indexed citations
19.
McArdle, Joseph J., D. Angaut‐Petit, A Mallart, et al.. (1981). Advantages of the triangularis sterni muscle of the mouse for investigations of synaptic phenomena. Journal of Neuroscience Methods. 4(2). 109–115. 103 indexed citations
20.
Tauc, L, A. Hoffmann, Shigeru Tsuji, D. H. Hinzen, & Lucette Faille. (1974). Transmission abolished on a cholinergic synapse after injection of acetylcholin-esterase into the presynaptic neurone. Nature. 250(5466). 496–498. 82 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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