D. Angaut‐Petit

1.3k total citations
35 papers, 1.1k citations indexed

About

D. Angaut‐Petit is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, D. Angaut‐Petit has authored 35 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Cellular and Molecular Neuroscience, 17 papers in Molecular Biology and 12 papers in Neurology. Recurrent topics in D. Angaut‐Petit's work include Ion channel regulation and function (11 papers), Botulinum Toxin and Related Neurological Disorders (10 papers) and Neurobiology and Insect Physiology Research (9 papers). D. Angaut‐Petit is often cited by papers focused on Ion channel regulation and function (11 papers), Botulinum Toxin and Related Neurological Disorders (10 papers) and Neurobiology and Insect Physiology Research (9 papers). D. Angaut‐Petit collaborates with scholars based in France, Spain and Germany. D. Angaut‐Petit's co-authors include Lucette Faille, Jordi Molgó, A Mallart, Joan X. Comella, Nacira Tabti, Joseph J. McArdle, Roland Bournaud, Alberto Ferrús, Jean-Marie Brigant and S. Thesleff and has published in prestigious journals such as The EMBO Journal, The Journal of Physiology and Brain Research.

In The Last Decade

D. Angaut‐Petit

35 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
D. Angaut‐Petit France 18 591 426 333 216 196 35 1.1k
David Tonge United Kingdom 24 997 1.7× 572 1.3× 247 0.7× 233 1.1× 254 1.3× 65 1.7k
JW Lichtman United States 11 983 1.7× 741 1.7× 145 0.4× 106 0.5× 249 1.3× 11 1.4k
R Ironton United Kingdom 8 550 0.9× 292 0.7× 223 0.7× 103 0.5× 103 0.5× 14 808
Roberto Navarrete United Kingdom 16 346 0.6× 651 1.5× 146 0.4× 207 1.0× 135 0.7× 21 1.1k
Anna Östberg United Kingdom 12 610 1.0× 470 1.1× 99 0.3× 114 0.5× 141 0.7× 21 943
Greg A. Weir United Kingdom 14 468 0.8× 467 1.1× 187 0.6× 327 1.5× 110 0.6× 24 1.0k
Hans J. ten Donkelaar Netherlands 16 262 0.4× 307 0.7× 115 0.3× 116 0.5× 94 0.5× 32 856
Albert A. Herrera United States 19 782 1.3× 535 1.3× 112 0.3× 66 0.3× 219 1.1× 26 1.1k
David P. Crockett United States 17 412 0.7× 197 0.5× 167 0.5× 118 0.5× 99 0.5× 34 870
Jeanette J. Norden United States 11 545 0.9× 541 1.3× 89 0.3× 91 0.4× 179 0.9× 16 912

Countries citing papers authored by D. Angaut‐Petit

Since Specialization
Citations

This map shows the geographic impact of D. Angaut‐Petit's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D. Angaut‐Petit with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D. Angaut‐Petit more than expected).

Fields of papers citing papers by D. Angaut‐Petit

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by D. Angaut‐Petit. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D. Angaut‐Petit. The network helps show where D. Angaut‐Petit may publish in the future.

Co-authorship network of co-authors of D. Angaut‐Petit

This figure shows the co-authorship network connecting the top 25 collaborators of D. Angaut‐Petit. A scholar is included among the top collaborators of D. Angaut‐Petit based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with D. Angaut‐Petit. D. Angaut‐Petit is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cifuentes-Díaz, Carmen, Lucette Faille, Danièle Goudou, et al.. (2002). Abnormal reinnervation of skeletal muscle in a tenascin-C-deficient mouse. Journal of Neuroscience Research. 67(1). 93–93. 1 indexed citations
2.
Cifuentes-Díaz, Carmen, Lucette Faille, Danièle Goudou, et al.. (2001). Abnormal reinnervation of skeletal muscle in a tenascin‐C‐deficient mouse. Journal of Neuroscience Research. 67(1). 93–99. 26 indexed citations
3.
Angaut‐Petit, D., et al.. (1998). Enhanced neurotransmitter release is associated with reduction of neuronal branching in a Drosophila mutant overexpressing frequenin. European Journal of Neuroscience. 10(2). 423–434. 32 indexed citations
4.
5.
Cifuentes-Díaz, Carmen, Elena Velasco, Frédéric A. Meunier, et al.. (1998). The peripheral nerve and the neuromuscular junction are affected in the tenascin-C-deficient mouse.. PubMed. 44(2). 357–79. 35 indexed citations
7.
Comella, Joan X., et al.. (1996). Nerve terminal sprouting in botulinum type-A treated mouse levator auris longus muscle. Neuromuscular Disorders. 6(3). 177–185. 70 indexed citations
8.
Molgó, Jordi, et al.. (1996). Synaptotagmin II immunoreactivity in normal and botulinum type-A treated mouse motor nerve terminals. Pflügers Archiv - European Journal of Physiology. 431(S6). R283–R284. 11 indexed citations
9.
Angaut‐Petit, D., Jordi Molgó, Lucette Faille, et al.. (1995). Mouse motor nerve terminal immunoreactivity to synaptotagmin II during sustained quantal transmitter release. Brain Research. 681(1-2). 213–217. 15 indexed citations
10.
Angaut‐Petit, D., Alberto Ferrús, & Lucette Faille. (1993). Plasticity of motor nerve terminals in Drosophila T(X, Y) V7 mutant: Effect of deregulation of the novel calcium-binding protein frequenin. Neuroscience Letters. 153(2). 227–231. 14 indexed citations
11.
Mallart, A, et al.. (1991). Nerve Terminal Excitability and Neuromuscular Transmission inT(X;Y)V7andShakerMutants ofDrosophila Melanogaster. Journal of Neurogenetics. 7(2-3). 75–84. 45 indexed citations
12.
Molgó, Jordi, Esperanza Del Pozo, Josep-Eladı́ Baños, & D. Angaut‐Petit. (1991). Changes of quantal transmitter release caused by gadolinium ions at the frog neuromuscular junction. British Journal of Pharmacology. 104(1). 133–138. 32 indexed citations
13.
Angaut‐Petit, D., Jordi Molgó, Joan X. Comella, Lucette Faille, & Nacira Tabti. (1990). Terminal sprouting in mouse neuromuscular junctions poisoned with botulinum type a toxin: Morphological and electrophysiological features. Neuroscience. 37(3). 799–808. 97 indexed citations
14.
Benoit, Évelyne, D. Angaut‐Petit, & A Mallart. (1989). Potassium channels in lizard nodes of Ranvier and motor endings. Pflügers Archiv - European Journal of Physiology. 414(S1). S133–S134. 1 indexed citations
15.
Mallart, A, Jordi Molgó, D. Angaut‐Petit, & S. Thesleff. (1989). Is the internal calcium regulation altered in Type A botulinum toxin-poisoned motor endings?. Brain Research. 479(1). 167–171. 15 indexed citations
16.
Angaut‐Petit, D., Évelyne Benoit, & A Mallart. (1989). Membrane currents in lizard motor nerve terminals and nodes of Ranvier. Pflügers Archiv - European Journal of Physiology. 415(1). 81–87. 10 indexed citations
17.
Angaut‐Petit, D., et al.. (1987). The levator auris longus muscle of the mouse: A convenient preparation for studies of short- and long-term presynaptic effects of drugs or toxins. Neuroscience Letters. 82(1). 83–88. 70 indexed citations
18.
Molgó, Jordi, D. Angaut‐Petit, & S. Thesleff. (1987). In botulinum type A-poisoned frog motor endings ouabain induces phasic transmitter release through Na+-Ca2+ exchange. Brain Research. 410(2). 385–389. 6 indexed citations
19.
Angaut‐Petit, D., et al.. (1982). Electrophysiological and morphological studies of a motor nerve in ‘motor endplate disease’ of the mouse. Proceedings of the Royal Society of London. Series B, Biological sciences. 215(1198). 117–125. 31 indexed citations
20.
McArdle, Joseph J., D. Angaut‐Petit, A Mallart, et al.. (1981). Advantages of the triangularis sterni muscle of the mouse for investigations of synaptic phenomena. Journal of Neuroscience Methods. 4(2). 109–115. 103 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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