Nacira Tabti

1.4k total citations
30 papers, 1.0k citations indexed

About

Nacira Tabti is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Nacira Tabti has authored 30 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 17 papers in Cellular and Molecular Neuroscience and 12 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Nacira Tabti's work include Ion channel regulation and function (25 papers), Cardiac electrophysiology and arrhythmias (11 papers) and Neuroscience and Neural Engineering (8 papers). Nacira Tabti is often cited by papers focused on Ion channel regulation and function (25 papers), Cardiac electrophysiology and arrhythmias (11 papers) and Neuroscience and Neural Engineering (8 papers). Nacira Tabti collaborates with scholars based in France, United States and Sweden. Nacira Tabti's co-authors include Bertrand Fontaine, Emmanuel Fournier, Damien Sternberg, Jordi Molgó, Pascal Laforêt, S. Thesleff, Jean‐Claude Willer, Joan X. Comella, Savine Vicart and D. Angaut‐Petit and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Neurology and The Journal of Physiology.

In The Last Decade

Nacira Tabti

30 papers receiving 996 citations

Peers

Nacira Tabti
F.G.I. Jennekens Netherlands
Richa Sud United Kingdom
Robyn Labrum United Kingdom
Josef Gehrmann United States
Wendy G. Resneck United States
Nacira Tabti
Citations per year, relative to Nacira Tabti Nacira Tabti (= 1×) peers C. Doriguzzi

Countries citing papers authored by Nacira Tabti

Since Specialization
Citations

This map shows the geographic impact of Nacira Tabti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nacira Tabti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nacira Tabti more than expected).

Fields of papers citing papers by Nacira Tabti

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nacira Tabti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nacira Tabti. The network helps show where Nacira Tabti may publish in the future.

Co-authorship network of co-authors of Nacira Tabti

This figure shows the co-authorship network connecting the top 25 collaborators of Nacira Tabti. A scholar is included among the top collaborators of Nacira Tabti based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nacira Tabti. Nacira Tabti is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fournier, Emmanuel & Nacira Tabti. (2019). Clinical electrophysiology of muscle diseases and episodic muscle disorders. Handbook of clinical neurology. 161. 269–280. 9 indexed citations
2.
Lainé, Jeanne, Gunnar Skoglund, Emmanuel Fournier, & Nacira Tabti. (2018). Development of the excitation-contraction coupling machinery and its relation to myofibrillogenesis in human iPSC-derived skeletal myocytes. Skeletal Muscle. 8(1). 1–1. 15 indexed citations
3.
Skoglund, Gunnar, Jeanne Lainé, Radbod Darabi, et al.. (2014). Physiological and ultrastructural features of human induced pluripotent and embryonic stem cell-derived skeletal myocytes in vitro. Proceedings of the National Academy of Sciences. 111(22). 8275–8280. 32 indexed citations
4.
Fournier, Emmanuel, et al.. (2009). Cold‐induced disruption of Na+ channel slow inactivation underlies paralysis in highly thermosensitive paramyotonia. The Journal of Physiology. 587(8). 1705–1714. 19 indexed citations
5.
Sternberg, Damien, Nacira Tabti, Savine Vicart, et al.. (2009). Homozygosity for dominant mutations increases severity of muscle channelopathies. Muscle & Nerve. 41(4). 470–477. 26 indexed citations
6.
Fontaine, Bertrand, Emmanuel Fournier, Damien Sternberg, Savine Vicart, & Nacira Tabti. (2007). Hypokalemic Periodic Paralysis: A Model for a Clinical and Research Approach to a Rare Disorder. Neurotherapeutics. 4(2). 225–232. 15 indexed citations
7.
Lhuillier, Loïc, Sandrine Luce, Damien Sternberg, et al.. (2006). Gating defects of a novel Na+ channel mutant causing hypokalemic periodic paralysis. Biochemical and Biophysical Research Communications. 348(2). 653–661. 26 indexed citations
8.
Fournier, Emmanuel, Karine Viala, Hélène Gervais, et al.. (2006). Cold extends electromyography distinction between ion channel mutations causing myotonia. Annals of Neurology. 60(3). 356–365. 107 indexed citations
9.
Luce, Sandrine, et al.. (2005). A1152D mutation of the Na+ channel causes paramyotonia congenita and emphasizes the role of DIII/S4–S5 linker in fast inactivation. The Journal of Physiology. 565(2). 415–427. 16 indexed citations
10.
Sternberg, Damien, et al.. (2003). Functional characterization and cold sensitivity of T1313A, a new mutation of the skeletal muscle sodium channel causing paramyotonia congenita in humans. The Journal of Physiology. 554(3). 635–647. 42 indexed citations
11.
Sternberg, Damien, Nacira Tabti, Emmanuel Fournier, Bernard Hainque, & Bertrand Fontaine. (2003). Lack of association of the potassium channel–associated peptide MiRP2-R83H variant with periodic paralysis. Neurology. 61(6). 857–859. 34 indexed citations
12.
Lhuillier, Loïc & Nacira Tabti. (1998). Influence of muscle cells on the development of calcium currents in Xenopus spinal neurons. Neuroscience. 83(4). 1283–1291. 5 indexed citations
13.
Lhuillier, Loïc, et al.. (1998). Functional expression of the Ile693Thr Na+ channel mutation associated with paramyotonia congenita in a human cell line. The Journal of Physiology. 507(3). 721–727. 31 indexed citations
14.
Tabti, Nacira & Mu‐ming Poo. (1994). Study on the induction of spontaneous transmitter release at early nerve-muscle contacts in Xenopus cultures. Neuroscience Letters. 173(1-2). 21–26. 6 indexed citations
15.
Girod, Romain, et al.. (1992). Pulsatile release of acetylcholine by nerve terminals (synaptosomes) isolated from Torpedo electric organ.. The Journal of Physiology. 450(1). 325–340. 12 indexed citations
16.
Angaut‐Petit, D., Jordi Molgó, Joan X. Comella, Lucette Faille, & Nacira Tabti. (1990). Terminal sprouting in mouse neuromuscular junctions poisoned with botulinum type a toxin: Morphological and electrophysiological features. Neuroscience. 37(3). 799–808. 97 indexed citations
17.
Tabti, Nacira & Mu‐ming Poo. (1990). Chapter 7 Spontaneous synaptic activity at developing neuromuscular junctions. Progress in brain research. 84. 63–72. 7 indexed citations
18.
Molgó, Jordi, et al.. (1989). A study of synchronization of quantal transmitter release from mammalian motor endings by the use of botulinal toxins type A and D.. The Journal of Physiology. 411(1). 195–205. 43 indexed citations
19.
Tabti, Nacira, et al.. (1989). Three potassium currents in mouse motor nerve terminals. Pflügers Archiv - European Journal of Physiology. 413(4). 395–400. 49 indexed citations
20.
Rieu, M. Chalret du, et al.. (1982). [Detection of peripheral diabetic neuropathy. A reappraisal of electrophysiological data].. PubMed. 11(47). 3485–9. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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