Lothar Kußmaul

1.8k total citations · 1 hit paper
15 papers, 1.5k citations indexed

About

Lothar Kußmaul is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Lothar Kußmaul has authored 15 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Cellular and Molecular Neuroscience and 4 papers in Physiology. Recurrent topics in Lothar Kußmaul's work include Mitochondrial Function and Pathology (3 papers), Neuroscience and Neuropharmacology Research (3 papers) and Alzheimer's disease research and treatments (3 papers). Lothar Kußmaul is often cited by papers focused on Mitochondrial Function and Pathology (3 papers), Neuroscience and Neuropharmacology Research (3 papers) and Alzheimer's disease research and treatments (3 papers). Lothar Kußmaul collaborates with scholars based in Germany, United Kingdom and Switzerland. Lothar Kußmaul's co-authors include Judy Hirst, Ralf Dringen, Bernd Hamprecht, Jan Mirko Gutterer, Johannes Hirrlinger, Bastian Hengerer, Frank Gillardon, Norbert Kraut, Karin M. Danzer and Wolfgang Rist and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Biochemistry and Journal of Neurochemistry.

In The Last Decade

Lothar Kußmaul

15 papers receiving 1.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The mechanism of superoxide production by NADH:ubiquinone oxidoreductase (complex I) from bovine heart mitochondria

2006 565 citations Lothar Kußmaul, Judy Hirst Proceedings of the National Academy of Sciences profile →

Peers

Lothar Kußmaul

PHYSI

CMN

NEURO

BIOCH

Heiko Noack Germany

Victoria C. Stewart United Kingdom

Maria Paola Cerù Italy

Jan Seyfried Germany

Sadaki Fujimoto Japan

Simon J.R. Heales United Kingdom

Patrizia Formichi Italy

Elena Radi Italy

Jan Mirko Gutterer Germany

Kent A. Robinson United States

Heiko Noack Germany

Lothar Kußmaul

579 ×0.7

384 ×1.0

PHYSI

214 ×0.8

CMN

310 ×1.6

NEURO

144 ×0.9

BIOCH

Citations per year, relative to Lothar Kußmaul Lothar Kußmaul (= 1×) peers Heiko Noack

Countries citing papers authored by Lothar Kußmaul

Since Specialization

Citations

This map shows the geographic impact of Lothar Kußmaul's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lothar Kußmaul with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lothar Kußmaul more than expected).

Fields of papers citing papers by Lothar Kußmaul

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lothar Kußmaul. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lothar Kußmaul. The network helps show where Lothar Kußmaul may publish in the future.

Co-authorship network of co-authors of Lothar Kußmaul

This figure shows the co-authorship network connecting the top 25 collaborators of Lothar Kußmaul. A scholar is included among the top collaborators of Lothar Kußmaul based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lothar Kußmaul. Lothar Kußmaul is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

15 of 15 papers shown

Nan, Xinsheng, Pedro Chacón, Lothar Kußmaul, et al.. (2019). A New Mouse Line Reporting the Translation of Brain-Derived Neurotrophic Factor Using Green Fluorescent Protein. eNeuro. 7(1). ENEURO.0462–19.2019. 9 indexed citations

Becker, Robert E., Natalia Gass, Lothar Kußmaul, et al.. (2019). NMDA receptor antagonists traxoprodil and lanicemine improve hippocampal-prefrontal coupling and reward-related networks in rats. Psychopharmacology. 236(12). 3451–3463. 8 indexed citations

Mangialasche, Francesca, Mauro Baglioni, Roberta Cecchetti, et al.. (2015). Lymphocytic Mitochondrial Aconitase Activity is Reduced in Alzheimer's Disease and Mild Cognitive Impairment. Journal of Alzheimer s Disease. 44(2). 649–660. 47 indexed citations

Kroker, Katja S., et al.. (2013). Restoring long-term potentiation impaired by amyloid-beta oligomers: Comparison of an acetylcholinesterase inhibitior and selective neuronal nicotinic receptor agonists. Brain Research Bulletin. 96. 28–38. 23 indexed citations

Kroker, Katja S., Cathrin Schnack, Christine A. F. Von Arnim, et al.. (2013). Globular and Protofibrillar Aβ Aggregates Impair Neurotransmission by Different Mechanisms. Biochemistry. 52(8). 1466–1476. 21 indexed citations

Kußmaul, Lothar, et al.. (2010). Activation of the mitochondrial protein quality control system and actin cytoskeletal alterations in cells harbouring the MELAS mitochondrial DNA mutation. Journal of the Neurological Sciences. 295(1-2). 46–52. 10 indexed citations

Gillardon, Frank, Wolfgang Rist, Lothar Kußmaul, et al.. (2007). Proteomic and functional alterations in brain mitochondria from Tg2576 mice occur before amyloid plaque deposition. PROTEOMICS. 7(4). 605–616. 118 indexed citations

Kußmaul, Lothar & Judy Hirst. (2006). The mechanism of superoxide production by NADH:ubiquinone oxidoreductase (complex I) from bovine heart mitochondria. Proceedings of the National Academy of Sciences. 103(20). 7607–7612. 565 indexed citations breakdown →

Danzeisen, Ruth, Frank Gillardon, Erich Buerger, et al.. (2005). Targeted Antioxidative and Neuroprotective Properties of the Dopamine Agonist Pramipexole and Its Nondopaminergic Enantiomer SND919CL2x [(+)2-Amino-4,5,6,7-tetrahydro-6-lpropylamino-benzathiazole Dihydrochloride]. Journal of Pharmacology and Experimental Therapeutics. 316(1). 189–199. 90 indexed citations

10.

Rahman, Basim, Lothar Kußmaul, Bernd Hamprecht, & Ralf Dringen. (2000). Glycogen is mobilized during the disposal of peroxides by cultured astroglial cells from rat brain. Neuroscience Letters. 290(3). 169–172. 45 indexed citations

11.

Hirrlinger, Johannes, Jan Mirko Gutterer, Lothar Kußmaul, Bernd Hamprecht, & Ralf Dringen. (2000). Microglial Cells in Culture Express a Prominent Glutathione System for the Defense against Reactive Oxygen Species. Developmental Neuroscience. 22(5-6). 384–392. 82 indexed citations

12.

Kußmaul, Lothar, Bernd Hamprecht, & Ralf Dringen. (1999). The Detoxification of Cumene Hydroperoxide by the Glutathione System of Cultured Astroglial Cells Hinges on Hexose Availability for the Regeneration of NADPH. Journal of Neurochemistry. 73(3). 1246–1253. 53 indexed citations

13.

Dringen, Ralf, Lothar Kußmaul, Jan Mirko Gutterer, Johannes Hirrlinger, & Bernd Hamprecht. (1999). The Glutathione System of Peroxide Detoxification Is Less Efficient in Neurons than in Astroglial Cells. Journal of Neurochemistry. 72(6). 2523–2530. 197 indexed citations

14.

Dringen, Ralf, Lothar Kußmaul, & Bernd Hamprecht. (1998). Detoxification of exogenous hydrogen peroxide and organic hydroperoxides by cultured astroglial cells assessed by microtiter plate assay. Brain Research Protocols. 2(3). 223–228. 161 indexed citations

15.

Dringen, Ralf, Lothar Kußmaul, & Bernd Hamprecht. (1998). Rapid clearance of tertiary butyl hydroperoxide by cultured astroglial cells via oxidation of glutathione. Glia. 23(2). 139–145. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact