Elena Radi

1.8k total citations · 2 hit papers
21 papers, 1.4k citations indexed

About

Elena Radi is a scholar working on Molecular Biology, Neurology and Clinical Biochemistry. According to data from OpenAlex, Elena Radi has authored 21 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 10 papers in Neurology and 5 papers in Clinical Biochemistry. Recurrent topics in Elena Radi's work include Cerebrovascular and genetic disorders (7 papers), Mitochondrial Function and Pathology (6 papers) and Neurological Disease Mechanisms and Treatments (3 papers). Elena Radi is often cited by papers focused on Cerebrovascular and genetic disorders (7 papers), Mitochondrial Function and Pathology (6 papers) and Neurological Disease Mechanisms and Treatments (3 papers). Elena Radi collaborates with scholars based in Italy and Romania. Elena Radi's co-authors include Antonio Federico, Patrizia Formichi, Carla Battisti, Elena Cardaioli, Paola Da Pozzo, Gian Nicola Gallus, Maria Teresa Dotti, Anna Di Stefano, Giuseppe Di Maio and Gabriele Cevenini and has published in prestigious journals such as Biochemical and Biophysical Research Communications, Journal of Cellular Physiology and Journal of the Neurological Sciences.

In The Last Decade

Elena Radi

21 papers receiving 1.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mitochondria, oxidative stress and neurodegeneration

2012 607 citations Antonio Federico, Elena Cardaioli et al. Journal of the Neurological Sciences profile →
Apoptosis and Oxidative Stress in Neurodegenerative Diseases

2014 512 citations Elena Radi, Patrizia Formichi et al. Journal of Alzheimer s Disease profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Elena Radi Italy	13	682	402	215	205	203	21	1.4k
Patrizia Formichi Italy	17	793 1.2×	445 1.1×	251 1.2×	221 1.1×	252 1.2×	41	1.7k
Natasa Dragicevic United States	9	464 0.7×	426 1.1×	186 0.9×	232 1.1×	240 1.2×	13	1.1k
Isaac G. Onyango United States	19	822 1.2×	674 1.7×	205 1.0×	220 1.1×	232 1.1×	32	1.6k
Sarika Singh India	24	465 0.7×	230 0.6×	267 1.2×	221 1.1×	214 1.1×	57	1.4k
Jolanta Dorszewska Poland	25	710 1.0×	517 1.3×	353 1.6×	281 1.4×	254 1.3×	94	1.9k
Grzegorz A. Czapski Poland	25	622 0.9×	478 1.2×	201 0.9×	245 1.2×	299 1.5×	56	1.7k
Ludis Morales Colombia	16	476 0.7×	291 0.7×	149 0.7×	175 0.9×	251 1.2×	35	1.2k
Maria Jose Perez J. Chile	17	635 0.9×	483 1.2×	143 0.7×	231 1.1×	197 1.0×	22	1.1k
Carla Battisti Italy	23	821 1.2×	458 1.1×	458 2.1×	276 1.3×	317 1.6×	91	2.1k
Robert P. Strosznajder Poland	25	1.0k 1.5×	574 1.4×	152 0.7×	241 1.2×	221 1.1×	69	1.9k

Countries citing papers authored by Elena Radi

Since Specialization

Citations

This map shows the geographic impact of Elena Radi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elena Radi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elena Radi more than expected).

Fields of papers citing papers by Elena Radi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Elena Radi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elena Radi. The network helps show where Elena Radi may publish in the future.

Co-authorship network of co-authors of Elena Radi

This figure shows the co-authorship network connecting the top 25 collaborators of Elena Radi. A scholar is included among the top collaborators of Elena Radi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elena Radi. Elena Radi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Formichi, Patrizia, Carla Battisti, Maria Margherita De Santi, et al.. (2017). Primary cilium alterations and expression changes of Patched1 proteins in niemann‐pick type C disease. Journal of Cellular Physiology. 233(1). 663–672. 20 indexed citations

Formichi, Patrizia, Elena Radi, Carla Battisti, et al.. (2016). Oxidative stress-induced apoptosis in peripheral blood lymphocytes from patients with POLG-related disorders. Journal of the Neurological Sciences. 368. 359–368. 5 indexed citations

Formichi, Patrizia, Elena Radi, Eleonora P. Giorgi, et al.. (2015). Analysis of opa1 isoforms expression and apoptosis regulation in autosomal dominant optic atrophy (ADOA) patients with mutations in the opa1 gene. Journal of the Neurological Sciences. 351(1-2). 99–108. 8 indexed citations

Radi, Elena, Patrizia Formichi, Carla Battisti, & Antonio Federico. (2014). Apoptosis and Oxidative Stress in Neurodegenerative Diseases. Journal of Alzheimer s Disease. 42(s3). S125–S152. 512 indexed citations breakdown →

Federico, Antonio, Elena Cardaioli, Paola Da Pozzo, et al.. (2012). Mitochondria, oxidative stress and neurodegeneration. Journal of the Neurological Sciences. 322(1-2). 254–262. 607 indexed citations breakdown →

Formichi, Patrizia, Elena Radi, Carla Battisti, et al.. (2012). Effects of cerebrolysin administration on oxidative stress-induced apoptosis in lymphocytes from CADASIL patients. Neurological Sciences. 34(4). 553–556. 6 indexed citations

Formichi, Patrizia, Elena Radi, Carla Battisti, et al.. (2012). Cerebrolysin administration reduces oxidative stress‐induced apoptosis in limphocytes from healthy individuals. Journal of Cellular and Molecular Medicine. 16(11). 2840–2843. 17 indexed citations

Formichi, Patrizia, Carla Battisti, Elena Radi, Giuseppe Di Maio, & Antonio Federico. (2012). APOPTOSIS, OXIDATIVE STRESS AND NEUROLOGICAL DISEASE. INFM-OAR (INFN Catania). 1(1). 40–40. 3 indexed citations

Radi, Elena, Patrizia Formichi, Giuseppe Di Maio, Carla Battisti, & Antonio Federico. (2011). Oxidative stress-induced apoptosis in two patients with Alagille syndrome. Journal of the Neurological Sciences. 308(1-2). 49–56. 2 indexed citations

10.

Maio, Giuseppe Di, Patrizia Formichi, Elena Radi, et al.. (2011). Human peripheral blood lymphocytes and fibroblasts as Notch3 expression models. Journal of Cellular Physiology. 227(4). 1771–1775. 5 indexed citations

11.

Radi, Elena, Patrizia Formichi, Giuseppe Di Maio, Carla Battisti, & Antonio Federico. (2011). Altered apoptosis regulation in Kufor–Rakeb syndrome patients with mutations in the ATP13A2 gene. Journal of Cellular and Molecular Medicine. 16(8). 1916–1923. 6 indexed citations

12.

Formichi, Patrizia, Lucilla Parnetti, Elena Radi, et al.. (2010). CSF Biomarkers Profile in CADASIL—A Model of Pure Vascular Dementia: Usefulness in Differential Diagnosis in the Dementia Disorder. International Journal of Alzheimer s Disease. 2010. 1–6. 26 indexed citations

13.

Formichi, Patrizia, Elena Radi, Carla Battisti, et al.. (2009). Apoptosis in CADASIL: An in vitro study of lymphocytes and fibroblasts from a cohort of Italian patients. Journal of Cellular Physiology. 219(2). 494–502. 24 indexed citations

14.

Formichi, Patrizia, Lucilla Parnetti, Elena Radi, et al.. (2008). CSF levels of β‐amyloid 1‐42, tau and phosphorylated tau protein in CADASIL. European Journal of Neurology. 15(11). 1252–1255. 16 indexed citations

15.

Formichi, Patrizia, Elena Radi, Carla Battisti, et al.. (2007). Psychosine‐induced apoptosis and cytokine activation in immune peripheral cells of Krabbe patients. Journal of Cellular Physiology. 212(3). 737–743. 41 indexed citations

16.

Battisti, Carla, Patrizia Formichi, Elena Radi, & Antonio Federico. (2007). Oxidative-stress-induced apoptosis in PBLs of two patients with Parkinson disease secondary to alpha-synuclein mutation. Journal of the Neurological Sciences. 267(1-2). 120–124. 23 indexed citations

17.

Formichi, Patrizia, Carla Battisti, Elena Radi, & Antonio Federico. (2006). Cerebrospinal fluid tau, Aß, and phosphorylated tau protein for the diagnosis of Alzheimer's disease. Journal of Cellular Physiology. 208(1). 39–46. 61 indexed citations

18.

Formichi, Patrizia, et al.. (2006). Human fibroblasts undergo oxidative stress‐induced apoptosis without internucleosomal DNA fragmentation. Journal of Cellular Physiology. 208(2). 289–297. 29 indexed citations

19.

Pozzo, Paola Da, Elena Cardaioli, Elena Radi, & Antonio Federico. (2004). Sequence analysis of the complete mitochondrial genome in patients with mitochondrial encephaloneuromyopathies lacking the common pathogenic DNA mutations. Biochemical and Biophysical Research Communications. 324(1). 360–364. 12 indexed citations

20.

Cardaioli, Elena, Paola Da Pozzo, Elena Radi, Maria Teresa Dotti, & Antonio Federico. (2004). A novel heteroplasmic tRNALeu(CUN) mtDNA point mutation associated with chronic progressive external ophthalmoplegia. Biochemical and Biophysical Research Communications. 327(3). 675–678. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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