Hidefumi Ito
Impact in
Papers in
- Neurology 84
- Amyotrophic Lateral Sclerosis Research 41
- Parkinson's Disease Mechanisms and Treatments 39
- Neurological diseases and metabolism 15
-
- Genetic Neurodegenerative Diseases 19
- Co-authors
- Ryōsuke TakahashiHirofumi KusakaMasafumi IharaAsao HiranoReika WateSatoshi NakanoHidekazu TomimotoTakakuni Maki
- Journals
- Acta Neuropathologica (10 papers)Journal of the Neurological Sciences (9 papers)Neuroreport (6 papers)Neurology (6 papers)Neuropathology (6 papers)
- Partner nations
- JapanUnited StatesUnited Kingdom
In The Last Decade
Hidefumi Ito
138 papers receiving 3.8k citations
Peers
Comparison fields: 5 of 110
- Neurology 1.0k
- Neurology 1.7k
- Genetics 564
- Cellular and Molecular Neuroscience 838
- Physiology 785
Countries citing papers authored by Hidefumi Ito
This map shows the geographic impact of Hidefumi Ito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hidefumi Ito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hidefumi Ito more than expected).
Fields of papers citing papers by Hidefumi Ito
This network shows the impact of papers produced by Hidefumi Ito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hidefumi Ito. The network helps show where Hidefumi Ito may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Hidefumi Ito, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2024 | 2 | |
| 3 | 2024 | 5 | |
| 4 | 2024 | 1 | |
| 5 | 2023 | 0 | |
| 6 | 2023 | 1 | |
| 7 | 2023 | 2 | |
| 8 | 2022 | 6 | |
| 9 | 2022 | 1 | |
| 10 | 2022 | 1 | |
| 11 | 2020 | 6 | |
| 12 | 2016 | 13 | |
| 13 | 2014 | 14 | |
| 14 | 2012 | 8 | |
| 15 | 2011 | 23 | |
| 16 | 2010 | 54 | |
| 17 | 2010 | 44 | |
| 18 | 2009 | 37 | |
| 19 | 2009 | 6 | |
| 20 | 2008 | 155 |
About Hidefumi Ito
Hidefumi Ito is a scholar working on Neurology, Neurology, Cellular and Molecular Neuroscience, Genetics and Physiology, having authored 145 papers that have together received 3.9k indexed citations. Recurring topics across this work include Amyotrophic Lateral Sclerosis Research (41 papers), Parkinson's Disease Mechanisms and Treatments (39 papers), Genetic Neurodegenerative Diseases (19 papers), Neurogenetic and Muscular Disorders Research (19 papers), Alzheimer's disease research and treatments (18 papers), Neurological diseases and metabolism (15 papers), Ubiquitin and proteasome pathways (12 papers) and Endoplasmic Reticulum Stress and Disease (10 papers). The work is most often cited by research in Neurology (1.0k citations), Neurology (1.7k citations), Genetics (564 citations), Cellular and Molecular Neuroscience (838 citations) and Physiology (785 citations). Hidefumi Ito has collaborated with scholars based in Japan, United States and United Kingdom. Frequent co-authors include Ryōsuke Takahashi, Hirofumi Kusaka, Masafumi Ihara, Asao Hirano, Reika Wate, Satoshi Nakano, Hidekazu Tomimoto, Takakuni Maki, Keiji Tanaka and Youshi Fujita. Their work appears in journals such as Acta Neuropathologica, Journal of the Neurological Sciences, Neuroreport, Neurology and Neuropathology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.